Faqian Li

Summary

Affiliation: University of Rochester
Country: USA

Publications

  1. doi request reprint Small neuroendocrine lesions in intrathoracic lymph nodes of patients with primary lung adenocarcinoma: real metastasis?
    Faqian Li
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, Rochester, NC, USA
    Am J Surg Pathol 34:1701-7. 2010
  2. doi request reprint Epigenetic modifications of histone h4 in lung neuroendocrine tumors
    Faqian Li
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, NY, USA
    Appl Immunohistochem Mol Morphol 19:389-94. 2011
  3. pmc Deletion of FoxO1 leads to shortening of QRS by increasing Na(+) channel activity through enhanced expression of both cardiac NaV1.5 and β3 subunit
    Benzhi Cai
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, Rochester, NY, USA
    J Mol Cell Cardiol 74:297-306. 2014
  4. pmc High frequency of coexpression of maspin with p63 and p53 in squamous cell carcinoma but not in adenocarcinoma of the lung
    Bonnie Choy
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center Rochester, NY 14642, USA
    Int J Clin Exp Pathol 6:2542-7. 2013
  5. doi request reprint Combination of napsin A and TTF-1 immunohistochemistry helps in differentiating primary lung adenocarcinoma from metastatic carcinoma in the lung
    Jiqing Ye
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, Rochester, NY 14642, USA
    Appl Immunohistochem Mol Morphol 19:313-7. 2011
  6. doi request reprint hASH1 is a specific immunohistochemical marker for lung neuroendocrine tumors
    Bo Ye
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, Rochester, NY 14642 Department of Laboratory Medicine and Pathology, University of Minnesota, Dwan Variety Club Cardio vascular Research Center, Minneapolis, MN 55455
    Hum Pathol 48:142-7. 2016
  7. pmc Dynamic monitoring of oxidative DNA double-strand break and repair in cardiomyocytes
    Bo Ye
    Department of Laboratory Medicine and Pathology, University of Minnesota, Room 293, Dwan Variety Club Cardiovascular Research Center, 425 East River Parkway, Minneapolis, MN 55455, USA Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, 601 Elmwood Avenue, Rochester, NY 14642, USA
    Cardiovasc Pathol 25:93-100. 2016
  8. pmc Canonical Wnt/β-catenin signaling in epicardial fibrosis of failed pediatric heart allografts with diastolic dysfunction
    Bo Ye
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, Rochester, NY 14642, USA
    Cardiovasc Pathol 22:54-7. 2013
  9. pmc APC controls asymmetric Wnt/β-catenin signaling and cardiomyocyte proliferation gradient in the heart
    Bo Ye
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, Rochester, NY 14642, USA Department of Laboratory Medicine and Pathology, University of Minnesota, Room 293, Dwan Variety Club Cardiovascular Research Center, 425 E River Pkwy, Minneapolis, MN 55455, USA
    J Mol Cell Cardiol 89:287-96. 2015
  10. doi request reprint Androgen activates β-catenin signaling in bladder cancer cells
    Yi Li
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, Rochester, New York 14642, USA
    Endocr Relat Cancer 20:293-304. 2013

