Gene Symbol: Nup205
Description: Nucleoporin 205kD
Alias: CG11943, Dmel\CG11943, Ov4, anon-EST:Posey281, nup205, nucleoporin 205kD, CG11943-PA, CG11943-PB, Nup205-PA, Nup205-PB, nucleoporin 205
Species: fruit fly

Top Publications

  1. Armstrong J, Texada M, Munjaal R, Baker D, Beckingham K. Gravitaxis in Drosophila melanogaster: a forward genetic screen. Genes Brain Behav. 2006;5:222-39 pubmed
    ..One gene (yuri) located close to a known locus for gravitaxis has been the subject of more extensive analysis including confirmation by transgenic rescue. ..
  2. Lyulcheva E, Taylor E, Michael M, Vehlow A, Tan S, Fletcher A, et al. Drosophila pico and its mammalian ortholog lamellipodin activate serum response factor and promote cell proliferation. Dev Cell. 2008;15:680-90 pubmed publisher
    ..Genetics data indicate that mal/SRF levels are important for pico-mediated tissue growth. We propose that MRL proteins link EGFR activation to mitogenic SRF signaling via changes in actin dynamics. ..
  3. Chen X, Xu L. Specific nucleoporin requirement for Smad nuclear translocation. Mol Cell Biol. 2010;30:4022-34 pubmed publisher
    ..Surprisingly, many of these nucleoporins, including Sec13, Nup75, Nup93, and Nup205, are scaffold nucleoporins considered important for the overall integrity of the nuclear pore complex (NPC) but ..
  4. Militti C, Maenner S, Becker P, Gebauer F. UNR facilitates the interaction of MLE with the lncRNA roX2 during Drosophila dosage compensation. Nat Commun. 2014;5:4762 pubmed publisher
  5. Kusch T. Brca2-Pds5 complexes mobilize persistent meiotic recombination sites to the nuclear envelope. J Cell Sci. 2015;128:717-27 pubmed publisher
    ..The identification of Brca2-Pds5 complexes as key mediators of this process provides a first mechanistic explanation for the contribution of lamins and cohesins to meiotic recombination. ..
  6. Zhang K, Donnelly C, Haeusler A, Grima J, Machamer J, Steinwald P, et al. The C9orf72 repeat expansion disrupts nucleocytoplasmic transport. Nature. 2015;525:56-61 pubmed publisher
    ..Nucleocytoplasmic transport defects may be a fundamental pathway for ALS and FTD that is amenable to pharmacotherapeutic intervention. ..
  7. Ryu T, Spatola B, Delabaere L, Bowlin K, Hopp H, Kunitake R, et al. Heterochromatic breaks move to the nuclear periphery to continue recombinational repair. Nat Cell Biol. 2015;17:1401-11 pubmed publisher

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