M Warny

Summary

Affiliation: Harvard University
Country: USA

Publications

  1. ncbi request reprint Monocytic cell necrosis is mediated by potassium depletion and caspase-like proteases
    M Warny
    Gastroenterology Division, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts 02215, USA
    Am J Physiol 276:C717-24. 1999
  2. ncbi request reprint P2Y(6) nucleotide receptor mediates monocyte interleukin-8 production in response to UDP or lipopolysaccharide
    M Warny
    Gastroenterology Divison, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts 02215, USA
    J Biol Chem 276:26051-6. 2001
  3. pmc p38 MAP kinase activation by Clostridium difficile toxin A mediates monocyte necrosis, IL-8 production, and enteritis
    M Warny
    Gastroenterology Division, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts 02215, USA
    J Clin Invest 105:1147-56. 2000
  4. ncbi request reprint Differential activation of mitogen-activated protein kinases in AGS gastric epithelial cells by cag+ and cag- Helicobacter pylori
    S Keates
    Gastroenterology Division, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA 02215, USA
    J Immunol 163:5552-9. 1999
  5. ncbi request reprint Association between antibody response to toxin A and protection against recurrent Clostridium difficile diarrhoea
    L Kyne
    Gerontology Division, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA 02215, USA
    Lancet 357:189-93. 2001
  6. pmc Saccharomyces boulardii stimulates intestinal immunoglobulin A immune response to Clostridium difficile toxin A in mice
    A Qamar
    Gastroenterology Division, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts 02215, USA
    Infect Immun 69:2762-5. 2001

Collaborators

Detail Information

Publications6

  1. ncbi request reprint Monocytic cell necrosis is mediated by potassium depletion and caspase-like proteases
    M Warny
    Gastroenterology Division, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts 02215, USA
    Am J Physiol 276:C717-24. 1999
    ..Thus, in monocytic cells, necrosis is a cell death pathway mediated by passive K+ efflux and activation of caspase-like proteases...
  2. ncbi request reprint P2Y(6) nucleotide receptor mediates monocyte interleukin-8 production in response to UDP or lipopolysaccharide
    M Warny
    Gastroenterology Divison, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts 02215, USA
    J Biol Chem 276:26051-6. 2001
    ..Furthermore, lipopolysaccharide mediates IL-8 production at least in part by autocrine P2Y(6) activation. These findings indicate a novel role for P2Y(6) in innate immune defenses...
  3. pmc p38 MAP kinase activation by Clostridium difficile toxin A mediates monocyte necrosis, IL-8 production, and enteritis
    M Warny
    Gastroenterology Division, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts 02215, USA
    J Clin Invest 105:1147-56. 2000
    ..p38 MAP kinase also mediates intestinal inflammation and mucosal damage induced by toxin A...
  4. ncbi request reprint Differential activation of mitogen-activated protein kinases in AGS gastric epithelial cells by cag+ and cag- Helicobacter pylori
    S Keates
    Gastroenterology Division, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA 02215, USA
    J Immunol 163:5552-9. 1999
    ..pylori cag+ strains may be instrumental in inducing gastroduodenal inflammation, ulceration, and neoplasia...
  5. ncbi request reprint Association between antibody response to toxin A and protection against recurrent Clostridium difficile diarrhoea
    L Kyne
    Gerontology Division, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA 02215, USA
    Lancet 357:189-93. 2001
    ..The aim of this study was to determine whether an acquired immune response to toxin A, during an episode of C. difficile diarrhoea, influences risk of recurrence...
  6. pmc Saccharomyces boulardii stimulates intestinal immunoglobulin A immune response to Clostridium difficile toxin A in mice
    A Qamar
    Gastroenterology Division, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts 02215, USA
    Infect Immun 69:2762-5. 2001
    ..003) and a 4.4-fold increase in specific intestinal anti-toxin A levels (P < 0.001). Enhancing host intestinal immune responses may be an important mechanism for S. boulardii-mediated protection against diarrheal illnesses...