T E Wilson

Summary

Affiliation: Washington University School of Medicine
Country: USA

Publications

  1. ncbi Efficient processing of DNA ends during yeast nonhomologous end joining. Evidence for a DNA polymerase beta (Pol4)-dependent pathway
    T E Wilson
    Department of Pathology, Division of Laboratory Medicine, Washington University School of Medicine, St Louis, Missouri 63110, USA
    J Biol Chem 274:23599-609. 1999
  2. ncbi Identification of the DNA binding site for NGFI-B by genetic selection in yeast
    T E Wilson
    Department of Pathology, Washington University School of Medicine, St. Louis, MO 63110
    Science 252:1296-300. 1991
  3. ncbi Yeast DNA ligase IV mediates non-homologous DNA end joining
    T E Wilson
    Washington University School of Medicine, Division of Laboratory Medicine, Department of Pathology, St Louis, Missouri 63110, USA
    Nature 388:495-8. 1997
  4. ncbi Repair of DNA strand breaks by the overlapping functions of lesion-specific and non-lesion-specific DNA 3' phosphatases
    J R Vance
    Department of Pathology, University of Michigan Medical School, Ann Arbor, Michigan 48109 0602, USA
    Mol Cell Biol 21:7191-8. 2001
  5. ncbi The orphan receptors NGFI-B and steroidogenic factor 1 establish monomer binding as a third paradigm of nuclear receptor-DNA interaction
    T E Wilson
    Department of Pathology, Washington University School of Medicine, St Louis, Missouri 63110
    Mol Cell Biol 13:5794-804. 1993
  6. ncbi Uncoupling of 3'-phosphatase and 5'-kinase functions in budding yeast. Characterization of Saccharomyces cerevisiae DNA 3'-phosphatase (TPP1)
    J R Vance
    Department of Pathology, University of Michigan Medical School, Ann Arbor, Michigan 48109-0602, USA
    J Biol Chem 276:15073-81. 2001
  7. ncbi Activity of DNA ligase IV stimulated by complex formation with XRCC4 protein in mammalian cells
    U Grawunder
    Washington University School of Medicine, Division of Molecular Oncology, Department of Pathology, St Louis, Missouri 63110, USA
    Nature 388:492-5. 1997
  8. ncbi In vivo mutational analysis of the NGFI-A zinc fingers
    T E Wilson
    Department of Pathology, Washington University School of Medicine, St Louis, Missouri 63110
    J Biol Chem 267:3718-24. 1992

Collaborators

Detail Information

Publications8

  1. ncbi Efficient processing of DNA ends during yeast nonhomologous end joining. Evidence for a DNA polymerase beta (Pol4)-dependent pathway
    T E Wilson
    Department of Pathology, Division of Laboratory Medicine, Washington University School of Medicine, St Louis, Missouri 63110, USA
    J Biol Chem 274:23599-609. 1999
    ..Pol4 is thus specifically recruited to perform gap-filling in an NHEJ pathway that must also involve as yet unidentified nucleases...
  2. ncbi Identification of the DNA binding site for NGFI-B by genetic selection in yeast
    T E Wilson
    Department of Pathology, Washington University School of Medicine, St. Louis, MO 63110
    Science 252:1296-300. 1991
    ..Cotransfection experiments in mammalian cells demonstrated that NGFI-B can activate transcription from the NBRE with or without its putative ligand binding domain...
  3. ncbi Yeast DNA ligase IV mediates non-homologous DNA end joining
    T E Wilson
    Washington University School of Medicine, Division of Laboratory Medicine, Department of Pathology, St Louis, Missouri 63110, USA
    Nature 388:495-8. 1997
    ..Thus, S. cerevisiae has two distinct and separate ligation pathways...
  4. ncbi Repair of DNA strand breaks by the overlapping functions of lesion-specific and non-lesion-specific DNA 3' phosphatases
    J R Vance
    Department of Pathology, University of Michigan Medical School, Ann Arbor, Michigan 48109 0602, USA
    Mol Cell Biol 21:7191-8. 2001
    ..Taken together, these results demonstrate a clear role for the lesion-specific enzyme, Tpp1, in the repair of a subset of DNA strand breaks...
  5. ncbi The orphan receptors NGFI-B and steroidogenic factor 1 establish monomer binding as a third paradigm of nuclear receptor-DNA interaction
    T E Wilson
    Department of Pathology, Washington University School of Medicine, St Louis, Missouri 63110
    Mol Cell Biol 13:5794-804. 1993
    ..This monomer-DNA interaction represents a third paradigm of DNA binding by nuclear receptors in addition to direct and inverted dimerization...
  6. ncbi Uncoupling of 3'-phosphatase and 5'-kinase functions in budding yeast. Characterization of Saccharomyces cerevisiae DNA 3'-phosphatase (TPP1)
    J R Vance
    Department of Pathology, University of Michigan Medical School, Ann Arbor, Michigan 48109-0602, USA
    J Biol Chem 276:15073-81. 2001
    ..Repair of transformed dephosphorylated plasmids, and 5'-hydroxyl blocking lesions more generally, likely proceeds by a cycle of base removal and resynthesis...
  7. ncbi Activity of DNA ligase IV stimulated by complex formation with XRCC4 protein in mammalian cells
    U Grawunder
    Washington University School of Medicine, Division of Molecular Oncology, Department of Pathology, St Louis, Missouri 63110, USA
    Nature 388:492-5. 1997
    ....
  8. ncbi In vivo mutational analysis of the NGFI-A zinc fingers
    T E Wilson
    Department of Pathology, Washington University School of Medicine, St Louis, Missouri 63110
    J Biol Chem 267:3718-24. 1992
    ..Surprisingly, not all of the mutations tested significantly impaired NGFI-A-specific DNA binding, suggesting that the function of these zinc fingers is more diverse than previously recognized...