Basil A Eldadah

Summary

Affiliation: National Institutes of Health
Country: USA

Publications

  1. ncbi request reprint Cardiac uptake-1 inhibition by high circulating norepinephrine levels in patients with pheochromocytoma
    Basil A Eldadah
    Clinical Neurocardiology Section, National Institute of Neurological Disorders and Stroke, National Institutes of Health, 10 Center Drive, MSC 1620, Bethesda, MD 20892 1620, USA
    Hypertension 43:1227-32. 2004
  2. doi request reprint Attenuated pre-ejection period response to tyramine in patients with cardiac sympathetic denervation
    Richard Imrich
    Clinical Neurocardiology Section, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, Maryland, USA
    Ann N Y Acad Sci 1148:486-9. 2008
  3. pmc Functional effects of cardiac sympathetic denervation in neurogenic orthostatic hypotension
    Richard Imrich
    National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, MD 20892, USA
    Parkinsonism Relat Disord 15:122-7. 2009
  4. pmc Biomarkers to detect central dopamine deficiency and distinguish Parkinson disease from multiple system atrophy
    David S Goldstein
    Clinical Neurocardiology Section, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, MD 20892 1620, USA
    Parkinsonism Relat Disord 14:600-7. 2008
  5. pmc Supine low-frequency power of heart rate variability reflects baroreflex function, not cardiac sympathetic innervation
    Jeffrey P Moak
    Children s National Medical Center, Washington, DC, USA
    Heart Rhythm 4:1523-9. 2007
  6. ncbi request reprint Supine low-frequency power of heart rate variability reflects baroreflex function, not cardiac sympathetic innervation
    Jeffrey P Moak
    Children s National Medical Center, Washington, DC, USA
    Cleve Clin J Med 76:S51-9. 2009
  7. doi request reprint Fatigue and fatigability in older adults
    Basil A Eldadah
    Division of Geriatrics and Clinical Gerontology, National Institute on Aging, 7201 Wisconsin Avenue, Bethesda, MD 20892 9205, USA
    PM R 2:406-13. 2010
  8. ncbi request reprint Neurocirculatory abnormalities in Parkinson disease with orthostatic hypotension: independence from levodopa treatment
    David S Goldstein
    Clinical Neurocardiology Section, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, MD 20892 1620, USA
    Hypertension 46:1333-9. 2005
  9. pmc Autoimmune autonomic ganglionopathy: treatment by plasma exchanges and rituximab
    Richard Imrich
    Clinical Neurocardiology Section, National Institute of Neurological Disorders and Stroke, Bethesda, MD 20892 1620, USA
    Clin Auton Res 19:259-62. 2009
  10. ncbi request reprint Failure of propranolol to prevent tilt-evoked systemic vasodilatation, adrenaline release and neurocardiogenic syncope
    Basil A Eldadah
    Clinical Neurocardiology Section, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, MD 20892 1620, USA
    Clin Sci (Lond) 111:209-16. 2006

Detail Information

Publications11

  1. ncbi request reprint Cardiac uptake-1 inhibition by high circulating norepinephrine levels in patients with pheochromocytoma
    Basil A Eldadah
    Clinical Neurocardiology Section, National Institute of Neurological Disorders and Stroke, National Institutes of Health, 10 Center Drive, MSC 1620, Bethesda, MD 20892 1620, USA
    Hypertension 43:1227-32. 2004
    ..The results call for caution in drawing quantitative conclusions about uptake-1 in the setting of high circulating concentrations of endogenous norepinephrine...
  2. doi request reprint Attenuated pre-ejection period response to tyramine in patients with cardiac sympathetic denervation
    Richard Imrich
    Clinical Neurocardiology Section, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, Maryland, USA
    Ann N Y Acad Sci 1148:486-9. 2008
    ..05). One of the functional consequences of cardiac sympathetic denervation is failure to increase contractility in response to stimuli that depend on endogenous norepinephrine release...
  3. pmc Functional effects of cardiac sympathetic denervation in neurogenic orthostatic hypotension
    Richard Imrich
    National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, MD 20892, USA
    Parkinsonism Relat Disord 15:122-7. 2009
    ..We studied the impact of such denervation on cardiac chronotropic and inotropic function...
  4. pmc Biomarkers to detect central dopamine deficiency and distinguish Parkinson disease from multiple system atrophy
    David S Goldstein
    Clinical Neurocardiology Section, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, MD 20892 1620, USA
    Parkinsonism Relat Disord 14:600-7. 2008
    ..This study compared neuroimaging, neurochemical, and olfactory potential biomarkers to detect central dopamine (DA) deficiency and distinguish PD from multiple system atrophy (MSA)...
  5. pmc Supine low-frequency power of heart rate variability reflects baroreflex function, not cardiac sympathetic innervation
    Jeffrey P Moak
    Children s National Medical Center, Washington, DC, USA
    Heart Rhythm 4:1523-9. 2007
    ..Alternatively, LF power might reflect baroreflexive modulation of autonomic outflows...
  6. ncbi request reprint Supine low-frequency power of heart rate variability reflects baroreflex function, not cardiac sympathetic innervation
    Jeffrey P Moak
    Children s National Medical Center, Washington, DC, USA
    Cleve Clin J Med 76:S51-9. 2009
    ..BRS at baseline predicted LF responses to tyramine and yohimbine. CONCLUSION: LF power reflects baroreflex function, not cardiac sympathetic innervation...
  7. doi request reprint Fatigue and fatigability in older adults
    Basil A Eldadah
    Division of Geriatrics and Clinical Gerontology, National Institute on Aging, 7201 Wisconsin Avenue, Bethesda, MD 20892 9205, USA
    PM R 2:406-13. 2010
    ..Because fatigue may represent a physiologic warning system, future clinical studies may benefit from a focus on fatigability, where both symptoms and function are considered...
  8. ncbi request reprint Neurocirculatory abnormalities in Parkinson disease with orthostatic hypotension: independence from levodopa treatment
    David S Goldstein
    Clinical Neurocardiology Section, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, MD 20892 1620, USA
    Hypertension 46:1333-9. 2005
    ..Parkinson disease with orthostatic hypotension features reflexive cardiovagal and sympathoneural failure and cardiac and partial extracardiac sympathetic denervation, independent of levodopa treatment...
  9. pmc Autoimmune autonomic ganglionopathy: treatment by plasma exchanges and rituximab
    Richard Imrich
    Clinical Neurocardiology Section, National Institute of Neurological Disorders and Stroke, Bethesda, MD 20892 1620, USA
    Clin Auton Res 19:259-62. 2009
    ....
  10. ncbi request reprint Failure of propranolol to prevent tilt-evoked systemic vasodilatation, adrenaline release and neurocardiogenic syncope
    Basil A Eldadah
    Clinical Neurocardiology Section, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, MD 20892 1620, USA
    Clin Sci (Lond) 111:209-16. 2006
    ..In this small, but tightly controlled, study, propranolol did not prevent tilt-induced vasodilatation, syncope or elevated plasma adrenaline...
  11. ncbi request reprint Coronary vascular resistance in primary chronic autonomic failure
    Sheng Ting Li
    Clin Auton Res 16:293-5. 2006
    ..Cardiac sympathetic outflow does not seem to contribute to coronary vascular resistance...