Catalina Perdomo

Summary

Affiliation: Boston University
Country: USA

Publications

  1. doi request reprint MiRNAs as regulators of the response to inhaled environmental toxins and airway carcinogenesis
    Catalina Perdomo
    Boston University School of Medicine, Boston, MA 02118, USA
    Mutat Res 717:32-7. 2011
  2. pmc MicroRNA 4423 is a primate-specific regulator of airway epithelial cell differentiation and lung carcinogenesis
    Catalina Perdomo
    Division of Computational Biomedicine, Genetics and Genomics Program, and Pulmonary Center, Department of Medicine, Boston University, Boston, MA 02118
    Proc Natl Acad Sci U S A 110:18946-51. 2013
  3. pmc A dynamic bronchial airway gene expression signature of chronic obstructive pulmonary disease and lung function impairment
    Katrina Steiling
    Division of Computational Biomedicine, Boston University School of Medicine, Boston, MA 02118, USA
    Am J Respir Crit Care Med 187:933-42. 2013
  4. ncbi request reprint miR-129 regulates cell proliferation by downregulating Cdk6 expression
    Junjie Wu
    Pulmonary Center, Department of Medicine, Boston University School of Medicine, Boston, MA, USA
    Cell Cycle 9:1809-18. 2010
  5. pmc MicroRNAs as modulators of smoking-induced gene expression changes in human airway epithelium
    Frank Schembri
    Pulmonary Center, Boston University School of Medicine, Boston, MA 02118, USA
    Proc Natl Acad Sci U S A 106:2319-24. 2009

Collaborators

Detail Information

Publications5

  1. doi request reprint MiRNAs as regulators of the response to inhaled environmental toxins and airway carcinogenesis
    Catalina Perdomo
    Boston University School of Medicine, Boston, MA 02118, USA
    Mutat Res 717:32-7. 2011
    ..These miRNA responses to inhaled toxins appear to be recapitulated in lung carcinogenesis, opening the possibility that modulation of the miRNA response could be a novel strategy for chemoprevention...
  2. pmc MicroRNA 4423 is a primate-specific regulator of airway epithelial cell differentiation and lung carcinogenesis
    Catalina Perdomo
    Division of Computational Biomedicine, Genetics and Genomics Program, and Pulmonary Center, Department of Medicine, Boston University, Boston, MA 02118
    Proc Natl Acad Sci U S A 110:18946-51. 2013
    ..Together, our results indicate that miR-4423 is a regulator of airway epithelium differentiation and that the abrogation of its function contributes to lung carcinogenesis. ..
  3. pmc A dynamic bronchial airway gene expression signature of chronic obstructive pulmonary disease and lung function impairment
    Katrina Steiling
    Division of Computational Biomedicine, Boston University School of Medicine, Boston, MA 02118, USA
    Am J Respir Crit Care Med 187:933-42. 2013
    ..Molecular phenotyping of chronic obstructive pulmonary disease (COPD) has been impeded in part by the difficulty in obtaining lung tissue samples from individuals with impaired lung function...
  4. ncbi request reprint miR-129 regulates cell proliferation by downregulating Cdk6 expression
    Junjie Wu
    Pulmonary Center, Department of Medicine, Boston University School of Medicine, Boston, MA, USA
    Cell Cycle 9:1809-18. 2010
    ..Together, our data indicate that miR-129 plays an important role in regulating cell proliferation by downregulation of Cdk6...
  5. pmc MicroRNAs as modulators of smoking-induced gene expression changes in human airway epithelium
    Frank Schembri
    Pulmonary Center, Boston University School of Medicine, Boston, MA 02118, USA
    Proc Natl Acad Sci U S A 106:2319-24. 2009
    ..These data indicate that mir-218 levels modulate the airway epithelial gene expression response to cigarette smoke and support a role for miRNAs in regulating host response to environmental toxins...