Vishva Dixit

Summary

Affiliation: Genentech Inc
Country: USA

Publications

  1. pmc Transcription factor Etv5 is essential for the maintenance of alveolar type II cells.
    Zhen Zhang
    Proc Natl Acad Sci U S A 114:3903-3908. 2017
  2. pmc Impaired c-Jun amino terminal kinase activity and T cell differentiation in death receptor 6-deficient mice
    H Zhao
    Department of Molecular Oncology, Genentech Inc, 1 DNA Way, South San Francisco, CA 94080, USA
    J Exp Med 194:1441-8. 2001
  3. pmc Yersinia virulence factor YopJ acts as a deubiquitinase to inhibit NF-kappa B activation
    Honglin Zhou
    Molecular Oncology Department, Genentech, Inc, San Francisco, CA 94080, USA
    J Exp Med 202:1327-32. 2005
  4. pmc Mitochondrial reactive oxygen species drive proinflammatory cytokine production
    Edwina Naik
    Genentech, Inc, South San Francisco, CA 94080, USA
    J Exp Med 208:417-20. 2011
  5. ncbi NF-kappaB signaling. Many roads lead to madrid
    Vishva Dixit
    Genentech, Inc, 1 DNA Way, Building 10, Room 290, South San Francisco, CA 94080, USA
    Cell 111:615-9. 2002
  6. doi Profile: Learning from death
    Melinda Wenner
    Nature 453:271-3. 2008

Collaborators

  • Nobuhiko Kayagaki
  • Tak W Mak
  • H Wang
  • Edwina Naik
  • Zhen Zhang
  • Melinda Wenner
  • Honglin Zhou
  • Wyne P Lee
  • Jeffrey Eastham Anderson
  • Sarah K Kummerfeld
  • Lilian Phu
  • Kim Newton
  • Joshua Webster
  • Donald S Kirkpatrick
  • Melissa R Junttila
  • Hong Li
  • Jinfeng Liu
  • Jiansheng Wu
  • H Zhao
  • Ingrid Wertz
  • Jianpin Yin
  • Beni Wolf
  • Denise M Monack
  • M Yan
  • I S Grewal
  • S Erickson

Detail Information

Publications7

  1. pmc Transcription factor Etv5 is essential for the maintenance of alveolar type II cells.
    Zhen Zhang
    Proc Natl Acad Sci U S A 114:3903-3908. 2017
    ..These findings identify Etv5 as a critical output of Ras signaling in AT2 cells, contributing to both lung homeostasis and tumor initiation...
  2. pmc Impaired c-Jun amino terminal kinase activity and T cell differentiation in death receptor 6-deficient mice
    H Zhao
    Department of Molecular Oncology, Genentech Inc, 1 DNA Way, South San Francisco, CA 94080, USA
    J Exp Med 194:1441-8. 2001
    ..Therefore, DR6, working through JNK, rather than apoptosis, functions to attenuate the Th2 response. This is the first demonstration of a role in the activation and differentiation of Th cells by DR6 in particular and DRs in general...
  3. pmc Yersinia virulence factor YopJ acts as a deubiquitinase to inhibit NF-kappa B activation
    Honglin Zhou
    Molecular Oncology Department, Genentech, Inc, San Francisco, CA 94080, USA
    J Exp Med 202:1327-32. 2005
    ..Moreover, an in vitro assay was established to demonstrate directly the deubiquitinating activity of purified YopJ...
  4. pmc Mitochondrial reactive oxygen species drive proinflammatory cytokine production
    Edwina Naik
    Genentech, Inc, South San Francisco, CA 94080, USA
    J Exp Med 208:417-20. 2011
    ..New work exploring the mechanisms linking ROS and inflammation find that ROS derived from mitochondria act as signal-transducing molecules that provoke the up-regulation of inflammatory cytokine subsets via distinct molecular pathways...
  5. ncbi NF-kappaB signaling. Many roads lead to madrid
    Vishva Dixit
    Genentech, Inc, 1 DNA Way, Building 10, Room 290, South San Francisco, CA 94080, USA
    Cell 111:615-9. 2002
    ..The emerging understanding of new roles for NF-kappaB and complexities of the regulation of its function was the focus of a recent Juan March workshop held recently in Madrid, Spain...
  6. doi Profile: Learning from death
    Melinda Wenner
    Nature 453:271-3. 2008