Anyonya R Guntur

Summary

Affiliation: University of Texas Health Science Center
Country: USA

Publications

  1. pmc N-cadherin adherens junctions mediate osteogenesis through PI3K signaling
    Anyonya R Guntur
    Department of Biochemistry University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, TX 78229, USA
    Bone 50:54-62. 2012
  2. pmc Conditional ablation of Pten in osteoprogenitors stimulates FGF signaling
    Anyonya R Guntur
    Department of Biochemistry, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, TX 78229, USA
    Development 138:1433-44. 2011
  3. ncbi request reprint Glycogen synthase kinase 3 controls endochondral bone development: contribution of fibroblast growth factor 18
    Ravi M Kapadia
    Department of Pathology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Dr, San Antonio, TX 78229 3900, USA
    Dev Biol 285:496-507. 2005

Collaborators

  • Ravi M Kapadia
  • Martina I Reinhold
  • Michael C Naski

Detail Information

Publications3

  1. pmc N-cadherin adherens junctions mediate osteogenesis through PI3K signaling
    Anyonya R Guntur
    Department of Biochemistry University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, TX 78229, USA
    Bone 50:54-62. 2012
    ..PTEN deletion, on the other hand, increased the number of cells expressing N-cadherin in the perichondrium. These observations show that N-cadherin mediated junctions between osteoblasts are needed for osteoblast gene transcription...
  2. pmc Conditional ablation of Pten in osteoprogenitors stimulates FGF signaling
    Anyonya R Guntur
    Department of Biochemistry, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, TX 78229, USA
    Development 138:1433-44. 2011
    ..Significantly, deletion of FGFR2 caused a partial rescue of the Pten-null phenotype. This study identifies activated FGF signaling as the major mediator of Pten deletion in osteoprogenitors...
  3. ncbi request reprint Glycogen synthase kinase 3 controls endochondral bone development: contribution of fibroblast growth factor 18
    Ravi M Kapadia
    Department of Pathology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Dr, San Antonio, TX 78229 3900, USA
    Dev Biol 285:496-507. 2005
    ..Consistent with this, we showed that the effect of GSK3 inhibition on chondrocyte proliferation is repressed in tissues lacking a receptor for FGF18, FGF receptor 3...