Daniel S Chertow

Summary

Affiliation: National Institutes of Health
Country: USA

Publications

  1. doi request reprint Influenza A and methicillin-resistant Staphylococcus aureus co-infection in rhesus macaques - A model of severe pneumonia
    Daniel S Chertow
    Critical Care Medicine Department, Clinical Center, National Institutes of Health, Bethesda, MD, USA Viral Pathogenesis and Evolution Section, Laboratory of Infectious Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD, USA Electronic address
    Antiviral Res 129:120-9. 2016
  2. doi request reprint Bacterial coinfection in influenza: a grand rounds review
    Daniel S Chertow
    Critical Care Medicine Department, Clinical Center, National Institutes of Health, 10 Center Dr, Room 2C145, Bethesda, MD 20892 1662, USA
    JAMA 309:275-82. 2013
  3. pmc 1918 Influenza receptor binding domain variants bind and replicate in primary human airway cells regardless of receptor specificity
    A Sally Davis
    Viral Pathogenesis and Evolution Section, National Institute of Allergy and Infectious Diseases NIAID, National Institutes of Health NIH, Bethesda, MD, United States Diagnostic Medicine and Pathobiology, Kansas State University College of Veterinary Medicine, Manhattan, KS, United States
    Virology 493:238-46. 2016
  4. pmc Treatment with the reactive oxygen species scavenger EUK-207 reduces lung damage and increases survival during 1918 influenza virus infection in mice
    John C Kash
    Laboratory of Infectious Diseases, National Institutes of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892, USA Electronic address
    Free Radic Biol Med 67:235-47. 2014
  5. pmc Contemporary avian influenza A virus subtype H1, H6, H7, H10, and H15 hemagglutinin genes encode a mammalian virulence factor similar to the 1918 pandemic virus H1 hemagglutinin
    Li Qi
    Viral Pathogenesis and Evolution Section, Laboratory of Infectious Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland, USA
    MBio 5:e02116. 2014
  6. pmc Autopsy series of 68 cases dying before and during the 1918 influenza pandemic peak
    Zong Mei Sheng
    Viral Pathogenesis and Evolution Section, Laboratory of Infectious Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892, USA
    Proc Natl Acad Sci U S A 108:16416-21. 2011
  7. pmc Validation of normal human bronchial epithelial cells as a model for influenza A infections in human distal trachea
    A Sally Davis
    Viral Pathogenesis and Evolution Section, National Institutes of Allergy and Infectious Diseases NIAID, National Institutes of Health NIH, Bethesda, Maryland ASD, DSC, JEM, As, JKT Diagnostic Medicine Pathobiology, Kansas State University College of Veterinary Medicine, Manhattan, Kansas ASD Critical Care Medicine Department, Clinical Center, NIH, Bethesda, Maryland DSC, JS, CL, JHS Electron Microscopy Unit, Research Technology Branch, NIAID, Hamilton, Montana DWD
    J Histochem Cytochem 63:312-28. 2015
  8. pmc Multidrug-resistant 2009 pandemic influenza A(H1N1) viruses maintain fitness and transmissibility in ferrets
    Matthew J Memoli
    Viral Pathogenesis and Evolution Section, Laboratory of Infectious Diseases, National Institutes of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland 20892, USA
    J Infect Dis 203:348-57. 2011
  9. pmc Lethal synergism of 2009 pandemic H1N1 influenza virus and Streptococcus pneumoniae coinfection is associated with loss of murine lung repair responses
    John C Kash
    Viral Pathogenesis and Evolution Section, Laboratory of Infectious Diseases, National Institutes of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland, USA
    MBio 2:. 2011
  10. ncbi request reprint Integration of Global Analyses of Host Molecular Responses with Clinical Data To Evaluate Pathogenesis and Advance Therapies for Emerging and Re-emerging Viral Infections
    Shane D Falcinelli
    Critical Care Medicine Department, Clinical Center, National Institutes of Health, Bethesda, Maryland 20814, United States
    ACS Infect Dis 2:787-799. 2016

Collaborators

  • Jeffery K Taubenberger
  • John C Kash
  • Matthew J Memoli
  • David M Morens
  • Olga A Maximova
  • A Sally Davis
  • Li Qi
  • Jason Kindrachuk
  • Shane D Falcinelli
  • James H Shelhamer
  • Carolea Logun
  • Jon Suzich
  • Louis M Schwartzman
  • Zong Mei Sheng
  • Sara Alsaaty
  • Jenna E Moyer
  • David W Dorward
  • Aline Sandouk
  • Kevan L Hartshorn
  • Richard D Slemons
  • Lindsey M Pujanauski
  • David Baxter
  • Kathie Anne Walters
  • Kelsey Scherler
  • Sherman McCall
  • Ronald M Przygodzki
  • Robert E Cunningham
  • XAVIER AMBROGGIO

