Hind Nassar

Summary

Affiliation: Wayne State University
Country: USA

Publications

  1. ncbi request reprint Pathogenesis of invasive micropapillary carcinoma: role of MUC1 glycoprotein
    Hind Nassar
    Department of Pathology, Wayne State University, Harper University Hospital Detroit, MI 48201, USA
    Mod Pathol 17:1045-50. 2004
  2. ncbi request reprint Carcinomas with micropapillary morphology: clinical significance and current concepts
    Hind Nassar
    Department of Pathology at Wayne State University, Harper University Hospital and the Karmanos Cancer Institute, Detroit, Michigan 48201, USA
    Adv Anat Pathol 11:297-303. 2004
  3. ncbi request reprint Clinicopathologic analysis of solid papillary carcinoma of the breast and associated invasive carcinomas
    Hind Nassar
    Wayne State University Harper Hospital and Karmanos Cancer Institute, Detroit, MI, USA
    Am J Surg Pathol 30:501-7. 2006
  4. doi request reprint Heterogeneity of Bcl-2 expression in metastatic breast carcinoma
    Andrea Proctor Subhawong
    Department of Pathology, The Johns Hopkins Hospital, Baltimore, MD 21231, USA
    Mod Pathol 23:1089-96. 2010
  5. doi request reprint The alternative lengthening of telomeres phenotype in breast carcinoma is associated with HER-2 overexpression
    Andrea Proctor Subhawong
    Department of Pathology, The Johns Hopkins Hospital, Baltimore, MD, USA
    Mod Pathol 22:1423-31. 2009
  6. ncbi request reprint Pathogenesis of colloid (pure mucinous) carcinoma of exocrine organs: Coupling of gel-forming mucin (MUC2) production with altered cell polarity and abnormal cell-stroma interaction may be the key factor in the morphogenesis and indolent behavior of collo
    N Volkan Adsay
    Department of Pathology, Karamanos Cancer Institute and Wayne State University, Detroit, Michigan 48201, USA
    Am J Surg Pathol 27:571-8. 2003
  7. doi request reprint Relationship between molecular subtype of invasive breast carcinoma and expression of gross cystic disease fluid protein 15 and mammaglobin
    Gloria H Lewis
    Department of Pathology, The Johns Hopkins Medical Institutions, Baltimore, MD, USA
    Am J Clin Pathol 135:587-91. 2011
  8. pmc MYC gene amplification is often acquired in lethal distant breast cancer metastases of unamplified primary tumors
    Aatur D Singhi
    Department of Pathology, Johns Hopkins Medical Institutions, Baltimore, MD, USA
    Mod Pathol 25:378-87. 2012
  9. ncbi request reprint Hu/Mu ProtIn oligonucleotide microarray: dual-species array for profiling protease and protease inhibitor gene expression in tumors and their microenvironment
    Donald R Schwartz
    Barbara Ann Karmanos Cancer Institute, Wayne State University, Detroit, Michigan, USA
    Mol Cancer Res 5:443-54. 2007
  10. ncbi request reprint High-grade neuroendocrine carcinoma of the ampulla of vater: a clinicopathologic and immunohistochemical analysis of 14 cases
    Hind Nassar
    Department of Pathology, Memorial Sloan Kettering Cancer Center, New York, NY 10021, USA
    Am J Surg Pathol 29:588-94. 2005

