A Rasola

Summary

Affiliation: University of Turin
Country: Italy

Publications

  1. ncbi Apoptosis enhancement by the HIV-1 Nef protein
    A Rasola
    Division of Molecular Oncology, Institute for Cancer Research, University of Torino Medical School, Candiolo, Italy
    J Immunol 166:81-8. 2001
  2. ncbi A flow cytometry assay simultaneously detects independent apoptotic parameters
    A Rasola
    Division of Molecular Oncology, Institute for Cancer Research, University of Torino Medical School, Candiolo, Italy
    Cytometry 45:151-7. 2001
  3. ncbi Apoptosis to necrosis switching downstream of apoptosome formation requires inhibition of both glycolysis and oxidative phosphorylation in a BCL-X(L)- and PKB/AKT-independent fashion
    D Gramaglia
    Division of Molecular Oncology, Institute for Cancer Research, University of Torino Medical School, Candiolo, Italy
    Cell Death Differ 11:342-53. 2004
  4. ncbi Calcium and cell death: the mitochondrial connection
    P Bernardi
    Department of Biomedical Sciences, University of Padova, Viale Giuseppe Colombo 3, 35121 Padova, Italy
    Subcell Biochem 45:481-506. 2007

Collaborators

Detail Information

Publications4

  1. ncbi Apoptosis enhancement by the HIV-1 Nef protein
    A Rasola
    Division of Molecular Oncology, Institute for Cancer Research, University of Torino Medical School, Candiolo, Italy
    J Immunol 166:81-8. 2001
    ..These data show that Nef enhances programmed cell death in different cell types by affecting multiple critical components of the apoptotic machinery independently from the Fas pathway...
  2. ncbi A flow cytometry assay simultaneously detects independent apoptotic parameters
    A Rasola
    Division of Molecular Oncology, Institute for Cancer Research, University of Torino Medical School, Candiolo, Italy
    Cytometry 45:151-7. 2001
    ....
  3. ncbi Apoptosis to necrosis switching downstream of apoptosome formation requires inhibition of both glycolysis and oxidative phosphorylation in a BCL-X(L)- and PKB/AKT-independent fashion
    D Gramaglia
    Division of Molecular Oncology, Institute for Cancer Research, University of Torino Medical School, Candiolo, Italy
    Cell Death Differ 11:342-53. 2004
    ..Apoptosis is not blocked per se by ATP depletion, as engagement of the Fas receptor directly activates caspases, thus bypassing ddFSK inhibition...
  4. ncbi Calcium and cell death: the mitochondrial connection
    P Bernardi
    Department of Biomedical Sciences, University of Padova, Viale Giuseppe Colombo 3, 35121 Padova, Italy
    Subcell Biochem 45:481-506. 2007
    ..Understanding the mechanisms through which the Ca2+ signal can be shifted from a physiological signal into a pathological effector is an unresolved problem of modern pathophysiology that holds great promise for disease treatment...