David G Cox

Summary

Publications

  1. pmc N-acetyltransferase 2 polymorphisms, tobacco smoking, and breast cancer risk in the breast and prostate cancer cohort consortium
    David G Cox
    Lyon Cancer Research Center, Centre Le on Be rard, France
    Am J Epidemiol 174:1316-22. 2011
  2. doi request reprint Therogenetics: transferring GWAS technology to the clinic
    David G Cox
    Universite de Lyon, F 69000 Lyon, France, Universite Lyon 1, F 69100 Villeurbanne, France, INSERM U1052, CNRS UMR5286, Centre de Recherche en Cancérologie de Lyon, F 69000 Lyon, France and Centre Léon Bérard, F 69008 Lyon, France
    Mutagenesis 30:213-5. 2015
  3. pmc Influence of multiple APOE genetic variants on cognitive function in a cohort of older men - results from the Normative Aging Study
    Diddier Prada
    Department of Environmental Health, Harvard School of Public Health, 665 Huntington Ave, Boston 02115, MA, USA
    BMC Psychiatry 14:223. 2014
  4. pmc Genome-wide association study of circulating estradiol, testosterone, and sex hormone-binding globulin in postmenopausal women
    Jennifer Prescott
    Channing Laboratory, Department of Medicine, Brigham and Women s Hospital and Harvard Medical School, Boston, Massachusetts, United States of America
    PLoS ONE 7:e37815. 2012

Collaborators

  • Loic Le Marchand
  • Michael J Thun
  • Pierre Etienne Heudel
  • Thomas Bachelot
  • Diddier Prada
  • Jennifer Prescott
  • Jia Zhong
  • Lifang Hou
  • Marco Sánchez-Guerra
  • Melinda C Power
  • Avron Spiro Iii
  • Pantel Vokonas
  • Andrea A Baccarelli
  • Elena Colicino
  • Marc G Weisskopf
  • Joel Schwartz
  • Luis A Herrera
  • Mitch Dowsett
  • Immaculata De Vivo
  • Peter Kraft
  • Stephen J Chanock
  • Elizabeth Folkerd
  • David J Hunter
  • Susan E Hankinson
  • Deborah Doody
  • Tina Audley
  • Judith Brown
  • Deborah J Thompson
  • Kevin B Jacobs
  • Douglas F Easton
  • Jean Leyland

Detail Information

Publications4

  1. pmc N-acetyltransferase 2 polymorphisms, tobacco smoking, and breast cancer risk in the breast and prostate cancer cohort consortium
    David G Cox
    Lyon Cancer Research Center, Centre Le on Be rard, France
    Am J Epidemiol 174:1316-22. 2011
    ..These results provide some support for the hypothesis that long-term cigarette smoking may be causally associated with breast cancer risk but underscore the need for caution when interpreting sparse data on gene-environment interactions...
  2. doi request reprint Therogenetics: transferring GWAS technology to the clinic
    David G Cox
    Universite de Lyon, F 69000 Lyon, France, Universite Lyon 1, F 69100 Villeurbanne, France, INSERM U1052, CNRS UMR5286, Centre de Recherche en Cancérologie de Lyon, F 69000 Lyon, France and Centre Léon Bérard, F 69008 Lyon, France
    Mutagenesis 30:213-5. 2015
    ..In this review, we expose some of this evidence, and propose tools needed to overcome current limitations to the identification of specific germline variants that influence risk of metastases in breast cancer patients. ..
  3. pmc Influence of multiple APOE genetic variants on cognitive function in a cohort of older men - results from the Normative Aging Study
    Diddier Prada
    Department of Environmental Health, Harvard School of Public Health, 665 Huntington Ave, Boston 02115, MA, USA
    BMC Psychiatry 14:223. 2014
    ..APOE is the biomarker with the greatest known influence on cognitive function; however, the effect of complex haplotypes involving polymorphisms rs449647, rs405509, rs440446, rs429358 and rs7412 has never been studied in older populations...
  4. pmc Genome-wide association study of circulating estradiol, testosterone, and sex hormone-binding globulin in postmenopausal women
    Jennifer Prescott
    Channing Laboratory, Department of Medicine, Brigham and Women s Hospital and Harvard Medical School, Boston, Massachusetts, United States of America
    PLoS ONE 7:e37815. 2012
    ..Our results suggest that the small magnitude of difference in hormone levels associated with common genetic variants is likely insufficient to detectably contribute to breast cancer risk...