J C Campbell
Affiliation: Johns Hopkins University
- An evaluation of a system-change training model to improve emergency department response to battered womenJ C Campbell
Johns Hopkins University, School of Nursing, Baltimore, MD 21205 2110, USA
Acad Emerg Med 8:131-8. 2001....
- Nerve lesions and the generation of painJ N Campbell
Department of Neurosurgery, Johns Hopkins University School of Medicine, 600 N Wolfe Street, Meyer 5 109, Baltimore, Maryland 21287, USA
Muscle Nerve 24:1261-73. 2001..The findings relative to the "intact" nociceptor provide a rationale by which to understand how therapies distal to the nerve injury site may diminish pain...
- Obturator nerve transfer as an option for femoral nerve repair: case reportAshley A Campbell
Department of Neurosurgery, The Johns Hopkins University School of Medicine, Baltimore, Maryland 21287, USA
Neurosurgery 66:375; discussion 375. 2010..Here, we present what is believed to be the first description of a successful transfer of the obturator nerve to the femoral nerve...
- Mechanisms of neuropathic painJames N Campbell
Johns Hopkins University School of Medicine, Baltimore, Maryland 21287, USA
Neuron 52:77-92. 2006..The rapid gain in knowledge about abnormal signaling promises breakthroughs in the treatment of these often debilitating disorders...
- The correlation between dietary soy phytoestrogens and neuropathic pain behavior in rats after partial denervationYoram Shir
Department of Anesthesiology and Pain Relief Unit, Hadassah University Hospital, Jerusalem, Israel
Anesth Analg 94:421-6, table of contents. 2002..Low and high plasma phytoestrogen levels were not analgesic in these tests. This report is the first to show that, at certain plasma concentrations, phytoestrogens reduce neuropathic pain in rats...
- Degeneration of myelinated efferent fibers induces spontaneous activity in uninjured C-fiber afferentsGang Wu
Department of Neurosurgery, Johns Hopkins University, Baltimore, Maryland 21287, USA
J Neurosci 22:7746-53. 2002..Ectopic spontaneous activity from injured afferents was not required for the development of the neuropathic pain behavior. These results provide additional evidence for a role of Wallerian degeneration in neuropathic pain...
- Naloxone increases pain induced by topical capsaicin in healthy human volunteersWilliam S Anderson
Department of Neurosurgery, School of Medicine, Johns Hopkins University, Meyer 5 109, 600 N Wolfe Street, Baltimore, MD 21287 0817, USA
Pain 99:207-16. 2002..01) and placebo (P < 0.01). The peak effect, reached at 12-20 min after naloxone delivery, was 59% greater than placebo. This experiment suggests that acute pain is actively suppressed by endogenous opioid-receptor activation...
- Fatigue and paradoxical enhancement of heat response in C-fiber nociceptors from cross-modal excitationYuan B Peng
Department of Neurosurgery, School of Medicine, Johns Hopkins University, Baltimore, Maryland 21287
J Neurosci 23:4766-74. 2003..The dependency of recovery from fatigue on the modality of the CS suggests that fatigue results from transduction-spike initiation mechanisms...
- Severity and duration of hyperalgesia in rat varies with type of nerve lesionMary Pat Lancelotta
Department of Neurosurgery, Johns Hopkins University School of Medicine, Baltimore, Maryland 21287 7509, USA
Neurosurgery 53:1200-8; discussion 1208-9. 2003..To learn how lesions with differing capacity for nerve regeneration affect the severity and duration of hyperalgesia in an animal model of neuropathic pain...
- Role of Endogenous Opioid System in the Placebo EffectJames Campbell; Fiscal Year: 2006..3) Identify relationships between the magnitude of the placebo response and regional mu opioid receptor availability in a non-painful condition. ..
- MECHANISMS OF NEUROPATHIC PAINJames Campbell; Fiscal Year: 2008..These studies will provide a biological rationale for the development of peripheral treatments for neuropathic pain. [unreadable] [unreadable] [unreadable]..