Teresa K Tarrant

Summary

Affiliation: University of North Carolina
Country: USA

Publications

  1. pmc G protein-coupled receptor kinase-3-deficient mice exhibit WHIM syndrome features and attenuated inflammatory responses
    Teresa K Tarrant
    1 CB 7280, 3300 Manning Dr, Chapel Hill, NC 27599, USA
    J Leukoc Biol 94:1243-51. 2013
  2. pmc Decreased Th17 and antigen-specific humoral responses in CX₃ CR1-deficient mice in the collagen-induced arthritis model
    Teresa K Tarrant
    Department of Medicine, University of North Carolina at Chapel Hill, CB 7280, 3300 Manning Drive, Chapel Hill, NC 27599, USA
    Arthritis Rheum 64:1379-87. 2012
  3. pmc Granulocyte chemotaxis and disease expression are differentially regulated by GRK subtype in an acute inflammatory arthritis model (K/BxN)
    Teresa K Tarrant
    Thurston Arthritis Research Center, Department of Medicine, Division of Rheumatology, Allergy and Immunology, University of North Carolina School of Medicine, Chapel Hill, NC 27599, USA
    Clin Immunol 129:115-22. 2008
  4. pmc G-protein signaling modulator-3, a gene linked to autoimmune diseases, regulates monocyte function and its deficiency protects from inflammatory arthritis
    Patrick M Giguère
    Department of Pharmacology, UNC School of Medicine, Chapel Hill, NC 27599 7365, USA
    Mol Immunol 54:193-8. 2013
  5. pmc G Protein Coupled Receptor Kinase 3 Regulates Breast Cancer Migration, Invasion, and Metastasis
    Matthew J Billard
    Thurston Arthritis Research Center and the Department of Medicine, Division of Rheumatology, Allergy, and Immunology, University of North Carolina, Chapel Hill, NC 27599, United States of America
    PLoS ONE 11:e0152856. 2016
  6. pmc Enhanced Th17-cell responses render CCR2-deficient mice more susceptible for autoimmune arthritis
    Rishi R Rampersad
    Department of Medicine, Thurston Arthritis Research Center, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina, United States of America
    PLoS ONE 6:e25833. 2011
  7. pmc Targeting the molecular and cellular interactions of the bone marrow niche in immunologic disease
    Jaime M Brozowski
    Department of Microbiology and Immunology, University of North Carolina, Chapel Hill, NC, 27599, USA
    Curr Allergy Asthma Rep 14:402. 2014
  8. pmc Autoimmunity in immunodeficiency
    Krista Todoric
    Division of Allergy and Immunology, Dept of Pediatrics, University of North Carolina Hospitals, Chapel Hill, NC 27599, USA
    Curr Allergy Asthma Rep 13:361-70. 2013
  9. pmc Cellular targeting in autoimmunity
    Jennifer L Rogers
    Division of Rheumatology, Allergy, and Immunology and the Thurston Arthritis Research Center, University of North Carolina School of Medicine, Chapel Hill, NC 27517, USA
    Curr Allergy Asthma Rep 12:495-510. 2012
  10. pmc Improved outcomes on subcutaneous IgG in patients with humoral immunodeficiency and co-morbid bowel disease
    Shaili N Shah
    University of North Carolina School of Medicine, 3300 Thurston Building, CB 7280, Chapel Hill, NC 27599 7280
    Clin Case Rep Rev 1:151-152. 2015

Collaborators

  • Peng Liu
  • Dhavalkumar D Patel
  • Jaime M Brozowski
  • Nancy Klauber-Demore
  • William C Zamboni
  • Paola A Gehrig
  • Ramesh K Ramanathan
  • Marilyn Diaz
  • Roman G Timoshchenko
  • Matthew J Billard
  • Krista Todoric
  • Jennifer L Rogers
  • Sunah Kang
  • Young K Truong
  • Shaili N Shah
  • Gina Song
  • Marcus W McGinnis
  • Patrick M Giguère
  • Mohamed Elrefaei
  • Rishi R Rampersad
  • Maria A Graziewicz
  • Andrew J Monteith
  • Stephen H Clarke
  • Maria F Sassano
  • D Stephen Serafin
  • David J Fitzhugh
  • John Schmitz
  • Ruth Lininger
  • Joel S Parker
  • Barbara J Vilen
  • Chuancang Jiang
  • Jonathan S Serody
  • Yuri Fedoriw
  • Gary Sahagian
  • Taylor F White
  • Charlene M Santos
  • Suzan K Hanna
  • Craig R Lee
  • David A Barrow
  • Victoria L Bae-Jump
  • Brian K Buckley
  • Geneviève Laroche
  • Daniel Mattox
  • Denise Esserman
  • David P Siderovski
  • Jessica B Koontz
  • Oded Foreman
  • Kristie Boose
  • John L Schmitz
  • Donald S Serafin
  • Feng Chang Lin
  • Martha McGee
  • Jason P Fine
  • Michael F Weeks
  • Tatiana Quintero-Matthews
  • Christopher T Vallanat
  • Alan M Fong
  • Denise A Esserman
  • Franco Di Padova
  • Jennifer Clark
  • Peter Sazani
  • LeShara Fulton
  • Brian Buckley
  • Jennifer Roberts
  • Ryszard Kole
  • Henrik Ørum
  • Henrik Hansen

