Experts and Doctors on corpus striatum in Mexico


Locale: Mexico
Topic: corpus striatum

Top Publications

  1. Santamaría del Ángel D, Labra Ruíz N, García Cruz M, Calderón Guzmán D, Valenzuela Peraza A, Juárez Olguín H. Comparative effects of catechin, epicatechin and N-Ω-nitroarginine on quinolinic acid-induced oxidative stress in rat striatum slices. Biomed Pharmacother. 2016;78:210-215 pubmed publisher
    ..The antioxidant properties exhibited by these molecules on the excitotoxic model tested herein support an active role of glutathione and prompt their use as therapeutic tools in models of neurodegenerative disorders. ..
  2. Avila Costa M, Gutierrez Valdez A, Ordoñez Librado J, Martinez V, Colin Barenque L, Espinosa Villanueva J, et al. Time course changes of the striatum neuropil after unilateral dopamine depletion and the usefulness of the contralateral striatum as a control structure. Neurol Res. 2008;30:1068-74 pubmed publisher
  3. Cervantes Arriaga A, Rodríguez Violante M, Morales Briceño H, Neri Nani G, Millán Cepeda R, Velázquez Osuna S. Frequency and clinical correlates of postural and striatal deformities in Parkinson's disease. Clin Neurol Neurosurg. 2016;142:140-144 pubmed publisher
    ..Postural and striatal deformities are common in Parkinson's disease. Striatal deformities appear to be more related to dopaminergic treatment, while postural deformities are associated with the motor state. ..
  4. Silva Adaya D, Perez De La Cruz V, Villeda Hernandez J, Carrillo Mora P, Gonzalez Herrera I, Garcia E, et al. Protective effect of L-kynurenine and probenecid on 6-hydroxydopamine-induced striatal toxicity in rats: implications of modulating kynurenate as a protective strategy. Neurotoxicol Teratol. 2011;33:303-12 pubmed publisher
    ..The combined use of L-KYN and PROB is a valuable tool to modulate glutamatergic and cholinergic activities, presumably by means of increased levels of endogenous KYNA. ..
  5. Bardullas U, Limón Pacheco J, Giordano M, Carrizales L, Mendoza Trejo M, Rodriguez V. Chronic low-level arsenic exposure causes gender-specific alterations in locomotor activity, dopaminergic systems, and thioredoxin expression in mice. Toxicol Appl Pharmacol. 2009;239:169-77 pubmed publisher
    ..These results indicate that chronic As exposure leads to gender-dependent alterations in dopaminergic markers and spontaneous locomotor activity, and down-regulation of the antioxidant capacity of the brain. ..
  6. Rios R, Zarazúa S, Santoyo M, Sepulveda Saavedra J, Romero Diaz V, Jimenez V, et al. Decreased nitric oxide markers and morphological changes in the brain of arsenic-exposed rats. Toxicology. 2009;261:68-75 pubmed publisher
    ..These results indicate that arsenic targets the central nitrergic system and disturbs brain structural organization at low exposure levels. ..
  7. Plitman E, de la Fuente Sandoval C, Reyes Madrigal F, Chavez S, Gómez Cruz G, León Ortiz P, et al. Elevated Myo-Inositol, Choline, and Glutamate Levels in the Associative Striatum of Antipsychotic-Naive Patients With First-Episode Psychosis: A Proton Magnetic Resonance Spectroscopy Study With Implications for Glial Dysfunction. Schizophr Bull. 2016;42:415-24 pubmed publisher
    ..Dysregulation of glial function might result in the disruption of glutamatergic neurotransmission, which may influence positive symptomatology in patients with FEP. ..
  8. Rojas P, Serrano García N, Medina Campos O, Pedraza Chaverri J, Maldonado P, Ruiz Sánchez E. S-Allylcysteine, a garlic compound, protects against oxidative stress in 1-methyl-4-phenylpyridinium-induced parkinsonism in mice. J Nutr Biochem. 2011;22:937-44 pubmed publisher
    ..These findings suggest that in mice, SAC attenuates MPP(+)-induced neurotoxicity in the striatum and that an antioxidant effect against oxidative stress may be partly responsible for its observed neuroprotective effects. ..
  9. Alcauter S, García Mondragón L, Gracia Tabuenca Z, Moreno M, Ortíz J, Barrios F. Resting state functional connectivity of the anterior striatum and prefrontal cortex predicts reading performance in school-age children. Brain Lang. 2017;174:94-102 pubmed publisher
    ..This work highlights the relevance of a broad network, functionally synchronized in the resting state, for the acquisition and perfecting of reading abilities in young children. ..

