E Yavin

Summary

Publications

  1. ncbi Delayed cell migration in the developing rat brain following maternal omega 3 alpha linolenic acid dietary deficiency
    E Yavin
    Department of Neurobiology, Weizmann Institute of Science, Rehovot 76100, Israel
    Neuroscience 162:1011-22. 2009
  2. ncbi Overexpression of dopamine receptor genes and their products in the postnatal rat brain following maternal n-3 fatty acid dietary deficiency
    F Kuperstein
    Department of Neurobiology, Weizmann Institute of Science Rehovot, Israel
    J Neurochem 95:1550-62. 2005
  3. ncbi Docosahexaenoic acid accumulation in the prenatal brain: prooxidant and antioxidant features
    E Yavin
    Department of Neurobiology, Weizmann Institute of Science, Rehovot, Israel
    J Mol Neurosci 16:229-35; discussion 279-84. 2001
  4. ncbi Ethyl docosahexaenoate-associated decrease in fetal brain lipid peroxide production is mediated by activation of prostanoid and nitric oxide pathways
    P Green
    Department of Neurobiology, Weizmann Institute of Science, 76100, Rehovot, Israel
    Biochim Biophys Acta 1531:156-64. 2001
  5. ncbi Improved representation of gene markers on microarray by PCR-Select subtracted cDNA targets
    E Yakubov
    Department of Neurobiology, Weizmann Institute of Science, Rehovot 76100, Israel
    Brain Res Mol Brain Res 137:110-8. 2005
  6. ncbi Enhanced free radical scavenging and decreased lipid peroxidation in the rat fetal brain after treatment with ethyl docosahexaenoate
    P Green
    Department of Neurobiology, Weizmann Institute of Science, Rehovet, Israel
    Biochim Biophys Acta 1532:203-12. 2001
  7. ncbi Docosahexaenoic acid sources for the developing brain during intrauterine life
    E Yavin
    Department of Neurobiology, Weizmann Institute of Science, Rehovot, Israel
    Nutr Health 15:219-24. 2001
  8. ncbi Docosahexaenoic acid: a pluripotent molecule acting as a membrane fluidizer, a cellular antioxidant and a modulator of gene expression
    E Yavin
    Department of Neurobiology, Weizmann Institute of Science, Israel
    Nutr Health 18:261-2. 2006
  9. ncbi Biphasic modulation of protein kinase C and enhanced cell toxicity by amyloid beta peptide and anoxia in neuronal cultures
    F Kuperstein
    Department of Neurobiology, The Weizmann Institute of Science, Rehovot, Israel
    J Neurochem 76:758-67. 2001
  10. ncbi Overexpression of genes in the CA1 hippocampus region of adult rat following episodes of global ischemia
    E Yakubov
    Department of Neurobiology, The Weizmann Institute of Science, Rehovot 76100, Israel
    Brain Res Mol Brain Res 127:10-26. 2004

Collaborators

  • N Salem
  • A Brand
  • E Yakubov
  • F Kuperstein
  • P Green
  • P Dinerman
  • S Gil
  • S Glozman
  • R Eylam
  • S Saban
  • P Fuchs
  • M Gottlieb
  • N Reiss
  • L Weiner
  • N Koudinova

Detail Information

Publications11

  1. ncbi Delayed cell migration in the developing rat brain following maternal omega 3 alpha linolenic acid dietary deficiency
    E Yavin
    Department of Neurobiology, Weizmann Institute of Science, Rehovot 76100, Israel
    Neuroscience 162:1011-22. 2009
    ..Transient obstruction of neuronal cell migration may have long-lasting consequences on the organization of neuronal assemblies, on the connection between neurons (lateral connections) and acquisition of function in the adult brain...
  2. ncbi Overexpression of dopamine receptor genes and their products in the postnatal rat brain following maternal n-3 fatty acid dietary deficiency
    F Kuperstein
    Department of Neurobiology, Weizmann Institute of Science Rehovot, Israel
    J Neurochem 95:1550-62. 2005
    ....
  3. ncbi Docosahexaenoic acid accumulation in the prenatal brain: prooxidant and antioxidant features
    E Yavin
    Department of Neurobiology, Weizmann Institute of Science, Rehovot, Israel
    J Mol Neurosci 16:229-35; discussion 279-84. 2001
    ..Part of the beneficial effect of Et-DHA administration on the fetal brain may be attributed to enhanced free-radical scavenging capability, a phenomenon not directly related to vitamin E or lipid-soluble antioxidant levels...
  4. ncbi Ethyl docosahexaenoate-associated decrease in fetal brain lipid peroxide production is mediated by activation of prostanoid and nitric oxide pathways
    P Green
    Department of Neurobiology, Weizmann Institute of Science, 76100, Rehovot, Israel
    Biochim Biophys Acta 1531:156-64. 2001
    ....
  5. ncbi Improved representation of gene markers on microarray by PCR-Select subtracted cDNA targets
    E Yakubov
    Department of Neurobiology, Weizmann Institute of Science, Rehovot 76100, Israel
    Brain Res Mol Brain Res 137:110-8. 2005
    ..An improved and universal protocol to provide a rapid assessment for gene profiling in biological specimens is indicated...
  6. ncbi Enhanced free radical scavenging and decreased lipid peroxidation in the rat fetal brain after treatment with ethyl docosahexaenoate
    P Green
    Department of Neurobiology, Weizmann Institute of Science, Rehovet, Israel
    Biochim Biophys Acta 1532:203-12. 2001
    ..The data are consistent with the possibility that the neuroprotective effect of Et-DHA might be due to possible free radical scavenging ability of the brain tissue and interference with LPO propagation...
  7. ncbi Docosahexaenoic acid sources for the developing brain during intrauterine life
    E Yavin
    Department of Neurobiology, Weizmann Institute of Science, Rehovot, Israel
    Nutr Health 15:219-24. 2001
    ..Transfer of DHA via the fetal gastrointestinal tract is an additional route to provide DHA to other fetal organs. As indicated by animal model studies, it holds the potential for DHA supply when the maternal pathway is compromised...
  8. ncbi Docosahexaenoic acid: a pluripotent molecule acting as a membrane fluidizer, a cellular antioxidant and a modulator of gene expression
    E Yavin
    Department of Neurobiology, Weizmann Institute of Science, Israel
    Nutr Health 18:261-2. 2006
  9. ncbi Biphasic modulation of protein kinase C and enhanced cell toxicity by amyloid beta peptide and anoxia in neuronal cultures
    F Kuperstein
    Department of Neurobiology, The Weizmann Institute of Science, Rehovot, Israel
    J Neurochem 76:758-67. 2001
    ..The data suggest that a biphasic modulation of protein kinase C and MARCKS by Abeta(1-40) combined with anoxic stress may play a role in Alzheimer's disease pathology...
  10. ncbi Overexpression of genes in the CA1 hippocampus region of adult rat following episodes of global ischemia
    E Yakubov
    Department of Neurobiology, The Weizmann Institute of Science, Rehovot 76100, Israel
    Brain Res Mol Brain Res 127:10-26. 2004
    ..These data indicate that ischemia elicits expression of an array of functional gene clusters that may be used as an index for stress severity and a template for target therapy design...
  11. ncbi Early ethanolamine phospholipid translocation marks stress-induced apoptotic cell death in oligodendroglial cells
    A Brand
    Department of Neurobiology, The Weizmann Institute of Science, Rehovot, Israel
    J Neurochem 78:1208-18. 2001
    ..Prevention of EPG species selective translocation to the outer membrane leaflet by altering phospholipid asymmetry may be important in the mechanism of rescue from cell death...