Detail Information

Publications14

  1. pmc G protein-coupled receptor kinase-3-deficient mice exhibit WHIM syndrome features and attenuated inflammatory responses
    Teresa K Tarrant
    1 CB 7280, 3300 Manning Dr, Chapel Hill, NC 27599, USA
    J Leukoc Biol 94:1243-51. 2013
    ....
  2. pmc Decreased Th17 and antigen-specific humoral responses in CX₃ CR1-deficient mice in the collagen-induced arthritis model
    Teresa K Tarrant
    Department of Medicine, University of North Carolina at Chapel Hill, CB 7280, 3300 Manning Drive, Chapel Hill, NC 27599, USA
    Arthritis Rheum 64:1379-87. 2012
    ..Because CX(3) CR1 is expressed on T cells and antigen-presenting cells, we also examined adaptive immune functions in this model...
  3. pmc Granulocyte chemotaxis and disease expression are differentially regulated by GRK subtype in an acute inflammatory arthritis model (K/BxN)
    Teresa K Tarrant
    Thurston Arthritis Research Center, Department of Medicine, Division of Rheumatology, Allergy and Immunology, University of North Carolina School of Medicine, Chapel Hill, NC 27599, USA
    Clin Immunol 129:115-22. 2008
    ..We hypothesized that GRK subtypes differentially regulate granulocyte chemotaxis and clinical disease expression in the K/BxN model...
  4. pmc G-protein signaling modulator-3, a gene linked to autoimmune diseases, regulates monocyte function and its deficiency protects from inflammatory arthritis
    Patrick M Giguère
    Department of Pharmacology, UNC School of Medicine, Chapel Hill, NC 27599 7365, USA
    Mol Immunol 54:193-8. 2013
    ..Our results suggest that GPSM3 is an important regulator of monocyte function involving mechanisms of differentiation, survival, and chemotaxis, and deficiency in GPSM3 expression is protective in acute inflammatory arthritis...
  5. pmc G Protein Coupled Receptor Kinase 3 Regulates Breast Cancer Migration, Invasion, and Metastasis
    Matthew J Billard
    Thurston Arthritis Research Center and the Department of Medicine, Division of Rheumatology, Allergy, and Immunology, University of North Carolina, Chapel Hill, NC 27599, United States of America
    PLoS ONE 11:e0152856. 2016
    ..These data demonstrate that GRK3 dysregulation may play an important part in TNBC metastasis. ..
  6. pmc Enhanced Th17-cell responses render CCR2-deficient mice more susceptible for autoimmune arthritis
    Rishi R Rampersad
    Department of Medicine, Thurston Arthritis Research Center, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina, United States of America
    PLoS ONE 6:e25833. 2011
    ....
  7. pmc Targeting the molecular and cellular interactions of the bone marrow niche in immunologic disease
    Jaime M Brozowski
    Department of Microbiology and Immunology, University of North Carolina, Chapel Hill, NC, 27599, USA
    Curr Allergy Asthma Rep 14:402. 2014
    ....
  8. pmc Autoimmunity in immunodeficiency
    Krista Todoric
    Division of Allergy and Immunology, Dept of Pediatrics, University of North Carolina Hospitals, Chapel Hill, NC 27599, USA
    Curr Allergy Asthma Rep 13:361-70. 2013
    ..The goals of this review will be to discuss these immunodeficiencies and to review the literature with respect to the proposed mechanisms for autoimmunity within each put forth to date. ..
  9. pmc Cellular targeting in autoimmunity
    Jennifer L Rogers
    Division of Rheumatology, Allergy, and Immunology and the Thurston Arthritis Research Center, University of North Carolina School of Medicine, Chapel Hill, NC 27517, USA
    Curr Allergy Asthma Rep 12:495-510. 2012
    ....
  10. pmc Improved outcomes on subcutaneous IgG in patients with humoral immunodeficiency and co-morbid bowel disease
    Shaili N Shah
    University of North Carolina School of Medicine, 3300 Thurston Building, CB 7280, Chapel Hill, NC 27599 7280
    Clin Case Rep Rev 1:151-152. 2015
    ..Further characterization of the effects of co-morbid conditions on immunoglobulin replacement is critical to providing improved and informed patient care...
  11. pmc Apoptotic Debris Accumulates on Hematopoietic Cells and Promotes Disease in Murine and Human Systemic Lupus Erythematosus
    Sunah Kang
    Department of Microbiology and Immunology, University of North Carolina, Chapel Hill, NC 27599
    J Immunol 196:4030-9. 2016
    ..Taken together, our findings reveal a previously unappreciated role for the cell surface accumulation of IgG-ICs in human and murine lupus. ..
  12. doi request reprint Roles of chemokines CCL2 and CCL5 in the pharmacokinetics of PEGylated liposomal doxorubicin in vivo and in patients with recurrent epithelial ovarian cancer
    Gina Song
    Division of Pharmacotherapy and Experimental Therapeutics, UNC Eshelman School of Pharmacy, University of North Carolina at Chapel Hill, Chapel Hill, NC, USA
    Nanomedicine 11:1797-807. 2015
    ..These data emphasize that the interplay between PLD and chemokines may have an important role in optimizing PLD therapy...
  13. doi request reprint Second generation automated anti-CCP test better predicts the clinical diagnosis of rheumatoid arthritis
    Mohamed Elrefaei
    McLendon Clinical Laboratories, University of North Carolina Hospitals, Chapel Hill, NC, USA
    J Clin Immunol 32:131-7. 2012
    ..These two new anti-CCP antibody tests have the benefit of automation and may have better positive predictive value for the diagnosis of RA than our current manual ELISA...
  14. pmc An endogenous TNF-alpha antagonist induced by splice-switching oligonucleotides reduces inflammation in hepatitis and arthritis mouse models
    Maria A Graziewicz
    Ercole Biotech, Inc, Research Triangle Park, North Carolina 27709, USA
    Mol Ther 16:1316-22. 2008
    ..This is the first report of upregulation of the endogenous, circulating TNF-alpha antagonist by oligonucleotide-induced splicing modulation...