Yi Chieh Nancy Du

Summary

Affiliation: Memorial Sloan-Kettering Cancer Center
Country: USA

Publications

  1. pmc Assessing tumor progression factors by somatic gene transfer into a mouse model: Bcl-xL promotes islet tumor cell invasion
    Yi Chieh Nancy Du
    Program in Cancer Biology and Genetics, Memorial Sloan Kettering Cancer Center, New York, New York, USA
    PLoS Biol 5:e276. 2007
  2. pmc Activation of PyMT in beta cells induces irreversible hyperplasia, but oncogene-dependent acinar cell carcinomas when activated in pancreatic progenitors
    Yi Chieh Nancy Du
    Program in Cancer Biology and Genetics, Memorial Sloan Kettering Cancer Center, New York, New York, USA
    PLoS ONE 4:e6932. 2009
  3. pmc Seeding and propagation of untransformed mouse mammary cells in the lung
    Katrina Podsypanina
    Program in Cancer Biology and Genetics, Memorial Sloan Kettering Cancer Center, New York, NY 10021, USA
    Science 321:1841-4. 2008
  4. pmc Receptor for hyaluronan-mediated motility isoform B promotes liver metastasis in a mouse model of multistep tumorigenesis and a tail vein assay for metastasis
    Yi Chieh Nancy Du
    Program in Cancer Biology and Genetics, Memorial Sloan Kettering Cancer Center, New York, NY 10065, USA
    Proc Natl Acad Sci U S A 108:16753-8. 2011
  5. pmc Bcl-xL promotes metastasis independent of its anti-apoptotic activity
    Soyoung Choi
    Department of Pathology and Laboratory Medicine, Weill Cornell Medicine, New York, New York 10065, USA
    Nat Commun 7:10384. 2016
  6. doi Attenuation of the retinoblastoma pathway in pancreatic neuroendocrine tumors due to increased cdk4/cdk6
    Laura H Tang
    Departments of Pathology and Surgery, Memorial Sloan Kettering Cancer Center, Weill Cornell Medical College, New York, New York, USA
    Clin Cancer Res 18:4612-20. 2012
  7. pmc p53 and p16Ink4a/p19Arf Loss Promotes Different Pancreatic Tumor Types from PyMT-Expressing Progenitor Cells
    Stephanie Azzopardi
    Department of Pathology and Laboratory Medicine, Weill Cornell Medicine, New York, NY 10065, USA
    Neoplasia 18:610-617. 2016

Detail Information

Publications7

  1. pmc Assessing tumor progression factors by somatic gene transfer into a mouse model: Bcl-xL promotes islet tumor cell invasion
    Yi Chieh Nancy Du
    Program in Cancer Biology and Genetics, Memorial Sloan Kettering Cancer Center, New York, New York, USA
    PLoS Biol 5:e276. 2007
    ..In addition, myosin Va was identified as a novel Bcl-xL-interacting protein that might mediate the effects of Bcl-xL on tumor cell migration and invasion...
  2. pmc Activation of PyMT in beta cells induces irreversible hyperplasia, but oncogene-dependent acinar cell carcinomas when activated in pancreatic progenitors
    Yi Chieh Nancy Du
    Program in Cancer Biology and Genetics, Memorial Sloan Kettering Cancer Center, New York, New York, USA
    PLoS ONE 4:e6932. 2009
    ....
  3. pmc Seeding and propagation of untransformed mouse mammary cells in the lung
    Katrina Podsypanina
    Program in Cancer Biology and Genetics, Memorial Sloan Kettering Cancer Center, New York, NY 10021, USA
    Science 321:1841-4. 2008
    ..Mammary cells lacking oncogenic transgenes displayed a similar capacity for long-term residence in the lungs but did not form ectopic tumors...
  4. pmc Receptor for hyaluronan-mediated motility isoform B promotes liver metastasis in a mouse model of multistep tumorigenesis and a tail vein assay for metastasis
    Yi Chieh Nancy Du
    Program in Cancer Biology and Genetics, Memorial Sloan Kettering Cancer Center, New York, NY 10065, USA
    Proc Natl Acad Sci U S A 108:16753-8. 2011
    ..Taken together, the results indicate that RHAMM(B) promotes hepatic metastasis by islet tumor cells, perhaps through growth factor receptor-mediated signaling...
  5. pmc Bcl-xL promotes metastasis independent of its anti-apoptotic activity
    Soyoung Choi
    Department of Pathology and Laboratory Medicine, Weill Cornell Medicine, New York, New York 10065, USA
    Nat Commun 7:10384. 2016
    ..Consistent with these findings, we detect nuclear Bcl-xL in human metastatic panNETs. Taken together, the metastatic function of Bcl-xL is independent of its anti-apoptotic activity and its residence in the mitochondria. ..
  6. doi Attenuation of the retinoblastoma pathway in pancreatic neuroendocrine tumors due to increased cdk4/cdk6
    Laura H Tang
    Departments of Pathology and Surgery, Memorial Sloan Kettering Cancer Center, Weill Cornell Medical College, New York, New York, USA
    Clin Cancer Res 18:4612-20. 2012
    ..In this study, we took a closer look at the role of the retinoblastoma pathway in human Pan-NETs...
  7. pmc p53 and p16Ink4a/p19Arf Loss Promotes Different Pancreatic Tumor Types from PyMT-Expressing Progenitor Cells
    Stephanie Azzopardi
    Department of Pathology and Laboratory Medicine, Weill Cornell Medicine, New York, NY 10065, USA
    Neoplasia 18:610-617. 2016
    ..Additionally, in a mouse model of β-cell hyperplasia, we demonstrate that p53 and Ink4a/Arf play cooperative roles in constraining the progression of PanNETs...