Affiliation: State University of New York
- Tbx5 is essential for heart developmentM E Horb
Department of Biochemistry and Cell Biology, Institute of Cell and Developmental Biology, State University of New York, Stony Brook, NY 11794 5215, USA
Development 126:1739-51. 1999..These results suggest that, in addition to its function in heart septation, Tbx5 has a more global role in cardiac specification and heart development in vertebrate embryos...
- Smurf1 regulates neural patterning and folding in Xenopus embryos by antagonizing the BMP/Smad1 pathwayEvguenia M Alexandrova
Department of Biochemistry and Cell Biology, Graduate Program in Molecular and Cellular Biology, Center for Developmental Genetics, Stony Brook University, Stony Brook, NY 11794 5215, USA
Dev Biol 299:398-410. 2006..Our data also reveal a novel role for Smurf1 and Smad1 in neural plate morphogenesis...
- The ARID domain protein dril1 is necessary for TGF(beta) signaling in Xenopus embryosElizabeth M Callery
Department of Biochemistry and Cell Biology and Center for Developmental Genetics, Stony Brook University, Stony Brook, NY 11794 5215, USA
Dev Biol 278:542-59. 2005..These experiments identify dril1 as a novel regulator of TGF(beta) signaling and a vital component of mesodermal patterning and embryonic morphogenesis...
- A new century of amphibian developmental biologyGerald H Thomsen
Semin Cell Dev Biol 17:78-9. 2006
- The Hedgehog gene family of the cnidarian, Nematostella vectensis, and implications for understanding metazoan Hedgehog pathway evolutionDavid Q Matus
Kewalo Marine Lab University of Hawaii, Honolulu, HI 76813, USA
Dev Biol 313:501-18. 2008....
- Smurf Ubiquitin Ligases in Xenopus DevelopmentGERALD THOMSEN; Fiscal Year: 2005..We have already isolated 89 Smurf1-interacting clones, including Smad1, a known Smurf1 target. ..
- TRAF4 in TGF-beta Signaling and Embryonic DevelopmentGERALD THOMSEN; Fiscal Year: 2009....
- Regulation of TGF-beta Signaling and Embryonic Development by GTPasesGerald H Thomsen; Fiscal Year: 2010..Our studies will provide crucial information about GTPBPs which may eventually translate into clinical treatments for diseases caused by abnormal TGF[unreadable] or GTPBP signaling. ..