More Information


  1. de la Fuente Sandoval C, León Ortiz P, Azcárraga M, Stephano S, Favila R, Díaz Galvis L, et al. Glutamate levels in the associative striatum before and after 4 weeks of antipsychotic treatment in first-episode psychosis: a longitudinal proton magnetic resonance spectroscopy study. JAMA Psychiatry. 2013;70:1057-66 pubmed publisher
    ..These results provide support for the hypothesis that improvement in clinical symptoms might be related to a decrease in glutamate levels. ..
  2. de la Fuente Sandoval C, León Ortiz P, Azcárraga M, Favila R, Stephano S, Graff Guerrero A. Striatal glutamate and the conversion to psychosis: a prospective 1H-MRS imaging study. Int J Neuropsychopharmacol. 2013;16:471-5 pubmed publisher
    ..02 and p < 0.01, respectively; effect size 1.39). These pilot findings suggest that the conversion to psychosis is associated with increased glutamate levels in the associative-striatum. ..
  3. Prieto G, Pérez Burgos A, Palomero Rivero M, Galarraga E, Drucker Colín R, Bargas J. Upregulation of D2-class signaling in dopamine-denervated striatum is in part mediated by D3 receptors acting on Ca V 2.1 channels via PIP2 depletion. J Neurophysiol. 2011;105:2260-74 pubmed publisher
  4. Mendieta L, Venegas B, Moreno N, Patricio A, Martinez I, Aguilera J, et al. The carboxyl-terminal domain of the heavy chain of tetanus toxin prevents dopaminergic degeneration and improves motor behavior in rats with striatal MPP(+)-lesions. Neurosci Res. 2009;65:98-106 pubmed publisher
  5. Medina Navarro R, Guerrero Linares I. Whole body hyperthermia reduces oxidative stress in the striatum of rats in an animal model of mitochondrial toxicity with 3-nitropropionic acid. Int J Hyperthermia. 2009;25:280-8 pubmed publisher
    ..These data contribute to a better understanding of the potential for thermal stress to modulate the type of oxidative stress usually present in neurodegenerative disorders associated with metabolic defects. ..
  6. Tellez R, Gómez Viquez L, Meneses A. GABA, glutamate, dopamine and serotonin transporters expression on memory formation and amnesia. Neurobiol Learn Mem. 2012;97:189-201 pubmed publisher
    ..Together these data show that memory formation, amnesia and anti-amnesic effects are associated to specific patters of transporters expression...
  7. Bardullas U, Giordano M, Rodriguez V. Chronic atrazine exposure causes disruption of the spontaneous locomotor activity and alters the striatal dopaminergic system of the male Sprague-Dawley rat. Neurotoxicol Teratol. 2011;33:263-72 pubmed publisher
    ..In order to unveil the full extent of atrazine's effects on the nervous system, other neurochemical systems should be considered in future studies. ..
  8. Díaz Pérez R, Castillo González J, Carcaño Díaz K, García Juárez J, Salazar Leal M, Muñoz Maldonado G, et al. Histopathological alterations in the striatum caused by Karwinskia humboldtiana (Buckthorn) fruit in an experimental model of peripheral neuropathy. Histol Histopathol. 2016;31:393-402 pubmed publisher
    ..This study is the first report that describes the histopathological alterations within the striatum caused by chronic intoxication with Kh fruit in the Wistar rat. ..
  9. Olvera Cortés M, Anguiano Rodríguez P, López Vázquez M, Alfaro J. Serotonin/dopamine interaction in learning. Prog Brain Res. 2008;172:567-602 pubmed publisher
    ..The complete significance of DA-serotonin interactions in cognitive functions could be addressed by future experimental and clinical studies. ..
  10. Arias Montaño J, Floran B, Floran L, Aceves J, Young J. Dopamine D(1) receptor facilitation of depolarization-induced release of gamma-amino-butyric acid in rat striatum is mediated by the cAMP/PKA pathway and involves P/Q-type calcium channels. Synapse. 2007;61:310-9 pubmed
    ..These observations provide evidence that dopamine D(1) agonist facilitation of the depolarization-induced release of GABA from striatal terminals is mediated by the cAMP/PKA pathway and involves mainly P/Q-type Ca(2+) channels. ..