Zaorui Zhao

Summary

Affiliation: University of Maryland
Country: USA

Publications

  1. pmc Comparing the predictive value of multiple cognitive, affective, and motor tasks after rodent traumatic brain injury
    Zaorui Zhao
    Department of Anesthesiology and the Center for Shock, Trauma, and Anesthesiology Research STAR, University of Maryland School of Medicine, Baltimore, Maryland 21201, USA
    J Neurotrauma 29:2475-89. 2012
  2. pmc Neuroprotective effects of geranylgeranylacetone in experimental traumatic brain injury
    Zaorui Zhao
    Department of Anesthesiology and the Center for Shock, Trauma and Anesthesiology Research STAR, University of Maryland School of Medicine, Baltimore, Maryland, USA
    J Cereb Blood Flow Metab 33:1897-908. 2013
  3. pmc Downregulation of miR-23a and miR-27a following experimental traumatic brain injury induces neuronal cell death through activation of proapoptotic Bcl-2 proteins
    Boris Sabirzhanov
    Department of Anesthesiology and Center for Shock, Trauma, and Anesthesiology Research, University of Maryland School of Medicine
    J Neurosci 34:10055-71. 2014
  4. pmc Voluntary Exercise Preconditioning Activates Multiple Antiapoptotic Mechanisms and Improves Neurological Recovery after Experimental Traumatic Brain Injury
    Zaorui Zhao
    Department of Anesthesiology and Center for Shock, Trauma, and Anesthesiology Research STAR, University of Maryland School of Medicine, Baltimore, Maryland
    J Neurotrauma 32:1347-60. 2015
  5. pmc Spinal cord injury causes brain inflammation associated with cognitive and affective changes: role of cell cycle pathways
    Junfang Wu
    Department of Anesthesiology and Center for Shock, Trauma and Anesthesiology Research STAR, University of Maryland School of Medicine, Baltimore, Maryland 21201
    J Neurosci 34:10989-1006. 2014
  6. pmc Late exercise reduces neuroinflammation and cognitive dysfunction after traumatic brain injury
    Chun Shu Piao
    Center for Shock, Trauma and Anesthesiology Research STAR and Department of Anesthesiology, University of Maryland School of Medicine, Baltimore, Maryland 21201, USA
    Neurobiol Dis 54:252-63. 2013
  7. pmc Cell cycle inhibition limits development and maintenance of neuropathic pain following spinal cord injury
    Junfang Wu
    aDepartment of Anesthesiology and Center for Shock, Trauma and Anesthesiology Research STAR, University of Maryland School of Medicine, Baltimore, MD, USA bDepartment of Anatomy and Neurobiology, University of Maryland School of Medicine, Baltimore, MD, USA cDepartment of Pain and Translational Symptom Science, University of Maryland School of Nursing, Baltimore, MD, USA
    Pain 157:488-503. 2016

Collaborators

Detail Information

Publications7

  1. pmc Comparing the predictive value of multiple cognitive, affective, and motor tasks after rodent traumatic brain injury
    Zaorui Zhao
    Department of Anesthesiology and the Center for Shock, Trauma, and Anesthesiology Research STAR, University of Maryland School of Medicine, Baltimore, Maryland 21201, USA
    J Neurotrauma 29:2475-89. 2012
    ....
  2. pmc Neuroprotective effects of geranylgeranylacetone in experimental traumatic brain injury
    Zaorui Zhao
    Department of Anesthesiology and the Center for Shock, Trauma and Anesthesiology Research STAR, University of Maryland School of Medicine, Baltimore, Maryland, USA
    J Cereb Blood Flow Metab 33:1897-908. 2013
    ..Given these neuroprotective actions and considering its longstanding clinical use, GGA should be considered for the clinical treatment of TBI. ..
  3. pmc Downregulation of miR-23a and miR-27a following experimental traumatic brain injury induces neuronal cell death through activation of proapoptotic Bcl-2 proteins
    Boris Sabirzhanov
    Department of Anesthesiology and Center for Shock, Trauma, and Anesthesiology Research, University of Maryland School of Medicine
    J Neurosci 34:10055-71. 2014
    ..These findings indicate that post-traumatic decreases in miR-23a and miR-27a contribute to neuronal cell death after TBI by upregulating proapoptotic Bcl-2 family members, thus providing a novel therapeutic target. ..
  4. pmc Voluntary Exercise Preconditioning Activates Multiple Antiapoptotic Mechanisms and Improves Neurological Recovery after Experimental Traumatic Brain Injury
    Zaorui Zhao
    Department of Anesthesiology and Center for Shock, Trauma, and Anesthesiology Research STAR, University of Maryland School of Medicine, Baltimore, Maryland
    J Neurotrauma 32:1347-60. 2015
    ..Given these neuroprotective actions, voluntary physical exercise may serve to limit the consequences of TBI. ..
  5. pmc Spinal cord injury causes brain inflammation associated with cognitive and affective changes: role of cell cycle pathways
    Junfang Wu
    Department of Anesthesiology and Center for Shock, Trauma and Anesthesiology Research STAR, University of Maryland School of Medicine, Baltimore, Maryland 21201
    J Neurosci 34:10989-1006. 2014
    ....
  6. pmc Late exercise reduces neuroinflammation and cognitive dysfunction after traumatic brain injury
    Chun Shu Piao
    Center for Shock, Trauma and Anesthesiology Research STAR and Department of Anesthesiology, University of Maryland School of Medicine, Baltimore, Maryland 21201, USA
    Neurobiol Dis 54:252-63. 2013
    ..These data underscore the critical importance of timing of exercise initiation after trauma and its relation to neuroinflammation, and challenge the widely held view that effective neuroprotection requires early intervention...
  7. pmc Cell cycle inhibition limits development and maintenance of neuropathic pain following spinal cord injury
    Junfang Wu
    aDepartment of Anesthesiology and Center for Shock, Trauma and Anesthesiology Research STAR, University of Maryland School of Medicine, Baltimore, MD, USA bDepartment of Anatomy and Neurobiology, University of Maryland School of Medicine, Baltimore, MD, USA cDepartment of Pain and Translational Symptom Science, University of Maryland School of Nursing, Baltimore, MD, USA
    Pain 157:488-503. 2016
    ..Thus, our data suggest that cell cycle modulation may provide an effective therapeutic strategy to reduce hyperesthesia after SCI, with a prolonged therapeutic window. ..