M Lundblad

Summary

Affiliation: Lund University
Country: Sweden

Publications

  1. ncbi request reprint Persistent changes in striatal gene expression induced by long-term L-DOPA treatment in a rat model of Parkinson's disease
    J E Westin
    Department of Physiological Sciences, Section of Neurobiology, Lund University, Wallenberg Neuroscience Centre, BMC A11, S-221 84 Lund, Sweden
    Eur J Neurosci 14:1171-6. 2001
  2. ncbi request reprint Pharmacological validation of behavioural measures of akinesia and dyskinesia in a rat model of Parkinson's disease
    M Lundblad
    Department of Physiological Sciences, Wallenberg Neuroscience Centre, Neurobiology Division, Lund University, BMC A11, S 221 84 Lund, Sweden
    Eur J Neurosci 15:120-32. 2002
  3. ncbi request reprint Cellular and behavioural effects of the adenosine A2a receptor antagonist KW-6002 in a rat model of l-DOPA-induced dyskinesia
    M Lundblad
    Department of Physiological Sciences, Wallenberg Neuroscience Centre, Neurobiology Division, Lund, Sweden
    J Neurochem 84:1398-410. 2003
  4. ncbi request reprint A model of L-DOPA-induced dyskinesia in 6-hydroxydopamine lesioned mice: relation to motor and cellular parameters of nigrostriatal function
    M Lundblad
    Department of Physiological Sciences, Neurobiology Division, Wallenberg Neuroscience Centre, Lund University, S 223 62 Lund, Sweden
    Neurobiol Dis 16:110-23. 2004
  5. ncbi request reprint Pharmacological validation of a mouse model of l-DOPA-induced dyskinesia
    M Lundblad
    Wallenberg Neuroscience Centre, Section of Basal Ganglia Pathophysiology, BMC A11, S 221 84 Lund, Sweden
    Exp Neurol 194:66-75. 2005
  6. doi request reprint Progressive neurodegenerative and behavioural changes induced by AAV-mediated overexpression of α-synuclein in midbrain dopamine neurons
    M Decressac
    Wallenberg Neuroscience Center, Department of Experimental Medical Sciences, Lund University, BMC A11, Lund 22184, Sweden
    Neurobiol Dis 45:939-53. 2012
  7. ncbi request reprint Effects of pulsatile L-DOPA treatment in the double lesion rat model of striatonigral degeneration (multiple system atrophy)
    N Stefanova
    Department of Neurology, University Hospital of Innsbruck, Innsbruck, Austria
    Neurobiol Dis 15:630-9. 2004

Collaborators

Detail Information

Publications7

  1. ncbi request reprint Persistent changes in striatal gene expression induced by long-term L-DOPA treatment in a rat model of Parkinson's disease
    J E Westin
    Department of Physiological Sciences, Section of Neurobiology, Lund University, Wallenberg Neuroscience Centre, BMC A11, S-221 84 Lund, Sweden
    Eur J Neurosci 14:1171-6. 2001
    ....
  2. ncbi request reprint Pharmacological validation of behavioural measures of akinesia and dyskinesia in a rat model of Parkinson's disease
    M Lundblad
    Department of Physiological Sciences, Wallenberg Neuroscience Centre, Neurobiology Division, Lund University, BMC A11, S 221 84 Lund, Sweden
    Eur J Neurosci 15:120-32. 2002
    ..Such deficits can be quantified using novel and relatively simple testing procedures, whereas rotometry cannot discriminate between dyskinetic and antiakinetic effects of antiparkinsonian treatments...
  3. ncbi request reprint Cellular and behavioural effects of the adenosine A2a receptor antagonist KW-6002 in a rat model of l-DOPA-induced dyskinesia
    M Lundblad
    Department of Physiological Sciences, Wallenberg Neuroscience Centre, Neurobiology Division, Lund, Sweden
    J Neurochem 84:1398-410. 2003
    ..Cotreatment with KW-6002 and L-DOPA potentiates the therapeutic effect but not the dyskinesiogenic potential of the latter drug...
  4. ncbi request reprint A model of L-DOPA-induced dyskinesia in 6-hydroxydopamine lesioned mice: relation to motor and cellular parameters of nigrostriatal function
    M Lundblad
    Department of Physiological Sciences, Neurobiology Division, Wallenberg Neuroscience Centre, Lund University, S 223 62 Lund, Sweden
    Neurobiol Dis 16:110-23. 2004
    ..The mouse model of L-DOPA-induced dyskinesia will provide a useful tool to study the molecular determinants of this movement disorder in transgenic mice strains...
  5. ncbi request reprint Pharmacological validation of a mouse model of l-DOPA-induced dyskinesia
    M Lundblad
    Wallenberg Neuroscience Centre, Section of Basal Ganglia Pathophysiology, BMC A11, S 221 84 Lund, Sweden
    Exp Neurol 194:66-75. 2005
    ..The present data indicate that the mouse AIMs are indeed a functional equivalent of l-DOPA-induced dyskinesia...
  6. doi request reprint Progressive neurodegenerative and behavioural changes induced by AAV-mediated overexpression of α-synuclein in midbrain dopamine neurons
    M Decressac
    Wallenberg Neuroscience Center, Department of Experimental Medical Sciences, Lund University, BMC A11, Lund 22184, Sweden
    Neurobiol Dis 45:939-53. 2012
    ..This model provides new interesting possibilities for studies of stage-specific pathologic mechanisms and identification of targets for disease-modifying therapeutic interventions linked to early or late stages of the disease...
  7. ncbi request reprint Effects of pulsatile L-DOPA treatment in the double lesion rat model of striatonigral degeneration (multiple system atrophy)
    N Stefanova
    Department of Neurology, University Hospital of Innsbruck, Innsbruck, Austria
    Neurobiol Dis 15:630-9. 2004
    ....