J Carl Pallais
Affiliation: Massachusetts General Hospital
- Acquired hypocalciuric hypercalcemia due to autoantibodies against the calcium-sensing receptorJ Carl Pallais
Department of Endocrinology, Massachusetts General Hospital, Boston, MA 02114, USA
N Engl J Med 351:362-9. 2004
- Autoimmune hypocalciuric hypercalcemia unresponsive to glucocorticoid therapy in a patient with blocking autoantibodies against the calcium-sensing receptorJ Carl Pallais
Endocrine Unit, Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts 02114, USA
J Clin Endocrinol Metab 96:672-80. 2011..Previously, glucocorticoid treatment has been shown to decrease the CaSR autoantibody titers and normalize the hypercalcemia in a patient with autoimmune hypocalciuric hypercalcemia (AHH)...
- Kiss1-/- mice exhibit more variable hypogonadism than Gpr54-/- miceRisto Lapatto
Reproductive Endocrine Unit, Department of Medicine, Massachusetts General Hospital, 55 Fruit Street, Boston, MA 02114, USA
Endocrinology 148:4927-36. 2007..Kiss1 knockout females demonstrate a bimodal phenotypic variability, with some animals having higher gonadal weight, larger vaginal opening, and persistent vaginal cornification...
- Neuroendocrine, gonadal, placental, and obstetric phenotypes in patients with IHH and mutations in the G-protein coupled receptor, GPR54J Carl Pallais
Reproductive Endocrine Unit of the Department of Medicine, Massachusetts General Hospital, Boston, MA 02114, United States
Mol Cell Endocrinol 254:70-7. 2006..Taken together, these observations help to tease apart the neuroendocrine and gonadal phenotypes of patients bearing mutations in GPR54...