Zoran Vukadinovic

Summary

Publications

  1. ncbi request reprint Elevated striatal dopamine attenuates nigrothalamic inputs and impairs transthalamic cortico-cortical communication in schizophrenia: A hypothesis
    Zoran Vukadinovic
    University of New Mexico School of Medicine, Department of Psychiatry, United States Electronic address
    Med Hypotheses 84:47-52. 2015
  2. ncbi request reprint NMDA receptor hypofunction and the thalamus in schizophrenia
    Zoran Vukadinovic
    University of New Mexico School of Medicine, Department of Psychiatry, United States Electronic address
    Physiol Behav 131:156-9. 2014
  3. ncbi request reprint Cannabis, psychosis and the thalamus: a theoretical review
    Zoran Vukadinovic
    University of New Mexico School of Medicine, Department of Psychiatry, NM, USA
    Neurosci Biobehav Rev 37:658-67. 2013
  4. ncbi request reprint Abnormalities in thalamic neurophysiology in schizophrenia: could psychosis be a result of potassium channel dysfunction?
    Zoran Vukadinovic
    Montefiore Medical Center, Albert Einstein College of Medicine, Department of Psychiatry and Behavioral Sciences, 111 E 210th Street, Bronx, NY 10467, USA
    Neurosci Biobehav Rev 36:960-8. 2012
  5. ncbi request reprint Sleep abnormalities in schizophrenia may suggest impaired trans-thalamic cortico-cortical communication: towards a dynamic model of the illness
    Zoran Vukadinovic
    Montefiore Medical Center, Albert Einstein College of Medicine, Department of Psychiatry and Behavioral Sciences, Residency Training Program, Bronx, NY, USA
    Eur J Neurosci 34:1031-9. 2011

Detail Information

Publications5

  1. ncbi request reprint Elevated striatal dopamine attenuates nigrothalamic inputs and impairs transthalamic cortico-cortical communication in schizophrenia: A hypothesis
    Zoran Vukadinovic
    University of New Mexico School of Medicine, Department of Psychiatry, United States Electronic address
    Med Hypotheses 84:47-52. 2015
    ..Conversely, dopamine D2 receptor blockers (antipsychotics) may help restore nigrothalamic GABAergic inputs, thereby increasing the burst propensity in the thalamus. ..
  2. ncbi request reprint NMDA receptor hypofunction and the thalamus in schizophrenia
    Zoran Vukadinovic
    University of New Mexico School of Medicine, Department of Psychiatry, United States Electronic address
    Physiol Behav 131:156-9. 2014
    ..Here, we argue that such agents (or the proposed NMDAR hypofunction in schizophrenia) weaken the driver inputs in the HO nuclei, thereby producing a cortico-thalamo-cortical disconnection and impairing sensorimotor integration. ..
  3. ncbi request reprint Cannabis, psychosis and the thalamus: a theoretical review
    Zoran Vukadinovic
    University of New Mexico School of Medicine, Department of Psychiatry, NM, USA
    Neurosci Biobehav Rev 37:658-67. 2013
    ..We also argue that the effects of Δ(9)THC are more detrimental compared with the effects of cannabidiol, as the former may increase the excitability of thalamic neurons by its direct effect on T-type calcium channels...
  4. ncbi request reprint Abnormalities in thalamic neurophysiology in schizophrenia: could psychosis be a result of potassium channel dysfunction?
    Zoran Vukadinovic
    Montefiore Medical Center, Albert Einstein College of Medicine, Department of Psychiatry and Behavioral Sciences, 111 E 210th Street, Bronx, NY 10467, USA
    Neurosci Biobehav Rev 36:960-8. 2012
    ..Pharmacological agents that reduce the excitability of thalamic cells and thereby promote burst firing by and large tend to have antipsychotic effects...
  5. ncbi request reprint Sleep abnormalities in schizophrenia may suggest impaired trans-thalamic cortico-cortical communication: towards a dynamic model of the illness
    Zoran Vukadinovic
    Montefiore Medical Center, Albert Einstein College of Medicine, Department of Psychiatry and Behavioral Sciences, Residency Training Program, Bronx, NY, USA
    Eur J Neurosci 34:1031-9. 2011
    ..These interactions may be impaired in schizophrenia and characterization of their mechanism may constitute a step towards developing a dynamic model of schizophrenia...