Detail Information

Publications10

  1. doi request reprint Influenza A and methicillin-resistant Staphylococcus aureus co-infection in rhesus macaques - A model of severe pneumonia
    Daniel S Chertow
    Critical Care Medicine Department, Clinical Center, National Institutes of Health, Bethesda, MD, USA Viral Pathogenesis and Evolution Section, Laboratory of Infectious Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD, USA Electronic address
    Antiviral Res 129:120-9. 2016
    ..Preclinical models of severe influenza infection that closely correlate to human disease are needed to assess efficacy of new vaccines and therapeutics...
  2. doi request reprint Bacterial coinfection in influenza: a grand rounds review
    Daniel S Chertow
    Critical Care Medicine Department, Clinical Center, National Institutes of Health, 10 Center Dr, Room 2C145, Bethesda, MD 20892 1662, USA
    JAMA 309:275-82. 2013
    ..Complex viral, bacterial, and host factors contribute to the pathogenesis of coinfection. Reductions in morbidity and mortality are dependent on prevention with available vaccines as well as early diagnosis and treatment...
  3. pmc 1918 Influenza receptor binding domain variants bind and replicate in primary human airway cells regardless of receptor specificity
    A Sally Davis
    Viral Pathogenesis and Evolution Section, National Institute of Allergy and Infectious Diseases NIAID, National Institutes of Health NIH, Bethesda, MD, United States Diagnostic Medicine and Pathobiology, Kansas State University College of Veterinary Medicine, Manhattan, KS, United States
    Virology 493:238-46. 2016
    ..We observed that all five variants bound and entered cells efficiently and that Sia preference did not predict entry of influenza A virus to primary human airway cells evaluated in this model. ..
  4. pmc Treatment with the reactive oxygen species scavenger EUK-207 reduces lung damage and increases survival during 1918 influenza virus infection in mice
    John C Kash
    Laboratory of Infectious Diseases, National Institutes of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892, USA Electronic address
    Free Radic Biol Med 67:235-47. 2014
    ....
  5. pmc Contemporary avian influenza A virus subtype H1, H6, H7, H10, and H15 hemagglutinin genes encode a mammalian virulence factor similar to the 1918 pandemic virus H1 hemagglutinin
    Li Qi
    Viral Pathogenesis and Evolution Section, Laboratory of Infectious Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland, USA
    MBio 5:e02116. 2014
    ..Consequently, zoonotic infections with avian influenza viruses bearing one of these hemagglutinins may cause enhanced disease in mammals...
  6. pmc Autopsy series of 68 cases dying before and during the 1918 influenza pandemic peak
    Zong Mei Sheng
    Viral Pathogenesis and Evolution Section, Laboratory of Infectious Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892, USA
    Proc Natl Acad Sci U S A 108:16416-21. 2011
    ..These findings have important implications for understanding the origins and evolution of pandemic influenza viruses...
  7. pmc Validation of normal human bronchial epithelial cells as a model for influenza A infections in human distal trachea
    A Sally Davis
    Viral Pathogenesis and Evolution Section, National Institutes of Allergy and Infectious Diseases NIAID, National Institutes of Health NIH, Bethesda, Maryland ASD, DSC, JEM, As, JKT Diagnostic Medicine Pathobiology, Kansas State University College of Veterinary Medicine, Manhattan, Kansas ASD Critical Care Medicine Department, Clinical Center, NIH, Bethesda, Maryland DSC, JS, CL, JHS Electron Microscopy Unit, Research Technology Branch, NIAID, Hamilton, Montana DWD
    J Histochem Cytochem 63:312-28. 2015
    ..Both indicated that the pattern of infection progression in these cells correlated with autopsy studies of fatal cases from the 2009 pandemic...
  8. pmc Multidrug-resistant 2009 pandemic influenza A(H1N1) viruses maintain fitness and transmissibility in ferrets
    Matthew J Memoli
    Viral Pathogenesis and Evolution Section, Laboratory of Infectious Diseases, National Institutes of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland 20892, USA
    J Infect Dis 203:348-57. 2011
    ....
  9. pmc Lethal synergism of 2009 pandemic H1N1 influenza virus and Streptococcus pneumoniae coinfection is associated with loss of murine lung repair responses
    John C Kash
    Viral Pathogenesis and Evolution Section, Laboratory of Infectious Diseases, National Institutes of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, Maryland, USA
    MBio 2:. 2011
    ..Moreover, this study provides novel insights into influenza virus and bacterial coinfection by showing correlation of lethal outcome with loss of airway basal epithelial cells and associated lung repair responses...
  10. ncbi request reprint Integration of Global Analyses of Host Molecular Responses with Clinical Data To Evaluate Pathogenesis and Advance Therapies for Emerging and Re-emerging Viral Infections
    Shane D Falcinelli
    Critical Care Medicine Department, Clinical Center, National Institutes of Health, Bethesda, Maryland 20814, United States
    ACS Infect Dis 2:787-799. 2016
    ..Lastly, we will address how increased integration of clinical and basic research will assist treatment and prevention efforts for emerging pathogens...