B R Stripp

Summary

Affiliation: University of Rochester
Country: USA

Publications

  1. ncbi request reprint Pulmonary phenotype of CCSP/UG deficient mice: a consequence of CCSP deficiency or altered Clara cell function?
    B R Stripp
    Department of Environmental Medicine, University of Rochester, Box EHSC, 575 Elmwood Avenue, Rochester, NY 14642, USA
    Ann N Y Acad Sci 923:202-9. 2000
  2. ncbi request reprint Clara cell secretory protein-deficient mice differ from wild-type mice in inflammatory chemokine expression to oxygen and ozone, but not to endotoxin
    C J Johnston
    Department of Environmental Medicine, University of Rochester, NY 14642, USA
    Exp Lung Res 25:7-21. 1999
  3. ncbi request reprint Altered lung gene expression in CCSP-null mice suggests immunoregulatory roles for Clara cells
    T M Watson
    Division of Respiratory Biology and Toxicology, Department of Environmental Medicine, University of Rochester School of Medicine and Dentistry, Rochester, New York 14642, USA
    Am J Physiol Lung Cell Mol Physiol 281:L1523-30. 2001
  4. ncbi request reprint Normal function and lack of fibronectin accumulation in kidneys of Clara cell secretory protein/uteroglobin deficient mice
    S D Reynolds
    Department of Environmental Medicine, School of Medicine and Dentistry, University of Rochester, NY 14642, USA
    Am J Kidney Dis 33:541-51. 1999
  5. ncbi request reprint Exposure to hyperoxia induces p53 expression in mouse lung epithelium
    M A O'Reilly
    Department of Pediatrics Neonatology, School of Medicine and Dentistry, University of Rochester, New York 14642, USA
    Am J Respir Cell Mol Biol 18:43-50. 1998
  6. ncbi request reprint Clara cell secretory protein deficiency increases oxidant stress response in conducting airways
    G W Mango
    Department of Environmental Medicine, University of Rochester, School of Medicine and Dentistry, Rochester, New York 14642, USA
    Am J Physiol 275:L348-56. 1998
  7. ncbi request reprint Clara cell secretory protein-expressing cells of the airway neuroepithelial body microenvironment include a label-retaining subset and are critical for epithelial renewal after progenitor cell depletion
    K U Hong
    Department of Environmental Medicine, University of Rochester, Rochester, New York, USA
    Am J Respir Cell Mol Biol 24:671-81. 2001

Collaborators

  • G Oberdorster
  • S D Reynolds
  • G W Mango
  • C J Johnston
  • J N Finkelstein
  • K U Hong
  • T M Watson
  • J Lund
  • M A O'Reilly
  • A Giangreco
  • C M Hurley
  • I M Boe
  • R Gelein
  • I M Bøe
  • R J Staversky
  • C G Plopper

Detail Information

Publications7

  1. ncbi request reprint Pulmonary phenotype of CCSP/UG deficient mice: a consequence of CCSP deficiency or altered Clara cell function?
    B R Stripp
    Department of Environmental Medicine, University of Rochester, Box EHSC, 575 Elmwood Avenue, Rochester, NY 14642, USA
    Ann N Y Acad Sci 923:202-9. 2000
    ..Moreover, CCSP-/-1 mice represent a valuable tool for probing functional roles for Clara cells in regulation of airway lining fluid composition and lung pollutant susceptibility...
  2. ncbi request reprint Clara cell secretory protein-deficient mice differ from wild-type mice in inflammatory chemokine expression to oxygen and ozone, but not to endotoxin
    C J Johnston
    Department of Environmental Medicine, University of Rochester, NY 14642, USA
    Exp Lung Res 25:7-21. 1999
    ....
  3. ncbi request reprint Altered lung gene expression in CCSP-null mice suggests immunoregulatory roles for Clara cells
    T M Watson
    Division of Respiratory Biology and Toxicology, Department of Environmental Medicine, University of Rochester School of Medicine and Dentistry, Rochester, New York 14642, USA
    Am J Physiol Lung Cell Mol Physiol 281:L1523-30. 2001
    ..These findings identify new roles for Clara cells and their secretions in airway homeostasis...
  4. ncbi request reprint Normal function and lack of fibronectin accumulation in kidneys of Clara cell secretory protein/uteroglobin deficient mice
    S D Reynolds
    Department of Environmental Medicine, School of Medicine and Dentistry, University of Rochester, NY 14642, USA
    Am J Kidney Dis 33:541-51. 1999
    ..We conclude that CCSP (Ug) deficiency, per se, is not the cause of severe renal pathology and systemic disease reported for Ug-/- mice...
  5. ncbi request reprint Exposure to hyperoxia induces p53 expression in mouse lung epithelium
    M A O'Reilly
    Department of Pediatrics Neonatology, School of Medicine and Dentistry, University of Rochester, New York 14642, USA
    Am J Respir Cell Mol Biol 18:43-50. 1998
    ..These findings support the concept that hyperoxia damages DNA of pulmonary epithelial cells, which respond by accumulating p53 and changes in epithelial cell-specific gene expression...
  6. ncbi request reprint Clara cell secretory protein deficiency increases oxidant stress response in conducting airways
    G W Mango
    Department of Environmental Medicine, University of Rochester, School of Medicine and Dentistry, Rochester, New York 14642, USA
    Am J Physiol 275:L348-56. 1998
    ..These results demonstrate a protective role for Clara cells and their secretions and indicate potential genetic mechanisms that may influence susceptibility to oxidant stress...
  7. ncbi request reprint Clara cell secretory protein-expressing cells of the airway neuroepithelial body microenvironment include a label-retaining subset and are critical for epithelial renewal after progenitor cell depletion
    K U Hong
    Department of Environmental Medicine, University of Rochester, Rochester, New York, USA
    Am J Respir Cell Mol Biol 24:671-81. 2001
    ..These results support the notion that vCE cells represent either an airway stem cell or are critical for stem cell maintenance, and suggest that PNECs are not sufficient for epithelial renewal...