Dawn Belt Davis

Summary

Affiliation: University of Wisconsin
Country: USA

Publications

  1. pmc FoxM1 is up-regulated by obesity and stimulates beta-cell proliferation
    Dawn Belt Davis
    Department of Medicine, University of Wisconsin, Madison, WI, USA
    Mol Endocrinol 24:1822-34. 2010
  2. pmc Cholecystokinin is up-regulated in obese mouse islets and expands beta-cell mass by increasing beta-cell survival
    Jeremy A Lavine
    Department of Biochemistry, University of Wisconsin, Madison, Wisconsin 53706, USA
    Endocrinology 151:3577-88. 2010
  3. pmc A gene expression network model of type 2 diabetes links cell cycle regulation in islets with diabetes susceptibility
    Mark P Keller
    Department of Biochemistry, University of Wisconsin, Madison, Wisconsin 53076, USA
    Genome Res 18:706-16. 2008
  4. pmc Tcf19 is a novel islet factor necessary for proliferation and survival in the INS-1 β-cell line
    Kimberly A Krautkramer
    Department of Medicine, Division of Endocrinology, Diabetes, and Metabolism, University of Wisconsin, Madison, Wisconsin
    Am J Physiol Endocrinol Metab 305:E600-10. 2013
  5. pmc Overexpression of pre-pro-cholecystokinin stimulates beta-cell proliferation in mouse and human islets with retention of islet function
    Jeremy A Lavine
    Department of Biochemistry, University of Wisconsin Madison, Madison, Wisconsin 53706, USA
    Mol Endocrinol 22:2716-28. 2008
  6. pmc Glucagon-Like Peptide-1 Regulates Cholecystokinin Production in β-Cells to Protect From Apoptosis
    Amelia K Linnemann
    Department of Medicine A K L, T J B, J A W, M E K, D B D, Division of Endocrinology, University of Wisconsin Madison, Madison, Wisconsin 53705 Department of Nutritional Sciences J C N, University of Wisconsin Madison, Madison, Wisconsin 53706 and William S Middleton Memorial Veterans Hospital D B D, Madison, Wisconsin 53705
    Mol Endocrinol 29:978-87. 2015
  7. pmc Transgenic expression of the human growth hormone minigene promotes pancreatic β-cell proliferation
    Mieke Baan
    Department of Medicine, Division of Endocrinology, University of Wisconsin Madison, Madison, Wisconsin and
    Am J Physiol Regul Integr Comp Physiol 309:R788-94. 2015
  8. pmc Attention to Background Strain Is Essential for Metabolic Research: C57BL/6 and the International Knockout Mouse Consortium
    Danielle A Fontaine
    Division of Endocrinology, Diabetes and Metabolism, Department of Medicine, University of Wisconsin Madison, Madison, WI
    Diabetes 65:25-33. 2016

Collaborators

  • Alan D Attie
  • Brian S Yandell
  • Eric E Schadt
  • Ping Wang
  • Christopher B Newgard
  • Luis A Fernandez
  • Jeremy A Lavine
  • Danielle A Fontaine
  • Mark P Keller
  • Amelia K Linnemann
  • Mary E Rabaglia
  • Mieke Baan
  • Ted W Harris
  • Michelle E Kimple
  • Kimberly A Krautkramer
  • Marc K Hellerstein
  • Philipp W Raess
  • Donald S Stapleton
  • Margery C Beinfeld
  • Justin R Bushkofsky
  • Dawn S Sherman
  • Therese J Battiola
  • Carly R Kibbe
  • Jaclyn A Wisinski
  • Joshua C Neuman
  • Leilani Marty-Santos
  • Amy L Whillock
  • Nathan A Truchan
  • Gregory J Schleis
  • Ondine Cleaver
  • James E Koltes
  • Linda C Samuelson
  • Joshua I Suhonen
  • Kathryn L Schueler
  • John A Dawson
  • Robert Kleinhanz
  • Carmen Argmann
  • Christina Kendziorski
  • Younjeong Choi
  • H Adam Steinberg
  • Alan S Kopin
  • Angie T Oler
  • Brent K Presley
  • Kathy L Schueler
  • Scott Turner
  • Elias Chaibub Neto
  • Steve Edwards

