Ghada A Soliman

Summary

Affiliation: Western Michigan University
Country: USA

Publications

  1. doi request reprint mTORC1 inhibition via rapamycin promotes triacylglycerol lipolysis and release of free fatty acids in 3T3-L1 adipocytes
    Ghada A Soliman
    Division of Metabolism, Endocrinology, and Diabetes, Department of Medicine, University of Michigan Medical School, 109 Zina Pitcher Place, Ann Arbor, MI 48109 2200, USA
    Lipids 45:1089-100. 2010
  2. pmc Regulation of mTOR complex 1 (mTORC1) by raptor Ser863 and multisite phosphorylation
    Kathryn G Foster
    Department of Cell and Developmental Biology, Division of Metabolism, Endocrinology, and Diabetes, University of Michigan Medical School, Ann Arbor, Michigan 48109 2200, USA
    J Biol Chem 285:80-94. 2010
  3. pmc mTOR Ser-2481 autophosphorylation monitors mTORC-specific catalytic activity and clarifies rapamycin mechanism of action
    Ghada A Soliman
    Department of Cell and Developmental Biology, University of Michigan Medical School, Ann Arbor, Michigan 48109 2200, USA
    J Biol Chem 285:7866-79. 2010
  4. pmc Site-specific mTOR phosphorylation promotes mTORC1-mediated signaling and cell growth
    Hugo A Acosta-Jaquez
    Department of Cell and Developmental Biology, University of Michigan Medical School, 109 Zina Pitcher Place, Ann Arbor, MI 48109 2200, USA
    Mol Cell Biol 29:4308-24. 2009

Collaborators

Detail Information

Publications4

  1. doi request reprint mTORC1 inhibition via rapamycin promotes triacylglycerol lipolysis and release of free fatty acids in 3T3-L1 adipocytes
    Ghada A Soliman
    Division of Metabolism, Endocrinology, and Diabetes, Department of Medicine, University of Michigan Medical School, 109 Zina Pitcher Place, Ann Arbor, MI 48109 2200, USA
    Lipids 45:1089-100. 2010
    ..Moreover, they reveal a novel metabolic function for mTORC1; mTORC1 signaling suppresses lipolysis, thus augmenting TAG storage...
  2. pmc Regulation of mTOR complex 1 (mTORC1) by raptor Ser863 and multisite phosphorylation
    Kathryn G Foster
    Department of Cell and Developmental Biology, Division of Metabolism, Endocrinology, and Diabetes, University of Michigan Medical School, Ann Arbor, Michigan 48109 2200, USA
    J Biol Chem 285:80-94. 2010
    ..Taken together, these data suggest that complex raptor phosphorylation functions as a biochemical rheostat that modulates mTORC1 signaling in accordance with environmental cues...
  3. pmc mTOR Ser-2481 autophosphorylation monitors mTORC-specific catalytic activity and clarifies rapamycin mechanism of action
    Ghada A Soliman
    Department of Cell and Developmental Biology, University of Michigan Medical School, Ann Arbor, Michigan 48109 2200, USA
    J Biol Chem 285:7866-79. 2010
    ....
  4. pmc Site-specific mTOR phosphorylation promotes mTORC1-mediated signaling and cell growth
    Hugo A Acosta-Jaquez
    Department of Cell and Developmental Biology, University of Michigan Medical School, 109 Zina Pitcher Place, Ann Arbor, MI 48109 2200, USA
    Mol Cell Biol 29:4308-24. 2009
    ....