Julia Schultz

Summary

Country: Germany

Publications

  1. pmc 14-3-3sigma gene silencing during melanoma progression and its role in cell cycle control and cellular senescence
    Julia Schultz
    Department of Cardiac Surgery, University of Rostock, Rostock, Germany
    Mol Cancer 8:53. 2009
  2. doi request reprint Tumor-promoting role of signal transducer and activator of transcription (Stat)1 in late-stage melanoma growth
    Julia Schultz
    Institute of Medical Biochemistry and Molecular Biology, University of Rostock, Rostock, Germany
    Clin Exp Metastasis 27:133-40. 2010
  3. doi request reprint The functional -443T/C osteopontin promoter polymorphism influences osteopontin gene expression in melanoma cells via binding of c-Myb transcription factor
    Julia Schultz
    Department of Dermatology and Venereology, University of Rostock, Rostock, Germany
    Mol Carcinog 48:14-23. 2009
  4. doi request reprint Dynamical effects of epigenetic silencing of 14-3-3sigma expression
    Julio Vera
    Systems Biology and Bioinformatics Group, Department of Computer Science, University of Rostock, 18051 Rostock, Germany
    Mol Biosyst 6:264-73. 2010
  5. doi request reprint Drp1 guarding of the mitochondrial network is important for glucose-stimulated insulin secretion in pancreatic beta cells
    Florian Reinhardt
    Institute of Medical Biochemistry and Molecular Biology, University of Rostock, D 18057 Rostock, Germany
    Biochem Biophys Res Commun 474:646-51. 2016
  6. doi request reprint MicroRNA let-7b targets important cell cycle molecules in malignant melanoma cells and interferes with anchorage-independent growth
    Julia Schultz
    Department of Dermatology and Venereology, University of Rostock, 18055 Rostock, Germany
    Cell Res 18:549-57. 2008
  7. doi request reprint Precise expression of Fis1 is important for glucose responsiveness of beta cells
    Julia Schultz
    Institute of Medical Biochemistry and Molecular BiologyUniversity of Rostock, Rostock, Germany
    J Endocrinol 230:81-91. 2016
  8. doi request reprint An mtDNA mutation accelerates liver aging by interfering with the ROS response and mitochondrial life cycle
    Jan Niemann
    Institute of Medical Biochemistry and Molecular Biology, University of Rostock, Rostock, Germany
    Free Radic Biol Med 102:174-187. 2016

Collaborators

  • Manfred Kunz
  • Julio Vera
  • Florian Reinhardt
  • Jan Niemann
  • Simone Baltrusch
  • Markus Tiedge
  • Cindy Johne
  • Rica Waterstradt
  • Susanne Schröder
  • Franziska Koch
  • Saleh M Ibrahim

Detail Information

Publications8

  1. pmc 14-3-3sigma gene silencing during melanoma progression and its role in cell cycle control and cellular senescence
    Julia Schultz
    Department of Cardiac Surgery, University of Rostock, Rostock, Germany
    Mol Cancer 8:53. 2009
    ..The 14-3-3sigma isoform acts as a tumor suppressor and is often inactivated during tumor development...
  2. doi request reprint Tumor-promoting role of signal transducer and activator of transcription (Stat)1 in late-stage melanoma growth
    Julia Schultz
    Institute of Medical Biochemistry and Molecular Biology, University of Rostock, Rostock, Germany
    Clin Exp Metastasis 27:133-40. 2010
    ..Taken together, these data suggest that Stat1 might play a role in late-stage melanoma progression. Interference with the Stat1 pathway could have therapeutic implications for late-stage melanoma patients...
  3. doi request reprint The functional -443T/C osteopontin promoter polymorphism influences osteopontin gene expression in melanoma cells via binding of c-Myb transcription factor
    Julia Schultz
    Department of Dermatology and Venereology, University of Rostock, Rostock, Germany
    Mol Carcinog 48:14-23. 2009
    ..These findings might be a feasible explanation for different OPN expression levels in metastatic tumors and may also have prognostic and therapeutic relevance...
  4. doi request reprint Dynamical effects of epigenetic silencing of 14-3-3sigma expression
    Julio Vera
    Systems Biology and Bioinformatics Group, Department of Computer Science, University of Rostock, 18051 Rostock, Germany
    Mol Biosyst 6:264-73. 2010
    ....
  5. doi request reprint Drp1 guarding of the mitochondrial network is important for glucose-stimulated insulin secretion in pancreatic beta cells
    Florian Reinhardt
    Institute of Medical Biochemistry and Molecular Biology, University of Rostock, D 18057 Rostock, Germany
    Biochem Biophys Res Commun 474:646-51. 2016
    ..In conclusion, Drp1 expression is important in pancreatic beta cells to maintain the regulation of insulin secretion. ..
  6. doi request reprint MicroRNA let-7b targets important cell cycle molecules in malignant melanoma cells and interferes with anchorage-independent growth
    Julia Schultz
    Department of Dermatology and Venereology, University of Rostock, 18055 Rostock, Germany
    Cell Res 18:549-57. 2008
    ..Taken together, these findings not only point to new regulatory mechanisms of early melanoma development, but also may open avenues for future targeted therapies of this tumor...
  7. doi request reprint Precise expression of Fis1 is important for glucose responsiveness of beta cells
    Julia Schultz
    Institute of Medical Biochemistry and Molecular BiologyUniversity of Rostock, Rostock, Germany
    J Endocrinol 230:81-91. 2016
    ..Our data confirm that FIS1 is a key regulator in pancreatic beta cells, because both glucose-stimulated insulin secretion and mitochondrial dynamics were clearly adapted to precise expression levels of this fission protein. ..
  8. doi request reprint An mtDNA mutation accelerates liver aging by interfering with the ROS response and mitochondrial life cycle
    Jan Niemann
    Institute of Medical Biochemistry and Molecular Biology, University of Rostock, Rostock, Germany
    Free Radic Biol Med 102:174-187. 2016
    ..Our results indicate that mtDNA mutations accelerate liver ballooning degeneration and carry a serious risk of premature organ aging...