BARBARA SNIDER

Summary

Affiliation: Washington University School of Medicine
Country: USA

Publications

  1. doi request reprint "Will I get Alzheimer disease?" when cognitively normal patients ask to be tested for Alzheimer disease
    B Joy Snider
    Washington University, Campus Box 8111 BT 229, 660 S Euclid Ave, St Louis, MO 63110, USA
    Continuum (Minneap Minn) 19:470-4. 2013
  2. pmc Cerebrospinal fluid biomarkers and rate of cognitive decline in very mild dementia of the Alzheimer type
    Barbara J Snider
    Department of Neurology, Washington University School of Medicine, Campus Box 8111, 660 S Euclid, St Louis, MO 63110, USA
    Arch Neurol 66:638-45. 2009
  3. ncbi request reprint Novel presenilin 1 mutation (S170F) causing Alzheimer disease with Lewy bodies in the third decade of life
    B Joy Snider
    Alzheimer s Disease Research Center, Washington University School of Medicine, St Louis, MO, USA
    Arch Neurol 62:1821-30. 2005
  4. ncbi request reprint Apoptosis and necrosis in cerebrovascular disease
    B J Snider
    Center for the Study of Nervous System Injury, Washington University School of Medicine, St Louis, Missouri 63110, USA
    Ann N Y Acad Sci 893:243-53. 1999
  5. ncbi request reprint Neocortical neurons cultured from mice with expanded CAG repeats in the huntingtin gene: unaltered vulnerability to excitotoxins and other insults
    B J Snider
    Center for the Study of Nervous System Injury and Department of Neurology, Washington University School of Medicine, Campus Box 8111, 660 South Euclid, St Louis, MO 63110, USA
    Neuroscience 120:617-25. 2003
  6. ncbi request reprint NMDA antagonists exacerbate neuronal death caused by proteasome inhibition in cultured cortical and striatal neurons
    B Joy Snider
    Center for the Study of Nervous System Injury and Department of Neurology, Washington University School of Medicine, St Louis, MO, USA
    Eur J Neurosci 15:419-28. 2002
  7. ncbi request reprint Transient encephalopathy in a postoperative non-alcoholic female with Marchiafava-Bignami disease
    Lesli E Rusche-Skolarus
    Department of Neurology, Washington University School of Medicine, St Louis, MO 63110, USA
    Clin Neurol Neurosurg 109:713-5. 2007
  8. pmc Optimal promoter usage for lentiviral vector-mediated transduction of cultured central nervous system cells
    Mingjie Li
    Department of Neurology, Washington University School of Medicine, St Louis, MO 63110, USA
    J Neurosci Methods 189:56-64. 2010
  9. pmc Cellular calcium deficiency plays a role in neuronal death caused by proteasome inhibitors
    Shengzhou Wu
    Laboratory of B Joy Snider, Hope Center for Neurological Disorders, Saint Louis, Missouri 63110, USA
    J Neurochem 109:1225-36. 2009
  10. ncbi request reprint Raising intracellular calcium attenuates neuronal apoptosis triggered by staurosporine or oxygen-glucose deprivation in the presence of glutamate receptor blockade
    Lorella M T Canzoniero
    Center for the Study of Nervous System Injury and Department of Neurology, Washington University School of Medicine, St Louis, MO 63110, USA
    Neurobiol Dis 15:520-8. 2004

