Rodrigo A Quintanilla

Summary

Country: Chile

Publications

  1. pmc Mitochondrial permeability transition pore induces mitochondria injury in Huntington disease
    Rodrigo A Quintanilla
    Department of Anesthesiology, University of Rochester Medical Center, 601 Elmwood Ave, Box 604 for courier Rm 4 6314, Rochester, NY 14642, USA
    Mol Neurodegener 8:45. 2013
  2. pmc Truncated tau and Aβ cooperatively impair mitochondria in primary neurons
    Rodrigo A Quintanilla
    Department of Anesthesiology, University of Rochester, Rochester, NY 14642, USA
    Neurobiol Aging 33:619.e25-35. 2012
  3. pmc Caspase-cleaved tau expression induces mitochondrial dysfunction in immortalized cortical neurons: implications for the pathogenesis of Alzheimer disease
    Rodrigo A Quintanilla
    Department of Anesthesiology, University of Rochester, Rochester, New York 14642 0002, USA
    J Biol Chem 284:18754-66. 2009
  4. pmc Rosiglitazone treatment prevents mitochondrial dysfunction in mutant huntingtin-expressing cells: possible role of peroxisome proliferator-activated receptor-gamma (PPARgamma) in the pathogenesis of Huntington disease
    Rodrigo A Quintanilla
    Department of Anesthesiology, University of Rochester, University Medical Center, 601 Elmwood Avenue, Rochester, NY 14642, USA
    J Biol Chem 283:25628-37. 2008
  5. pmc Role of mitochondrial dysfunction in the pathogenesis of Huntington's disease
    Rodrigo A Quintanilla
    Department of Anesthesiology, University of Rochester, Rochester, NY 14642 0002, USA
    Brain Res Bull 80:242-7. 2009

Detail Information

Publications5

  1. pmc Mitochondrial permeability transition pore induces mitochondria injury in Huntington disease
    Rodrigo A Quintanilla
    Department of Anesthesiology, University of Rochester Medical Center, 601 Elmwood Ave, Box 604 for courier Rm 4 6314, Rochester, NY 14642, USA
    Mol Neurodegener 8:45. 2013
    ....
  2. pmc Truncated tau and Aβ cooperatively impair mitochondria in primary neurons
    Rodrigo A Quintanilla
    Department of Anesthesiology, University of Rochester, Rochester, NY 14642, USA
    Neurobiol Aging 33:619.e25-35. 2012
    ..These new findings show that Asp421-cleaved tau and Aβ cooperate to impair mitochondria, which likely contributes to the neuronal dysfunction in Alzheimer disease...
  3. pmc Caspase-cleaved tau expression induces mitochondrial dysfunction in immortalized cortical neurons: implications for the pathogenesis of Alzheimer disease
    Rodrigo A Quintanilla
    Department of Anesthesiology, University of Rochester, Rochester, New York 14642 0002, USA
    J Biol Chem 284:18754-66. 2009
    ..Increased calcineurin activity has been reported in AD brain, and thus, inhibition of this phosphatase may provide a therapeutic target for the treatment of AD...
  4. pmc Rosiglitazone treatment prevents mitochondrial dysfunction in mutant huntingtin-expressing cells: possible role of peroxisome proliferator-activated receptor-gamma (PPARgamma) in the pathogenesis of Huntington disease
    Rodrigo A Quintanilla
    Department of Anesthesiology, University of Rochester, University Medical Center, 601 Elmwood Avenue, Rochester, NY 14642, USA
    J Biol Chem 283:25628-37. 2008
    ....
  5. pmc Role of mitochondrial dysfunction in the pathogenesis of Huntington's disease
    Rodrigo A Quintanilla
    Department of Anesthesiology, University of Rochester, Rochester, NY 14642 0002, USA
    Brain Res Bull 80:242-7. 2009
    ....