Affiliation: Tulane University
- Interleukin-10 anti-inflammatory response to Borrelia burgdorferi, the agent of Lyme disease: a possible role for suppressors of cytokine signaling 1 and 3Vida A Dennis
Division of Bacteriology and Parasitology, Tulane National Primate Research Center, 18703 Three Rivers Rd, Covington, LA 70433, USA
Infect Immun 74:5780-9. 2006..Our data are evidence to suggest that expression of SOCS is part of the mechanism of IL-10-mediated inhibition of inflammatory cytokines elicited by B. burgdorferi and its lipoproteins...
- Live Borrelia burgdorferi spirochetes elicit inflammatory mediators from human monocytes via the Toll-like receptor signaling pathwayVida A Dennis
Division of Bacteriology and Parasitology, Tulane National Primate Research Center, Covington, Louisiana 70433, USA
Infect Immun 77:1238-45. 2009..burgdorferi spirochetes and furthermore suggest that the TLR-dependent interaction between these cells and live spirochetes is mediated by spirochetal lipoproteins but not by flagellin...
- Suppressor of Cytokine Signaling and Control of Inflammation in Lyme DiseaseVIDA DENNIS; Fiscal Year: 2007..Achieving this proposal's goal will explain how inflammation is regulated, and help understand the basis of susceptibility and resistance to Lyme disease. ..