Andrew A Bremer

Summary

Affiliation: Vanderbilt University
Country: USA

Publications

  1. pmc Consumption of fructose- but not glucose-sweetened beverages for 10 weeks increases circulating concentrations of uric acid, retinol binding protein-4, and gamma-glutamyl transferase activity in overweight/obese humans
    Chad L Cox
    Department of Molecular Biosciences, School of Veterinary Medicine, University of California, Davis, One Shields Avenue, Davis, CA, 95616, USA
    Nutr Metab (Lond) 9:68. 2012
  2. ncbi request reprint Racial trends in sugar-sweetened beverage consumption among US adolescents: 1988-2004
    Andrew A Bremer
    Department of Pediatrics, Vanderbilt University School of Medicine, Nashville, TN 37232 9170, USA
    Int J Adolesc Med Health 23:279-86. 2011
  3. ncbi request reprint Differences in male and female adolescents from various racial groups in the relationship between insulin resistance-associated parameters with sugar-sweetened beverage intake and physical activity levels
    Andrew A Bremer
    Department of Pediatrics, Division of Endocrinology, Vanderbilt University School of Medicine, Nashville, TN 37232 9170, USA
    Clin Pediatr (Phila) 49:1134-42. 2010
  4. pmc Polycystic ovary syndrome in the pediatric population
    Andrew A Bremer
    Department of Pediatrics, Division of Endocrinology, Vanderbilt University School of Medicine, Nashville, Tennessee 37232 9170, USA
    Metab Syndr Relat Disord 8:375-94. 2010
  5. doi request reprint Relationship between insulin resistance-associated metabolic parameters and anthropometric measurements with sugar-sweetened beverage intake and physical activity levels in US adolescents: findings from the 1999-2004 National Health and Nutrition Examinat
    Andrew A Bremer
    Department of Pediatrics, Division of Endocrinology, University of California Davis School of Medicine, 2516 Stockton Boulevard, Sacramento, CA 95817 2208, USA
    Arch Pediatr Adolesc Med 163:328-35. 2009
  6. pmc Fructose-fed rhesus monkeys: a nonhuman primate model of insulin resistance, metabolic syndrome, and type 2 diabetes
    Andrew A Bremer
    Department of Pediatrics, Vanderbilt University, Nashville, Tennessee, USA
    Clin Transl Sci 4:243-52. 2011
  7. pmc Adipose tissue dysfunction in nascent metabolic syndrome
    Andrew A Bremer
    Department of Pediatrics, Vanderbilt University, Nashville, TN 37232 9170, USA
    J Obes 2013:393192. 2013
  8. ncbi request reprint High frequency of cardiac and behavioral complaints as presenting symptoms of hyperthyroidism in children
    Lindsey A Loomba-Albrecht
    Department of Pediatrics, Division of Endocrinology, University of California Davis Medical Center, Sacramento, CA 95817 2208, USA
    J Pediatr Endocrinol Metab 24:209-13. 2011
  9. pmc Adipose tissue dysregulation in patients with metabolic syndrome
    Andrew A Bremer
    Department of Pediatrics, Vanderbilt University, Nashville, Tennessee 37232, USA
    J Clin Endocrinol Metab 96:E1782-8. 2011
  10. pmc Toward a unifying hypothesis of metabolic syndrome
    Andrew A Bremer
    Department of Pediatrics, Vanderbilt University School of Medicine, Nashville, Tennessee, USA
    Pediatrics 129:557-70. 2012