Detail Information

Publications8

  1. pmc FoxM1 is up-regulated by obesity and stimulates beta-cell proliferation
    Dawn Belt Davis
    Department of Medicine, University of Wisconsin, Madison, WI, USA
    Mol Endocrinol 24:1822-34. 2010
    ..Our data suggest that beta-cell proliferation occurs in adult obese humans in an attempt to expand beta-cell mass to compensate for insulin resistance, and that the FoxM1 transcriptional program plays a key role in this process...
  2. pmc Cholecystokinin is up-regulated in obese mouse islets and expands beta-cell mass by increasing beta-cell survival
    Jeremy A Lavine
    Department of Biochemistry, University of Wisconsin, Madison, Wisconsin 53706, USA
    Endocrinology 151:3577-88. 2010
    ..In summary, CCK is up-regulated by islet cells during obesity and functions as a paracrine or autocrine factor to increase beta-cell survival and expand beta-cell mass to compensate for obesity-induced insulin resistance...
  3. pmc A gene expression network model of type 2 diabetes links cell cycle regulation in islets with diabetes susceptibility
    Mark P Keller
    Department of Biochemistry, University of Wisconsin, Madison, Wisconsin 53076, USA
    Genome Res 18:706-16. 2008
    ....
  4. pmc Tcf19 is a novel islet factor necessary for proliferation and survival in the INS-1 β-cell line
    Kimberly A Krautkramer
    Department of Medicine, Division of Endocrinology, Diabetes, and Metabolism, University of Wisconsin, Madison, Wisconsin
    Am J Physiol Endocrinol Metab 305:E600-10. 2013
    ..Therefore, Tcf19 is necessary for both proliferation and survival and is a novel regulator of these pathways...
  5. pmc Overexpression of pre-pro-cholecystokinin stimulates beta-cell proliferation in mouse and human islets with retention of islet function
    Jeremy A Lavine
    Department of Biochemistry, University of Wisconsin Madison, Madison, Wisconsin 53706, USA
    Mol Endocrinol 22:2716-28. 2008
    ..CCK triggers beta-cell proliferation without disrupting islet function, up-regulates a distinct set of cell cycle regulators in islets, and signals independently of the CCK receptors...
  6. pmc Glucagon-Like Peptide-1 Regulates Cholecystokinin Production in β-Cells to Protect From Apoptosis
    Amelia K Linnemann
    Department of Medicine A K L, T J B, J A W, M E K, D B D, Division of Endocrinology, University of Wisconsin Madison, Madison, Wisconsin 53705 Department of Nutritional Sciences J C N, University of Wisconsin Madison, Madison, Wisconsin 53706 and William S Middleton Memorial Veterans Hospital D B D, Madison, Wisconsin 53705
    Mol Endocrinol 29:978-87. 2015
    ..This intraislet incretin loop may be one mechanism whereby GLP-1 protects β-cells from apoptosis...
  7. pmc Transgenic expression of the human growth hormone minigene promotes pancreatic β-cell proliferation
    Mieke Baan
    Department of Medicine, Division of Endocrinology, University of Wisconsin Madison, Madison, Wisconsin and
    Am J Physiol Regul Integr Comp Physiol 309:R788-94. 2015
    ..Our work suggests that each will need to be carefully screened for inadvertent hGH production and critically evaluated for the use of proper controls. ..
  8. pmc Attention to Background Strain Is Essential for Metabolic Research: C57BL/6 and the International Knockout Mouse Consortium
    Danielle A Fontaine
    Division of Endocrinology, Diabetes and Metabolism, Department of Medicine, University of Wisconsin Madison, Madison, WI
    Diabetes 65:25-33. 2016
    ..Our aim is to increase awareness of this issue in the diabetes research community and to provide practical strategies to enable researchers to avoid mixed strain animals when using IKMC knockout mice. ..