Research Grants

Collaborators

Detail Information

Publications15

  1. doi request reprint "Will I get Alzheimer disease?" when cognitively normal patients ask to be tested for Alzheimer disease
    B Joy Snider
    Washington University, Campus Box 8111 BT 229, 660 S Euclid Ave, St Louis, MO 63110, USA
    Continuum (Minneap Minn) 19:470-4. 2013
    ..There is potential risk to the patient in terms of insurability, employability, and psychological consequences. Physicians will face this situation with increasing frequency as the AD biomarker field progresses...
  2. pmc Cerebrospinal fluid biomarkers and rate of cognitive decline in very mild dementia of the Alzheimer type
    Barbara J Snider
    Department of Neurology, Washington University School of Medicine, Campus Box 8111, 660 S Euclid, St Louis, MO 63110, USA
    Arch Neurol 66:638-45. 2009
    ..Cerebrospinal fluid (CSF) levels of Abeta peptide 1-42 (Abeta 42), tau, and phosphorylated tau (ptau) are potential biomarkers of Alzheimer disease...
  3. ncbi request reprint Novel presenilin 1 mutation (S170F) causing Alzheimer disease with Lewy bodies in the third decade of life
    B Joy Snider
    Alzheimer s Disease Research Center, Washington University School of Medicine, St Louis, MO, USA
    Arch Neurol 62:1821-30. 2005
    ..We describe herein a kindred with very early-onset FAD (age, <40 years) with unusual pathological features and a novel mutation in the presenilin 1 (PSEN1) gene (S170F) and review the existing literature on very early-onset FAD...
  4. ncbi request reprint Apoptosis and necrosis in cerebrovascular disease
    B J Snider
    Center for the Study of Nervous System Injury, Washington University School of Medicine, St Louis, Missouri 63110, USA
    Ann N Y Acad Sci 893:243-53. 1999
    ..Neurons may undergo both apoptosis and necrosis after ischemic insults, and thus it may be therapeutically desirable to block both processes...
  5. ncbi request reprint Neocortical neurons cultured from mice with expanded CAG repeats in the huntingtin gene: unaltered vulnerability to excitotoxins and other insults
    B J Snider
    Center for the Study of Nervous System Injury and Department of Neurology, Washington University School of Medicine, Campus Box 8111, 660 South Euclid, St Louis, MO 63110, USA
    Neuroscience 120:617-25. 2003
    ..These observations suggest that expression of expanded polyglutamine-containing huntingtin does not acutely alter the vulnerability of cortical neurons to excitotoxic, oxidative or apoptotic insults...
  6. ncbi request reprint NMDA antagonists exacerbate neuronal death caused by proteasome inhibition in cultured cortical and striatal neurons
    B Joy Snider
    Center for the Study of Nervous System Injury and Department of Neurology, Washington University School of Medicine, St Louis, MO, USA
    Eur J Neurosci 15:419-28. 2002
    ..In particular, concern is raised that glutamate receptor antagonists might exacerbate, rather than attenuate, proteasome inhibition-induced neuronal death...
  7. ncbi request reprint Transient encephalopathy in a postoperative non-alcoholic female with Marchiafava-Bignami disease
    Lesli E Rusche-Skolarus
    Department of Neurology, Washington University School of Medicine, St Louis, MO 63110, USA
    Clin Neurol Neurosurg 109:713-5. 2007
    ..She returned to baseline after vitamin supplementation. We believe it is important to diagnose MBD because it is a potentially reversible encephalopathy...
  8. pmc Optimal promoter usage for lentiviral vector-mediated transduction of cultured central nervous system cells
    Mingjie Li
    Department of Neurology, Washington University School of Medicine, St Louis, MO 63110, USA
    J Neurosci Methods 189:56-64. 2010
    ..These findings may prove useful in the design of lentiviral vectors for use in cell culture models of the nervous system...
  9. pmc Cellular calcium deficiency plays a role in neuronal death caused by proteasome inhibitors
    Shengzhou Wu
    Laboratory of B Joy Snider, Hope Center for Neurological Disorders, Saint Louis, Missouri 63110, USA
    J Neurochem 109:1225-36. 2009
    ..These findings may have implications for the treatment of disorders associated with protein misfolding in which proteasome impairment and programmed cell death may occur...
  10. ncbi request reprint Raising intracellular calcium attenuates neuronal apoptosis triggered by staurosporine or oxygen-glucose deprivation in the presence of glutamate receptor blockade
    Lorella M T Canzoniero
    Center for the Study of Nervous System Injury and Department of Neurology, Washington University School of Medicine, St Louis, MO 63110, USA
    Neurobiol Dis 15:520-8. 2004
    ..Raising [Ca(2+)](i) either by activating voltage-sensitive Ca(2+) channels with (-) Bay K8644 or by application of low concentrations of kainate attenuated both staurosporine and oxygen-glucose deprivation-induced apoptosis...
  11. ncbi request reprint A proteasomal stress response: pre-treatment with proteasome inhibitors increases proteasome activity and reduces neuronal vulnerability to oxidative injury
    Chul Sang Lee
    Center for the Study of Nervous System Injury and Department of Neurology, Washington University School of Medicine, St Louis, Missouri 63110, USA
    J Neurochem 91:996-1006. 2004
    ....
  12. ncbi request reprint Potassium channel blockers attenuate hypoxia- and ischemia-induced neuronal death in vitro and in vivo
    Ling Wei
    Center for the Study of Nervous System Injury and Department of Neurology, Washington University School of Medicine, St Louis, MO 63110, USA
    Stroke 34:1281-6. 2003
    ....
  13. pmc A role for the ubiquitin-proteasome system in activity-dependent presynaptic silencing
    Xiaoping Jiang
    Department of Psychiatry, Washington University School of Medicine, St Louis, Missouri 63110, USA
    J Neurosci 30:1798-809. 2010
    ..These results suggest that modulation of the UPS by electrical activity contributes to persistent presynaptic silencing by promoting the degradation of key presynaptic proteins...
  14. ncbi request reprint Lentiviral transduction of murine oligodendrocytes in vivo
    Sally R McIver
    Hope Center for Neurological Disorders, Washington University School of Medicine, St Louis, Missouri 63110, USA
    J Neurosci Res 82:397-403. 2005
    ....
  15. ncbi request reprint Neurotrophin and GDNF family ligands promote survival and alter excitotoxic vulnerability of neurons derived from murine embryonic stem cells
    Chul Sang Lee
    Washington University School of Medicine, Center for the Study of Nervous System Injury, St Louis, MO 63110, USA
    Exp Neurol 191:65-76. 2005
    ..These findings demonstrate that the response of ES-derived neurons to neurotrophins and GDNF family ligands is largely similar to that of other cultured central neurons...

Research Grants3

  1. Neuroprotective Effects of Enhanced Proteasome Function
    BARBARA SNIDER; Fiscal Year: 2004
    ....
  2. Neuroprotective Effects of Enhanced Proteasome Function
    BARBARA SNIDER; Fiscal Year: 2005
    ....
  3. Neuroprotective Effects of Enhanced Proteasome Function
    BARBARA SNIDER; Fiscal Year: 2006
    ....