Antonei B Csoka
Affiliation: University of Pittsburgh
- Genome-scale expression profiling of Hutchinson-Gilford progeria syndrome reveals widespread transcriptional misregulation leading to mesodermal/mesenchymal defects and accelerated atherosclerosisAntonei B Csoka
Department of Molecular Biology, Cell Biology and Biochemistry, Brown University, Providence, RI 02912, USA
Aging Cell 3:235-43. 2004..The identification of a large number of genes implicated in atherosclerosis is especially valuable, because it provides clues to pathological processes that can now be investigated in HGPS patients or animal models...
- Recurrent de novo point mutations in lamin A cause Hutchinson-Gilford progeria syndromeMaria Eriksson
Department of Human Genetics, New York State Institute for Basic Research in Developmental Disabilities, Staten Island, New York 10314, USA
Nature 423:293-8. 2003..The discovery of the molecular basis of this disease may shed light on the general phenomenon of human ageing...
- Lamin A/C expression is a marker of mouse and human embryonic stem cell differentiationDan Constantinescu
Department of Cell Biology and Physiology, University of Pittsburgh School of Medicine, Pittsburgh Development Center of Magee Womens Research Institute, 204 Craft Ave, Pittsburgh, Pennsylvania 15213, USA
Stem Cells 24:177-85. 2006..Our results identify the absence of A-type lamin expression as a novel marker for undifferentiated ES cells and further support a role for nuclear lamins in cell maintenance and differentiation...