Collaborators

Detail Information

Publications17

  1. ncbi request reprint Pathogenesis of invasive micropapillary carcinoma: role of MUC1 glycoprotein
    Hind Nassar
    Department of Pathology, Wayne State University, Harper University Hospital Detroit, MI 48201, USA
    Mod Pathol 17:1045-50. 2004
    ....
  2. ncbi request reprint Carcinomas with micropapillary morphology: clinical significance and current concepts
    Hind Nassar
    Department of Pathology at Wayne State University, Harper University Hospital and the Karmanos Cancer Institute, Detroit, Michigan 48201, USA
    Adv Anat Pathol 11:297-303. 2004
    ..In this article the author reviews the available literature on tumors displaying a micropapillary component...
  3. ncbi request reprint Clinicopathologic analysis of solid papillary carcinoma of the breast and associated invasive carcinomas
    Hind Nassar
    Wayne State University Harper Hospital and Karmanos Cancer Institute, Detroit, MI, USA
    Am J Surg Pathol 30:501-7. 2006
    ..In conclusion, SPCs are heterogeneous lesions that arise in older women and have an indolent behavior. Lymph node and distant metastases are uncommon and generally limited to cases with (conventional) invasive components...
  4. doi request reprint Heterogeneity of Bcl-2 expression in metastatic breast carcinoma
    Andrea Proctor Subhawong
    Department of Pathology, The Johns Hopkins Hospital, Baltimore, MD 21231, USA
    Mod Pathol 23:1089-96. 2010
    ..Instead, downregulation of bcl-2 expression may occur in the setting of hormone therapy resistance. Our findings call into question the potential utility of anti-bcl-2 therapy in metastatic breast cancer...
  5. doi request reprint The alternative lengthening of telomeres phenotype in breast carcinoma is associated with HER-2 overexpression
    Andrea Proctor Subhawong
    Department of Pathology, The Johns Hopkins Hospital, Baltimore, MD, USA
    Mod Pathol 22:1423-31. 2009
    ..As cancers using the ALT pathway are predicted to be resistant to therapies based on telomerase inhibition, these results may have therapeutic consequences...
  6. ncbi request reprint Pathogenesis of colloid (pure mucinous) carcinoma of exocrine organs: Coupling of gel-forming mucin (MUC2) production with altered cell polarity and abnormal cell-stroma interaction may be the key factor in the morphogenesis and indolent behavior of collo
    N Volkan Adsay
    Department of Pathology, Karamanos Cancer Institute and Wayne State University, Detroit, Michigan 48201, USA
    Am J Surg Pathol 27:571-8. 2003
    ..This distinctive mucin, accumulated in the stroma surrounding the CC cells, may act as a containing factor, slackening the spread of the cells...
  7. doi request reprint Relationship between molecular subtype of invasive breast carcinoma and expression of gross cystic disease fluid protein 15 and mammaglobin
    Gloria H Lewis
    Department of Pathology, The Johns Hopkins Medical Institutions, Baltimore, MD, USA
    Am J Clin Pathol 135:587-91. 2011
    ..Correlation with clinical findings may be needed to exclude the possibility of a metastasis to the breast when BLCs or UTNCs are encountered in a limited sample such as a core biopsy sample...
  8. pmc MYC gene amplification is often acquired in lethal distant breast cancer metastases of unamplified primary tumors
    Aatur D Singhi
    Department of Pathology, Johns Hopkins Medical Institutions, Baltimore, MD, USA
    Mod Pathol 25:378-87. 2012
    ..These observations underscore the importance of MYC in breast cancer progression/metastasis, as well as its relevance as a potential therapeutic target in otherwise incurable metastatic disease...
  9. ncbi request reprint Hu/Mu ProtIn oligonucleotide microarray: dual-species array for profiling protease and protease inhibitor gene expression in tumors and their microenvironment
    Donald R Schwartz
    Barbara Ann Karmanos Cancer Institute, Wayne State University, Detroit, Michigan, USA
    Mol Cancer Res 5:443-54. 2007
    ....
  10. ncbi request reprint High-grade neuroendocrine carcinoma of the ampulla of vater: a clinicopathologic and immunohistochemical analysis of 14 cases
    Hind Nassar
    Department of Pathology, Memorial Sloan Kettering Cancer Center, New York, NY 10021, USA
    Am J Surg Pathol 29:588-94. 2005
    ..Thus, there are differences in the molecular phenotypes of these two types of ampullary carcinoma, supporting the distinction of poorly differentiated carcinomas with a neuroendocrine phenotype from those without...
  11. ncbi request reprint Secondary tumors of the pancreas: an analysis of a surgical and autopsy database and review of the literature
    N Volkan Adsay
    Department of Pathology, The Karmanos Cancer Institute and Wayne State University, Harper Hospital, 3990 John R Street, Detroit, MI 48201, USA
    Virchows Arch 444:527-35. 2004
    ..These are predominantly hematopoietic malignancies or carcinomas of renal or gastric origin. Secondary tumors should be entertained in both the clinical and pathological differential diagnosis of pancreatic neoplasia...
  12. ncbi request reprint Comedo-ductal carcinoma in situ: A paradoxical role for programmed cell death
    Malathy P V Shekhar
    Breast Cancer Program, Karmanos Cancer Institute, Department of Pathology, Wayne State University, Detroit, Michigan, USA
    Cancer Biol Ther 7:1774-82. 2008
    ..It is possible that spontaneous apoptosis facilitates elimination of cells thus permitting expansion and malignant transformation of cancer cells that are resistant to spontaneous apoptosis...
  13. doi request reprint Ductal carcinoma in situ in African American versus Caucasian American women: analysis of clinicopathologic features and outcome
    Hind Nassar
    Department of Pathology, The Johns Hopkins Medical Institutions, Baltimore, MD, USA
    Cancer 115:3181-8. 2009
    ..The characteristics of ductal carcinoma in situ (DCIS) in the AA population have not been extensively studied...
  14. doi request reprint Incidental minimal atypical lobular hyperplasia on core needle biopsy: correlation with findings on follow-up excision
    Andrea Proctor Subhawong
    Department of Pathology, The Johns Hopkins University School of Medicine, Baltimore, MD, USA
    Am J Surg Pathol 34:822-8. 2010
    ..However, management of ALH is controversial. We review our experience with incidental minimal ALH on core biopsy, and correlate with excision and follow-up results...
  15. pmc Most basal-like breast carcinomas demonstrate the same Rb-/p16+ immunophenotype as the HPV-related poorly differentiated squamous cell carcinomas which they resemble morphologically
    Andrea Proctor Subhawong
    Department of Pathology, The Johns Hopkins Medical Institutions, MD 21231 2410, USA
    Am J Surg Pathol 33:163-75. 2009
    ..This subset may represent a more homogenous group than BLC as defined currently...
  16. ncbi request reprint Aberrant expression of X-linked genes RbAp46, Rsk4, and Cldn2 in breast cancer
    Archana Thakur
    Department of Pathology, Karmanos Cancer Institute, Wayne State University, 110 East Warren Avenue, Detroit, MI 48201, USA
    Mol Cancer Res 5:171-81. 2007
    ..The activation status of the X-chromosome may influence the expression levels of X-linked oncogenes or tumor suppressor genes...
  17. ncbi request reprint The major autopsy findings in adult patients after hematopoietic stem cell transplantation
    Said Khayyata
    Department of Pathology, Harper University Hospital, Wayne State University, Detroit, MI, USA
    Ann Transplant 12:11-8. 2007
    ..Hematopoietic stem cell transplantation (HSCT) is a curative treatment option for hematological malignancies, but this treatment can be associated with a mortality risk...