itga9

Summary

Gene Symbol: itga9
Description: integrin, alpha 9
Alias: zgc:63474, integrin alpha-9
Species: zebrafish

Top Publications

  1. Sukardi H, Zhang X, Lui E, Ung C, Mathavan S, Gong Z, et al. Liver X receptor agonist T0901317 induced liver perturbation in zebrafish: histological, gene set enrichment and expression analyses. Biochim Biophys Acta. 2012;1820:33-43 pubmed publisher
    ..Our study underscores the potential of using zebrafish model coupled with transcriptomic analysis to capture pharmacological and toxicological or pathological events induced by LXR modulators. ..
  2. Yang Y, Enis D, Zheng H, Chia S, Yang J, Chen M, et al. Cell Adhesion Mediated by VCAM-ITGα9 Interactions Enables Lymphatic Development. Arterioscler Thromb Vasc Biol. 2015;35:1179-89 pubmed publisher
    ..We propose that the relative importance of cellular adhesive ligands is magnified under conditions of rapid tissue growth when the cell number increases faster than cell matrix, such as in the early zebrafish embryo. ..
  3. Karpanen T, Padberg Y, van de Pavert S, Dierkes C, Morooka N, Peterson Maduro J, et al. An Evolutionarily Conserved Role for Polydom/Svep1 During Lymphatic Vessel Formation. Circ Res. 2017;120:1263-1275 pubmed publisher
  4. Lien Y, Ou T, Lin Y, Kuo P, Lin H. Duplication and diversification of the spermidine/spermine N1-acetyltransferase 1 genes in zebrafish. PLoS ONE. 2013;8:e54017 pubmed publisher
  5. Musso G, Tasan M, Mosimann C, Beaver J, Plovie E, Carr L, et al. Novel cardiovascular gene functions revealed via systematic phenotype prediction in zebrafish. Development. 2014;141:224-35 pubmed publisher
    ..Thus, we show that systematic prioritization in zebrafish can accelerate the pace of developmental gene function discovery. ..
  6. Schuster K, Leeke B, Meier M, Wang Y, Newman T, Burgess S, et al. A neural crest origin for cohesinopathy heart defects. Hum Mol Genet. 2015;24:7005-16 pubmed publisher
    ..Our results give insight into the etiology of heart defects in the cohesinopathies, and raise the possibility that mild mutations in cohesin genes may be causative of a fraction of congenital heart disease in human populations. ..