Jamie D Croxtall

Summary

Affiliation: Queen Mary
Country: UK

Publications

  1. pmc Different glucocorticoids vary in their genomic and non-genomic mechanism of action in A549 cells
    Jamie D Croxtall
    Department of Biochemical Pharmacology, The William Harvey Research Institute, St Bartholomew s and the Royal London School of Medicine and Dentistry Queen Mary and Westfield College, Charterhouse Square, London EC1M 6BQ
    Br J Pharmacol 135:511-9. 2002
  2. pmc Attenuation of glucocorticoid functions in an Anx-A1-/- cell line
    Jamie D Croxtall
    Department of Biochemical Pharmacology, William Harvey Research Institute, Queen Mary, University of London, Charterhouse Square, London EC1M 6BQ, UK
    Biochem J 371:927-35. 2003
  3. pmc Differential modulation of glucocorticoid action by FK506 in A549 cells
    Jamie D Croxtall
    Department of Biochemical Pharmacology, William Harvey Research Institute, Queen Mary, University of London, Charterhouse Square, London EC1M 6BQ, UK
    Biochem J 376:285-90. 2003
  4. ncbi request reprint Aberrant inflammation and resistance to glucocorticoids in annexin 1-/- mouse
    Robert Hannon
    Department of Biochemical Pharmacology, William Harvey Research Institute, Queen Mary, University of London, Charterhouse Square, EC1M 6BQ, UK
    FASEB J 17:253-5. 2003
  5. ncbi request reprint Investigation into the involvement of phospholipases A2 and map kinases in modulation of AA release and cell growth in A549 cells
    Qamrul G Choudhury
    Department of Biochemical Pharmacology, William Harvey Research Institute, St Bartholomew s and the Royal London School of Medicine and Dentistry Queen Mary and Westfield College, Charterhouse Square, London EC1M 6BQ
    Adv Exp Med Biol 507:33-40. 2002
  6. ncbi request reprint A novel role for phospholipase A2 isoforms in the checkpoint control of acute inflammation
    Derek W Gilroy
    Department of Experimental Pathology, William Harvey Research Institute, St Bartholomew s and the Royal London School of Medicine and Dentistry, London
    FASEB J 18:489-98. 2004

Collaborators

Detail Information

Publications6

  1. pmc Different glucocorticoids vary in their genomic and non-genomic mechanism of action in A549 cells
    Jamie D Croxtall
    Department of Biochemical Pharmacology, The William Harvey Research Institute, St Bartholomew s and the Royal London School of Medicine and Dentistry Queen Mary and Westfield College, Charterhouse Square, London EC1M 6BQ
    Br J Pharmacol 135:511-9. 2002
    ..The implications for these findings are discussed...
  2. pmc Attenuation of glucocorticoid functions in an Anx-A1-/- cell line
    Jamie D Croxtall
    Department of Biochemical Pharmacology, William Harvey Research Institute, Queen Mary, University of London, Charterhouse Square, London EC1M 6BQ, UK
    Biochem J 371:927-35. 2003
    ..Taken together, these observations suggest strongly that Anx-A1 functions as an inhibitor of signal-transduction pathways that lead to cell proliferation and may help to explain how glucocorticoids regulate these processes...
  3. pmc Differential modulation of glucocorticoid action by FK506 in A549 cells
    Jamie D Croxtall
    Department of Biochemical Pharmacology, William Harvey Research Institute, Queen Mary, University of London, Charterhouse Square, London EC1M 6BQ, UK
    Biochem J 376:285-90. 2003
    ..We show here that the FKB51 (FK506-binding protein 51)-FKB52 switch is differentially regulated by glucocorticoid and FK506 treatment strategy...
  4. ncbi request reprint Aberrant inflammation and resistance to glucocorticoids in annexin 1-/- mouse
    Robert Hannon
    Department of Biochemical Pharmacology, William Harvey Research Institute, Queen Mary, University of London, Charterhouse Square, EC1M 6BQ, UK
    FASEB J 17:253-5. 2003
    ..There are also gender differences in activated leukocyte behavior in the Anx-1-/- mice that are not seen in the wild-type animals, suggesting an interaction between sex hormones and inflammation in Anx-1-/- animals...
  5. ncbi request reprint Investigation into the involvement of phospholipases A2 and map kinases in modulation of AA release and cell growth in A549 cells
    Qamrul G Choudhury
    Department of Biochemical Pharmacology, William Harvey Research Institute, St Bartholomew s and the Royal London School of Medicine and Dentistry Queen Mary and Westfield College, Charterhouse Square, London EC1M 6BQ
    Adv Exp Med Biol 507:33-40. 2002
  6. ncbi request reprint A novel role for phospholipase A2 isoforms in the checkpoint control of acute inflammation
    Derek W Gilroy
    Department of Experimental Pathology, William Harvey Research Institute, St Bartholomew s and the Royal London School of Medicine and Dentistry, London
    FASEB J 18:489-98. 2004
    ..This study is the first of its kind to address the respective roles of PLA2 isoforms in acute resolving inflammation and to identify type VI iPLA2 as a potentially selective target for the treatment of inflammatory diseases...