B H Hu

Summary

Affiliation: General Hospital of PLA
Country: China

Publications

  1. ncbi request reprint Intracochlear infusion of buthionine sulfoximine potentiates carboplatin ototoxicity in the chinchilla
    B H Hu
    Center for Hearing and Deafness, State University of New York at Buffalo, 14214, USA
    Hear Res 128:125-34. 1999
  2. ncbi request reprint Intense noise-induced apoptosis in hair cells of guinea pig cochleae
    B H Hu
    Institute of Otolaryngology, Chinese PLA General Hospital, Beijing, People s Republic of China
    Acta Otolaryngol 120:19-24. 2000
  3. ncbi request reprint Changes in F-actin labeling in the outer hair cell and the Deiters cell in the chinchilla cochlea following noise exposure
    B H Hu
    Department of Communicative Disorders and Sciences, State University of New York at Buffalo, 14214, USA
    Hear Res 110:209-18. 1997
  4. ncbi request reprint The role of glutathione in carboplatin ototoxicity in the chinchilla
    D Henderson
    Center for Hearing and Deafness, State University of New York at Buffalo, 3435 Main Street, 215 Parker Hall, Buffalo, New York 14214 USA, Email
    Noise Health 3:1-10. 2000
  5. pmc Toll-like receptor 4 modulates the cochlear immune response to acoustic injury
    R R Vethanayagam
    Center for Hearing and Deafness, State University of New York at Buffalo, 137 Cary Hall, 3435 Main Street, Buffalo, NY 14214, USA
    Cell Death Dis 7:e2245. 2016
  6. pmc Differential expression of apoptosis-related genes in the cochlea of noise-exposed rats
    B H Hu
    Center for Hearing and Deafness, State University of New York at Buffalo, Buffalo, NY 14214, USA
    Neuroscience 161:915-25. 2009
  7. pmc Activation of the antigen presentation function of mononuclear phagocyte populations associated with the basilar membrane of the cochlea after acoustic overstimulation
    W Yang
    Center for Hearing and Deafness, State University of New York at Buffalo, Buffalo, NY 14214, USA Department of Otolaryngology and Head and Neck Surgery, Institute of Otolaryngology, Chinese PLA General Hospital, Beijing 100853, China Electronic address
    Neuroscience 303:1-15. 2015
  8. pmc Transcriptome characterization by RNA-Seq reveals the involvement of the complement components in noise-traumatized rat cochleae
    M Patel
    Center for Hearing and Deafness, State University of New York at Buffalo, 137 Cary Hall, 3435 Main Street, Buffalo, NY 14214, USA Electronic address
    Neuroscience 248:1-16. 2013

Collaborators

Detail Information

Publications8

  1. ncbi request reprint Intracochlear infusion of buthionine sulfoximine potentiates carboplatin ototoxicity in the chinchilla
    B H Hu
    Center for Hearing and Deafness, State University of New York at Buffalo, 14214, USA
    Hear Res 128:125-34. 1999
    ..The results clearly show that BSO can enhance carboplatin ototoxicity in the chinchilla, supporting a role of GSH and reactive oxygen species in platinum ototoxicity...
  2. ncbi request reprint Intense noise-induced apoptosis in hair cells of guinea pig cochleae
    B H Hu
    Institute of Otolaryngology, Chinese PLA General Hospital, Beijing, People s Republic of China
    Acta Otolaryngol 120:19-24. 2000
    ....
  3. ncbi request reprint Changes in F-actin labeling in the outer hair cell and the Deiters cell in the chinchilla cochlea following noise exposure
    B H Hu
    Department of Communicative Disorders and Sciences, State University of New York at Buffalo, 14214, USA
    Hear Res 110:209-18. 1997
    ..A more pliable organ of Corti may have reduced the possibility of fracture or ripping of cell junctions during the motion of the basilar membrane induced by acoustic overstimulation...
  4. ncbi request reprint The role of glutathione in carboplatin ototoxicity in the chinchilla
    D Henderson
    Center for Hearing and Deafness, State University of New York at Buffalo, 3435 Main Street, 215 Parker Hall, Buffalo, New York 14214 USA, Email
    Noise Health 3:1-10. 2000
    ..The results implicate reactive oxygen species (ROS) as a common factor in ototoxic reactions because suppression of glutathione antioxidant leads to greater ototoxic reactions...
  5. pmc Toll-like receptor 4 modulates the cochlear immune response to acoustic injury
    R R Vethanayagam
    Center for Hearing and Deafness, State University of New York at Buffalo, 137 Cary Hall, 3435 Main Street, Buffalo, NY 14214, USA
    Cell Death Dis 7:e2245. 2016
    ..Together, these results suggest that Tlr4 regulates multiple aspects of the immune response in the cochlea and contributes to cochlear pathogenesis after acoustic injury. ..
  6. pmc Differential expression of apoptosis-related genes in the cochlea of noise-exposed rats
    B H Hu
    Center for Hearing and Deafness, State University of New York at Buffalo, Buffalo, NY 14214, USA
    Neuroscience 161:915-25. 2009
    ..These constitutively expressed genes, together with the inducible genes, may participate in the induction of cochlear apoptotic activity...
  7. pmc Activation of the antigen presentation function of mononuclear phagocyte populations associated with the basilar membrane of the cochlea after acoustic overstimulation
    W Yang
    Center for Hearing and Deafness, State University of New York at Buffalo, Buffalo, NY 14214, USA Department of Otolaryngology and Head and Neck Surgery, Institute of Otolaryngology, Chinese PLA General Hospital, Beijing 100853, China Electronic address
    Neuroscience 303:1-15. 2015
    ..Together, these results suggest that the antigen-presenting function of the mononuclear phagocyte population is activated in response to acoustic trauma, which could bridge the innate immune response to adaptive immunity. ..
  8. pmc Transcriptome characterization by RNA-Seq reveals the involvement of the complement components in noise-traumatized rat cochleae
    M Patel
    Center for Hearing and Deafness, State University of New York at Buffalo, 137 Cary Hall, 3435 Main Street, Buffalo, NY 14214, USA Electronic address
    Neuroscience 248:1-16. 2013
    ..Importantly, these proteins exhibited expression changes following acoustic trauma. Collectively, the results of the current investigation suggest the involvement of the complement components in cochlear responses to acoustic trauma...