Petra Kameritsch

Summary

Affiliation: University of Munich
Country: Germany

Publications

  1. doi request reprint Cx43 increases serum induced filopodia formation via activation of p21-activated protein kinase 1
    Petra Kameritsch
    Walter Brendel Centre of Experimental Medicine, Ludwig Maximilians Universität München and Munich University Hospital, Marchioninistr 27, 81377 Munchen, Germany DZHK German Centre for Cardiovascular Research, partner site Munich Heart Alliance, Biedersteiner Str 29, 80802 Munchen, Germany Electronic address
    Biochim Biophys Acta 1853:2907-17. 2015
  2. pmc NO, via its target Cx37, modulates calcium signal propagation selectively at myoendothelial gap junctions
    Kristin Pogoda
    Walter Brendel Centre of Experimental Medicine, Munich Heart Alliance, Ludwig Maximilians Universitat Munchen, Munich, Germany
    Cell Commun Signal 12:33. 2014
  3. pmc Gap junctional communication promotes apoptosis in a connexin-type-dependent manner
    P Kameritsch
    Walter Brendel Centre of Experimental Medicine, Ludwig Maximilians Universität München and Munich University Hospital, Munich, Germany
    Cell Death Dis 4:e584. 2013
  4. doi request reprint Gap junctional communication controls the overall endothelial calcium response to vasoactive agonists
    Petra Kameritsch
    Walter Brendel Centre of Experimental Medicine, Ludwig Maximilians Universitat, Marchioninistr 27, 81377 Munich, Germany
    Cardiovasc Res 93:508-15. 2012
  5. doi request reprint The carboxyl tail of Cx43 augments p38 mediated cell migration in a gap junction-independent manner
    Juliane Behrens
    Walter Brendel Centre of Experimental Medicine, Ludwig Maximilians Universitat Munchen, Marchioninistr 27, D 81377 Munich, Germany
    Eur J Cell Biol 89:828-38. 2010
  6. ncbi request reprint Nitric oxide specifically reduces the permeability of Cx37-containing gap junctions to small molecules
    Petra Kameritsch
    Institute of Physiology, Ludwig Maximilians University Munich, Schillerstr, Munich, Germany
    J Cell Physiol 203:233-42. 2005

Collaborators

  • Wolfram Nagel
  • Kristin Pogoda
  • Ulrich Pohl
  • Juliane Behrens
  • Monika Füller
  • Stefan Wallner

Detail Information

Publications6

  1. doi request reprint Cx43 increases serum induced filopodia formation via activation of p21-activated protein kinase 1
    Petra Kameritsch
    Walter Brendel Centre of Experimental Medicine, Ludwig Maximilians Universität München and Munich University Hospital, Marchioninistr 27, 81377 Munchen, Germany DZHK German Centre for Cardiovascular Research, partner site Munich Heart Alliance, Biedersteiner Str 29, 80802 Munchen, Germany Electronic address
    Biochim Biophys Acta 1853:2907-17. 2015
    ..Our results provide a novel mechanism by which Cx43 can modify actin cytoskeletal dynamics and may thereby enhance cell migration. ..
  2. pmc NO, via its target Cx37, modulates calcium signal propagation selectively at myoendothelial gap junctions
    Kristin Pogoda
    Walter Brendel Centre of Experimental Medicine, Munich Heart Alliance, Ludwig Maximilians Universitat Munchen, Munich, Germany
    Cell Commun Signal 12:33. 2014
    ....
  3. pmc Gap junctional communication promotes apoptosis in a connexin-type-dependent manner
    P Kameritsch
    Walter Brendel Centre of Experimental Medicine, Ludwig Maximilians Universität München and Munich University Hospital, Munich, Germany
    Cell Death Dis 4:e584. 2013
    ..Our observations suggest that Cx43 and Cx40 but not Cx37 promote apoptosis via gap junctional transfer of pro-apoptotic signals between cells...
  4. doi request reprint Gap junctional communication controls the overall endothelial calcium response to vasoactive agonists
    Petra Kameritsch
    Walter Brendel Centre of Experimental Medicine, Ludwig Maximilians Universitat, Marchioninistr 27, 81377 Munich, Germany
    Cardiovasc Res 93:508-15. 2012
    ..We investigated whether interendothelial spreading of the Ca(2+) signal via gap junctions (GJs) plays a role for the overall Ca(2+)(i) increase in response to vasoactive agonists...
  5. doi request reprint The carboxyl tail of Cx43 augments p38 mediated cell migration in a gap junction-independent manner
    Juliane Behrens
    Walter Brendel Centre of Experimental Medicine, Ludwig Maximilians Universitat Munchen, Marchioninistr 27, D 81377 Munich, Germany
    Eur J Cell Biol 89:828-38. 2010
    ..These results demonstrate that expression of Cx43 augments migration via modulation of p38 MAP kinase activity. The carboxyl tail of Cx43 plays an essential role in this signalling pathway which is independent of gap junction function...
  6. ncbi request reprint Nitric oxide specifically reduces the permeability of Cx37-containing gap junctions to small molecules
    Petra Kameritsch
    Institute of Physiology, Ludwig Maximilians University Munich, Schillerstr, Munich, Germany
    J Cell Physiol 203:233-42. 2005
    ..Our results demonstrate that NO inhibits the intercellular transfer of small molecules by a specific influence on Cx37, suggesting a potential role of NO in controlling certain aspects of vascular GJIC...