C Rabadan-Diehl

Summary

Affiliation: National Institutes of Health
Country: USA

Publications

  1. ncbi request reprint Isolation and characterization of the promoter region of the rat vasopressin V1b receptor gene
    C Rabadan-Diehl
    Section on Endocrine Physiology, Developmental Endocrinology Branch, National Institute of Child Health and Human Development, NIMH, NIH, Bethesda, MD 20892 1862, USA
    J Neuroendocrinol 12:437-44. 2000
  2. ncbi request reprint Glucocorticoids increase vasopressin V1b receptor coupling to phospholipase C
    C Rabadan-Diehl
    Section on Endocrine, Physiology, Developmental Endocrinology Branch, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20892, USA
    Endocrinology 139:3220-6. 1998
  3. ncbi request reprint Vasopressinergic regulation of the hypothalamic-pituitary-adrenal axis: implications for stress adaptation
    G Aguilera
    Section on Endocrine Physiology, Developmental Endocrinology Branch, National Institute of Child Health and Human Development, NIH, Bldg 10, Rm 10N262, 10 Center Drive MSC 1862, Bethesda, MD 20892 1862, USA
    Regul Pept 96:23-9. 2000
  4. ncbi request reprint Regulation of pituitary corticotropin releasing hormone receptors
    G Aguilera
    Section on Endocrine Physiology, Developmental Endocrinology Branch, National Institute of Child Health and Human Development, NIH, Bethesda, Maryland 20892, USA
    Peptides 22:769-74. 2001
  5. ncbi request reprint Transcriptional and post-transcriptional mechanisms regulating the rat pituitary vasopressin V1b receptor gene
    G Aguilera
    Developmental Endocrinology Branch, National Institutes of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20892, USA
    J Mol Endocrinol 30:99-108. 2003
  6. ncbi request reprint Regulation of vasopressin V1b receptors in the anterior pituitary gland of the rat
    G Aguilera
    Section on Endocrine Physiology, Developmental Endocrinology Branch, National Institute of Child Health and Human Development, NIH, Bethesda, MD, USA
    Exp Physiol 85:19S-26S. 2000
  7. ncbi request reprint Inhibition of corticotropin releasing hormone type-1 receptor translation by an upstream AUG triplet in the 5' untranslated region
    G Xu
    Section on Endocrine Physiology, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland, USA
    Mol Pharmacol 59:485-92. 2001
  8. ncbi request reprint Inhibition of vasopressin V1b receptor translation by upstream open reading frames in the 5'-untranslated region
    C Rabadan-Diehl
    Section on Endocrine Physiology, Developmental Endocrinology Branch, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, MD 20892, USA
    J Neuroendocrinol 19:309-19. 2007

Collaborators

Detail Information

Publications8

  1. ncbi request reprint Isolation and characterization of the promoter region of the rat vasopressin V1b receptor gene
    C Rabadan-Diehl
    Section on Endocrine Physiology, Developmental Endocrinology Branch, National Institute of Child Health and Human Development, NIMH, NIH, Bethesda, MD 20892 1862, USA
    J Neuroendocrinol 12:437-44. 2000
    ..The data provide bases for future analysis of the regulatory elements controlling V1b receptor transcription...
  2. ncbi request reprint Glucocorticoids increase vasopressin V1b receptor coupling to phospholipase C
    C Rabadan-Diehl
    Section on Endocrine, Physiology, Developmental Endocrinology Branch, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20892, USA
    Endocrinology 139:3220-6. 1998
    ..The data demonstrate that glucocorticoids increase coupling of the V1b-R with PLC thereby providing a mechanism by which VP facilitates corticotroph responsiveness in spite of elevated levels of plasma glucocorticoids during stress...
  3. ncbi request reprint Vasopressinergic regulation of the hypothalamic-pituitary-adrenal axis: implications for stress adaptation
    G Aguilera
    Section on Endocrine Physiology, Developmental Endocrinology Branch, National Institute of Child Health and Human Development, NIH, Bldg 10, Rm 10N262, 10 Center Drive MSC 1862, Bethesda, MD 20892 1862, USA
    Regul Pept 96:23-9. 2000
    ..The data suggest that vasopressinergic regulation of the HPA axis is critical for sustaining corticotroph responsiveness in the presence of high circulating glucocorticoid levels during chronic stress...
  4. ncbi request reprint Regulation of pituitary corticotropin releasing hormone receptors
    G Aguilera
    Section on Endocrine Physiology, Developmental Endocrinology Branch, National Institute of Child Health and Human Development, NIH, Bethesda, Maryland 20892, USA
    Peptides 22:769-74. 2001
    ..The lack of correlation between CRH binding and CRH1 receptor mRNA indicates that the major sites for pituitary CRH1 receptor regulation are at the post-transcriptional level...
  5. ncbi request reprint Transcriptional and post-transcriptional mechanisms regulating the rat pituitary vasopressin V1b receptor gene
    G Aguilera
    Developmental Endocrinology Branch, National Institutes of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20892, USA
    J Mol Endocrinol 30:99-108. 2003
    ....
  6. ncbi request reprint Regulation of vasopressin V1b receptors in the anterior pituitary gland of the rat
    G Aguilera
    Section on Endocrine Physiology, Developmental Endocrinology Branch, National Institute of Child Health and Human Development, NIH, Bethesda, MD, USA
    Exp Physiol 85:19S-26S. 2000
    ..In addition, the recent characterization of a major part of the V1b receptor gene provides a basis for studying the molecular mechanisms regulating the V1b receptor...
  7. ncbi request reprint Inhibition of corticotropin releasing hormone type-1 receptor translation by an upstream AUG triplet in the 5' untranslated region
    G Xu
    Section on Endocrine Physiology, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland, USA
    Mol Pharmacol 59:485-92. 2001
    ..The study shows that the upstream AUG in 5'-UTR of CRHR1 mRNA inhibits receptor expression by inhibiting mRNA translation and suggests the short open reading frame in the 5'-UTR plays a role in regulating translation of the CRH receptor...
  8. ncbi request reprint Inhibition of vasopressin V1b receptor translation by upstream open reading frames in the 5'-untranslated region
    C Rabadan-Diehl
    Section on Endocrine Physiology, Developmental Endocrinology Branch, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, MD 20892, USA
    J Neuroendocrinol 19:309-19. 2007
    ..The present study shows that uORFs in the 5'-UTR of the V1bR mRNA inhibit V1bR translation, and suggests that translation of a 38-amino acid membrane peptide encoded by uORF1 exerts tonic inhibition of V1bR translation...