Yuwaraj Kadariya

Summary

Affiliation: Fox Chase Cancer Center
Country: USA

Publications

  1. pmc Regulation of human methylthioadenosine phosphorylase gene by the CBF (CCAAT binding factor)/NF-Y (nuclear factor-Y)
    Yuwaraj Kadariya
    Department of Laboratory Medicine, Mie University School of Medicine, Tsu, Mie 514 8507, Japan
    Biochem J 387:175-83. 2005
  2. ncbi request reprint Molecular characterization of 5'-deoxy-5'-methylthioadenosine phosphorylase-deficient mutant clones of murine lymphoma cell line R1.1
    Yuwaraj Kadariya
    Department of Laboratory Medicine, Mie University School of Medicine and Central Clinical Laboratories, Mie University Hospital, Tsu, Mie 514 8507, Japan
    Cancer Sci 94:519-22. 2003
  3. ncbi request reprint Deletion of dinucleotide repeat (Delta 14 allele) in the methylthioadenosine phosphorylase (MTAP) promoter and the allelotype of MTAP promoter in the Japanese population
    Yuwaraj Kadariya
    Department of Laboratory Medicine, Mie University Faculty of Medicine, Tsu, Mie 514 8507, Japan
    Jpn J Cancer Res 93:369-73. 2002

Detail Information

Publications3

  1. pmc Regulation of human methylthioadenosine phosphorylase gene by the CBF (CCAAT binding factor)/NF-Y (nuclear factor-Y)
    Yuwaraj Kadariya
    Department of Laboratory Medicine, Mie University School of Medicine, Tsu, Mie 514 8507, Japan
    Biochem J 387:175-83. 2005
    ..Our results show that the hMTAP expression is regulated by a CBF and that the distal one of two CCAAT motifs plays a major role in the transcriptional activation of hMTAP gene...
  2. ncbi request reprint Molecular characterization of 5'-deoxy-5'-methylthioadenosine phosphorylase-deficient mutant clones of murine lymphoma cell line R1.1
    Yuwaraj Kadariya
    Department of Laboratory Medicine, Mie University School of Medicine and Central Clinical Laboratories, Mie University Hospital, Tsu, Mie 514 8507, Japan
    Cancer Sci 94:519-22. 2003
    ..Thus, C223Y may account for the partial deficiency in the F clone, but the complete deficiency with a remarkably low MTAP mRNA level in the H5 clone may be a result of transcriptional abnormality in addition to C223Y mutation...
  3. ncbi request reprint Deletion of dinucleotide repeat (Delta 14 allele) in the methylthioadenosine phosphorylase (MTAP) promoter and the allelotype of MTAP promoter in the Japanese population
    Yuwaraj Kadariya
    Department of Laboratory Medicine, Mie University Faculty of Medicine, Tsu, Mie 514 8507, Japan
    Jpn J Cancer Res 93:369-73. 2002
    ..These results indicated that the Delta 14 allele has nothing to do with MTAP deficiency in DHL-9. The Delta 14 allele is distributed among the general population irrespective of gender...