J Fadel

Summary

Affiliation: Vanderbilt University
Country: USA

Publications

  1. ncbi request reprint The neurotensin antagonist SR 48692 attenuates haloperidol-induced striatal Fos expression in the rat
    J Fadel
    Departments of Psychiatry and Pharmacology and Center for Molecular Neuroscience, Vanderbilt, University Medical Center, Nashville, TN 37212, USA
    Neurosci Lett 303:17-20. 2001
  2. ncbi request reprint Anatomical substrates of orexin-dopamine interactions: lateral hypothalamic projections to the ventral tegmental area
    J Fadel
    Department of Psychiatry, Vanderbilt University Medical Center, PHV Suite 313, 1601 23rd Avenue South, Nashville, TN 37212, USA
    Neuroscience 111:379-87. 2002
  3. ncbi request reprint Stimulation of cortical acetylcholine release by orexin A
    J Fadel
    Department of Pharmacology, Physiology and Neuroscience, University of South Carolina School of Medicine, 6439 Garners Ferry Road, Columbia, SC 29208, USA
    Neuroscience 130:541-7. 2005
  4. ncbi request reprint Food-elicited increases in cortical acetylcholine release require orexin transmission
    D Frederick-Duus
    Department of Pharmacology, Physiology and Neuroscience, University of South Carolina School of Medicine, 6439 Garners Ferry Road, Columbia, SC 29208, USA
    Neuroscience 149:499-507. 2007
  5. ncbi request reprint Basal forebrain glutamatergic modulation of cortical acetylcholine release
    J Fadel
    Department of Psychology, The Ohio State University, Columbus, Ohio 43210, USA
    Synapse 39:201-12. 2001
  6. ncbi request reprint Amphetamine-stimulated cortical acetylcholine release: role of the basal forebrain
    H M Arnold
    Department of Psychology, 31 Townshend Hall, The Ohio State University, Columbus, OH 43210, USA
    Brain Res 894:74-87. 2001

Collaborators

Detail Information

Publications6

  1. ncbi request reprint The neurotensin antagonist SR 48692 attenuates haloperidol-induced striatal Fos expression in the rat
    J Fadel
    Departments of Psychiatry and Pharmacology and Center for Molecular Neuroscience, Vanderbilt, University Medical Center, Nashville, TN 37212, USA
    Neurosci Lett 303:17-20. 2001
    ..In view of proposed functional roles of the striatal patch and matrix, we suggest that neurotensin may be important in the therapeutic rather than side effects of antipsychotic drugs...
  2. ncbi request reprint Anatomical substrates of orexin-dopamine interactions: lateral hypothalamic projections to the ventral tegmental area
    J Fadel
    Department of Psychiatry, Vanderbilt University Medical Center, PHV Suite 313, 1601 23rd Avenue South, Nashville, TN 37212, USA
    Neuroscience 111:379-87. 2002
    ..We conclude that the lateral hypothalamic/perifornical projection represents an anatomical substrate by which interoceptive-related signals may influence forebrain dopamine function...
  3. ncbi request reprint Stimulation of cortical acetylcholine release by orexin A
    J Fadel
    Department of Pharmacology, Physiology and Neuroscience, University of South Carolina School of Medicine, 6439 Garners Ferry Road, Columbia, SC 29208, USA
    Neuroscience 130:541-7. 2005
    ..Collectively, these data suggest that orexin projections to the basal forebrain form an important link between hypothalamic arousal and forebrain attentional systems...
  4. ncbi request reprint Food-elicited increases in cortical acetylcholine release require orexin transmission
    D Frederick-Duus
    Department of Pharmacology, Physiology and Neuroscience, University of South Carolina School of Medicine, 6439 Garners Ferry Road, Columbia, SC 29208, USA
    Neuroscience 149:499-507. 2007
    ....
  5. ncbi request reprint Basal forebrain glutamatergic modulation of cortical acetylcholine release
    J Fadel
    Department of Psychology, The Ohio State University, Columbus, Ohio 43210, USA
    Synapse 39:201-12. 2001
    ..The specific roles of basal forebrain glutamate receptor subtypes in mediating cortical ACh release differ and depend on the level of activity of basal forebrain cholinergic neurons...
  6. ncbi request reprint Amphetamine-stimulated cortical acetylcholine release: role of the basal forebrain
    H M Arnold
    Department of Psychology, 31 Townshend Hall, The Ohio State University, Columbus, OH 43210, USA
    Brain Res 894:74-87. 2001
    ..These data suggest that amphetamine increases cortical acetylcholine release via a complex neuronal network rather than simply increasing basal forebrain D1 or D2 receptor activity...