Collaborators

Detail Information

Publications13

  1. doi request reprint Small neuroendocrine lesions in intrathoracic lymph nodes of patients with primary lung adenocarcinoma: real metastasis?
    Faqian Li
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, Rochester, NC, USA
    Am J Surg Pathol 34:1701-7. 2010
    ..These findings suggest that neuroendocrine lesion can be incidentally identified in ILNs. Close clinical follow-up is warranted as metastasis from or synchronous lesions in adjacent organs cannot be excluded...
  2. doi request reprint Epigenetic modifications of histone h4 in lung neuroendocrine tumors
    Faqian Li
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, NY, USA
    Appl Immunohistochem Mol Morphol 19:389-94. 2011
    ..These histone modifications may serve as tumor biomarkers that may aid in diagnosis and prediction of clinical outcome...
  3. pmc Deletion of FoxO1 leads to shortening of QRS by increasing Na(+) channel activity through enhanced expression of both cardiac NaV1.5 and β3 subunit
    Benzhi Cai
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, Rochester, NY, USA
    J Mol Cell Cardiol 74:297-306. 2014
    ..5 and β3 subunit expressions as well as Na(+) channel activity in the heart and that FoxO1 is involved in the modulation of NaV1.5 expression in ischemic heart disease. ..
  4. pmc High frequency of coexpression of maspin with p63 and p53 in squamous cell carcinoma but not in adenocarcinoma of the lung
    Bonnie Choy
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center Rochester, NY 14642, USA
    Int J Clin Exp Pathol 6:2542-7. 2013
    ..Therapeutic targeting on maspin, p63 and p53 molecules might be beneficial in the management of patients with squamous cell carcinomas of the lung in the future. ..
  5. doi request reprint Combination of napsin A and TTF-1 immunohistochemistry helps in differentiating primary lung adenocarcinoma from metastatic carcinoma in the lung
    Jiqing Ye
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, Rochester, NY 14642, USA
    Appl Immunohistochem Mol Morphol 19:313-7. 2011
    ....
  6. doi request reprint hASH1 is a specific immunohistochemical marker for lung neuroendocrine tumors
    Bo Ye
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, Rochester, NY 14642 Department of Laboratory Medicine and Pathology, University of Minnesota, Dwan Variety Club Cardio vascular Research Center, Minneapolis, MN 55455
    Hum Pathol 48:142-7. 2016
    ..8 ± 18.0). Our findings indicate that hASH1 is a specific marker to distinguish neuroendocrine tumors from SCC and ADC. Additionally, hASH1 is a useful diagnostic marker for segregating SCLC from other neuroendocrine tumors. ..
  7. pmc Dynamic monitoring of oxidative DNA double-strand break and repair in cardiomyocytes
    Bo Ye
    Department of Laboratory Medicine and Pathology, University of Minnesota, Room 293, Dwan Variety Club Cardiovascular Research Center, 425 East River Parkway, Minneapolis, MN 55455, USA Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, 601 Elmwood Avenue, Rochester, NY 14642, USA
    Cardiovasc Pathol 25:93-100. 2016
    ..Our findings indicate that DSBs can be induced by H2O2 and ATM is the main kinase to mediate DSB repair in CMs. Therefore, monitoring DSB repair can assess oxidative injury and response in CMs. ..
  8. pmc Canonical Wnt/β-catenin signaling in epicardial fibrosis of failed pediatric heart allografts with diastolic dysfunction
    Bo Ye
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, Rochester, NY 14642, USA
    Cardiovasc Pathol 22:54-7. 2013
    ..In this study, we tested the hypothesis that canonical Wnt/β-catenin signaling is activated in epicardial fibrosis of end-stage dysfunctional pediatric allografts...
  9. pmc APC controls asymmetric Wnt/β-catenin signaling and cardiomyocyte proliferation gradient in the heart
    Bo Ye
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, Rochester, NY 14642, USA Department of Laboratory Medicine and Pathology, University of Minnesota, Room 293, Dwan Variety Club Cardiovascular Research Center, 425 E River Pkwy, Minneapolis, MN 55455, USA
    J Mol Cell Cardiol 89:287-96. 2015
    ..We determined if canonical Wnt signaling has directional and graded activity to maintain this CM proliferation gradient. Moreover, we investigated whether perturbation of Wnt signaling intensity could modulate CM proliferative activity...
  10. doi request reprint Androgen activates β-catenin signaling in bladder cancer cells
    Yi Li
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, Rochester, New York 14642, USA
    Endocr Relat Cancer 20:293-304. 2013
    ....
  11. pmc Reactive oxygen species suppress cardiac NaV1.5 expression through Foxo1
    Weike Mao
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, Rochester, New York, USA
    PLoS ONE 7:e32738. 2012
    ..These studies indicate that Foxo1 negatively regulates Na(V)1.5 expression in cardiomyocytes and reactive oxygen species suppress Na(V)1.5 expression through Foxo1...
  12. doi request reprint Down-regulation of cytoplasmic PLZF correlates with high tumor grade and tumor aggression in non-small cell lung carcinoma
    Guang Qian Xiao
    Department of Pathology, University of Rochester Medical Center, Rochester, NY 14642 Electronic address
    Hum Pathol 46:1607-15. 2015
    ..05). These results support a prognostic value for loss of cytoplasmic PLZF expression in the stratification of NSCLC and a possible role of cytoplasmic shift and down-regulation of PLZF in the pathogenesis of NSCLC. ..
  13. doi request reprint High-grade neuroendocrine carcinomas of the lung highly express enhancer of zeste homolog 2, but carcinoids do not
    Jennifer J Findeis-Hosey
    Department of Pathology and Laboratory Medicine, University of Rochester Medical Center, Rochester, NY 14642, USA
    Hum Pathol 42:867-72. 2011
    ....