S E Browne

Summary

Affiliation: Harvard University
Country: USA

Publications

  1. ncbi Neurodegenerative disease
    S E Browne
    Neurology Research, Massachusetts General Hospital, Harvard Medical School, Warren 408, Boston, MA 02114, USA
    IDrugs 2:4-6. 1999
  2. ncbi The cerebral metabolic effects of manipulating glutamatergic systems within the basal forebrain in conscious rats
    S E Browne
    Wellcome Surgical Institute, University of Glasgow, Scotland
    Eur J Neurosci 10:649-63. 1998
  3. ncbi Oxidative stress in Huntington's disease
    S E Browne
    Department of Neurology, Massachusetts General Hospital and Harvard Medical School, Boston 02114, USA
    Brain Pathol 9:147-63. 1999
  4. ncbi Creatine increase survival and delays motor symptoms in a transgenic animal model of Huntington's disease
    O A Andreassen
    Neurochemistry Laboratory, Neurology Service, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusets, USA
    Neurobiol Dis 8:479-91. 2001
  5. ncbi The cerebral metabolic consequences of nitric oxide synthase deficiency: glucose utilization in endothelial and neuronal nitric oxide synthase null mice
    S E Browne
    Laboratory of Neurochemistry, Massachusetts General Hospital and Harvard Medical School, Boston 02114, USA
    J Cereb Blood Flow Metab 19:144-8. 1999
  6. ncbi Inhibition of neuronal nitric oxide synthase by 7-nitroindazole protects against MPTP-induced neurotoxicity in mice
    J B Schulz
    Neurochemistry Laboratory, Massachusetts General Hospital, Boston 02114
    J Neurochem 64:936-9. 1995

Detail Information

Publications6

  1. ncbi Neurodegenerative disease
    S E Browne
    Neurology Research, Massachusetts General Hospital, Harvard Medical School, Warren 408, Boston, MA 02114, USA
    IDrugs 2:4-6. 1999
    ....
  2. ncbi The cerebral metabolic effects of manipulating glutamatergic systems within the basal forebrain in conscious rats
    S E Browne
    Wellcome Surgical Institute, University of Glasgow, Scotland
    Eur J Neurosci 10:649-63. 1998
    ..Results support a dissociation between the functional sequelae of NMDA and non-NMDA receptor-mediated events in the basal forebrain, and long-term compensatory functional adaptation following cortical denervation...
  3. ncbi Oxidative stress in Huntington's disease
    S E Browne
    Department of Neurology, Massachusetts General Hospital and Harvard Medical School, Boston 02114, USA
    Brain Pathol 9:147-63. 1999
    ..Here we review the evidence for oxidative damage and potential mechanisms of neuronal death in HD...
  4. ncbi Creatine increase survival and delays motor symptoms in a transgenic animal model of Huntington's disease
    O A Andreassen
    Neurochemistry Laboratory, Neurology Service, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusets, USA
    Neurobiol Dis 8:479-91. 2001
    ..These results are similar to those observed using dietary creatine supplementation in the R6/2 transgenic mouse model of HD and provide further evidence that creatine may exert therapeutic effects in HD...
  5. ncbi The cerebral metabolic consequences of nitric oxide synthase deficiency: glucose utilization in endothelial and neuronal nitric oxide synthase null mice
    S E Browne
    Laboratory of Neurochemistry, Massachusetts General Hospital and Harvard Medical School, Boston 02114, USA
    J Cereb Blood Flow Metab 19:144-8. 1999
    ..Glucose utilization levels did not significantly differ between nNOS and eNOS knockout mice and C57B1/6 mice in any of the 48 brain regions examined, but were relatively lower in some subcortical regions in SV129 mice...
  6. ncbi Inhibition of neuronal nitric oxide synthase by 7-nitroindazole protects against MPTP-induced neurotoxicity in mice
    J B Schulz
    Neurochemistry Laboratory, Massachusetts General Hospital, Boston 02114
    J Neurochem 64:936-9. 1995
    ..Our results suggest that NO. plays a role in MPTP neurotoxicity as well as novel therapeutic strategies for Parkinson's disease...