Collaborators

Detail Information

Publications20

  1. pmc Consumption of fructose- but not glucose-sweetened beverages for 10 weeks increases circulating concentrations of uric acid, retinol binding protein-4, and gamma-glutamyl transferase activity in overweight/obese humans
    Chad L Cox
    Department of Molecular Biosciences, School of Veterinary Medicine, University of California, Davis, One Shields Avenue, Davis, CA, 95616, USA
    Nutr Metab (Lond) 9:68. 2012
    ..abstract:..
  2. ncbi request reprint Racial trends in sugar-sweetened beverage consumption among US adolescents: 1988-2004
    Andrew A Bremer
    Department of Pediatrics, Vanderbilt University School of Medicine, Nashville, TN 37232 9170, USA
    Int J Adolesc Med Health 23:279-86. 2011
    ....
  3. ncbi request reprint Differences in male and female adolescents from various racial groups in the relationship between insulin resistance-associated parameters with sugar-sweetened beverage intake and physical activity levels
    Andrew A Bremer
    Department of Pediatrics, Division of Endocrinology, Vanderbilt University School of Medicine, Nashville, TN 37232 9170, USA
    Clin Pediatr (Phila) 49:1134-42. 2010
    ..To describe racial and gender differences in insulin resistance-associated parameters due to sugar-sweetened beverage (SSB) intake and physical activity (PA) levels in the adolescent population...
  4. pmc Polycystic ovary syndrome in the pediatric population
    Andrew A Bremer
    Department of Pediatrics, Division of Endocrinology, Vanderbilt University School of Medicine, Nashville, Tennessee 37232 9170, USA
    Metab Syndr Relat Disord 8:375-94. 2010
    ..For the syndrome's management, emphasis is placed on lifestyle and symptom-directed treatment...
  5. doi request reprint Relationship between insulin resistance-associated metabolic parameters and anthropometric measurements with sugar-sweetened beverage intake and physical activity levels in US adolescents: findings from the 1999-2004 National Health and Nutrition Examinat
    Andrew A Bremer
    Department of Pediatrics, Division of Endocrinology, University of California Davis School of Medicine, 2516 Stockton Boulevard, Sacramento, CA 95817 2208, USA
    Arch Pediatr Adolesc Med 163:328-35. 2009
    ..To evaluate the relationship between insulin resistance-associated metabolic parameters and anthropometric measurements with sugar-sweetened beverage intake and physical activity levels...
  6. pmc Fructose-fed rhesus monkeys: a nonhuman primate model of insulin resistance, metabolic syndrome, and type 2 diabetes
    Andrew A Bremer
    Department of Pediatrics, Vanderbilt University, Nashville, Tennessee, USA
    Clin Transl Sci 4:243-52. 2011
    ....
  7. pmc Adipose tissue dysfunction in nascent metabolic syndrome
    Andrew A Bremer
    Department of Pediatrics, Vanderbilt University, Nashville, TN 37232 9170, USA
    J Obes 2013:393192. 2013
    ..This adipokine profile may contribute to increased insulin resistance and low-grade inflammation, promoting the increased risk of T2DM and CVD...
  8. ncbi request reprint High frequency of cardiac and behavioral complaints as presenting symptoms of hyperthyroidism in children
    Lindsey A Loomba-Albrecht
    Department of Pediatrics, Division of Endocrinology, University of California Davis Medical Center, Sacramento, CA 95817 2208, USA
    J Pediatr Endocrinol Metab 24:209-13. 2011
    ..Furthermore, difficulties in recognizing atypical presentations may delay diagnosis and increase morbidity...
  9. pmc Adipose tissue dysregulation in patients with metabolic syndrome
    Andrew A Bremer
    Department of Pediatrics, Vanderbilt University, Nashville, Tennessee 37232, USA
    J Clin Endocrinol Metab 96:E1782-8. 2011
    ..Numerous groups have shown increased circulating biomarkers of inflammation in MetS. However, there are scanty data on the cellular sources contributing to this low-grade inflammation...
  10. pmc Toward a unifying hypothesis of metabolic syndrome
    Andrew A Bremer
    Department of Pediatrics, Vanderbilt University School of Medicine, Nashville, Tennessee, USA
    Pediatrics 129:557-70. 2012
    ....
  11. doi request reprint Insulin resistance and metabolic syndrome in the pediatric population
    Rachel A Nelson
    Department of Pediatrics, University of California Davis Medical Center, Sacramento, California 95817 2208, USA
    Metab Syndr Relat Disord 8:1-14. 2010
    ..The need for collaborative changes at the family, school, city, state, and national levels to address the growing prevalence of the metabolic syndrome in the pediatric age group is also reviewed...
  12. doi request reprint A novel glucokinase gene mutation and its effect on glycemic/C-peptide fluctuations in a patient with maturity-onset diabetes of the young type 2
    Lindsey A Loomba-Albrecht
    Department of Pediatrics, Division of Endocrinology, University of California Davis Medical Center, Sacramento, CA 95817 2208, United States
    Diabetes Res Clin Pract 87:e23-5. 2010
    ..We report a case of MODY2 caused by a novel GCK mutation and demonstrate differential glycemic/C-peptide responses to treatment with insulin, no medication, and an oral sulfonylurea...
  13. doi request reprint Investigation of the mechanisms contributing to the compensatory increase in insulin secretion during dexamethasone-induced insulin resistance in rhesus macaques
    Bethany P Cummings
    Department of Molecular Biosciences, School of Veterinary Medicine, University of California Davis, One Shields Avenue, Davis, California 95616, USA
    J Endocrinol 216:207-15. 2013
    ..Furthermore, increases in postprandial GIP secretion with dexamethasone treatment appear to contribute to the effect of dexamethasone treatment to increase insulin secretion...
  14. ncbi request reprint How much is enough? Evaluation of adrenal function in children who undergo growth hormone stimulation test
    Revi P Mathewi
    Ian Burr Division of Pediatric Endocrinology, Vanderbilt University Medical School, Nashville, TN 37232, USA
    J Pediatr Endocrinol Metab 25:733-9. 2012
    ..4 microg/dL. In the event of a suboptimal cortisol response during an ITT, only a single 1-microg CST is recommended for assessment of adrenal function...
  15. doi request reprint Familial hypomagnesemia with hypercalciuria and nephrocalcinosis in three siblings having the same genetic lesion but different clinical presentations
    Hilary H Seeley
    Department of Pediatrics, University of California, San Francisco, California 94143, USA
    World J Pediatr 8:177-80. 2012
    ..This article summarizes the varying clinical manifestations of three siblings with familial hypomagnesemia with hypercalciuria and nephrocalcinosis (FHHNC) caused by the same genetic lesion...
  16. doi request reprint The serine phosphorylation hypothesis of polycystic ovary syndrome: a unifying mechanism for hyperandrogenemia and insulin resistance
    Andrew A Bremer
    Department of Pediatrics, Division of Endocrinology, University of California Davis, Sacramento, California 95817 2208, USA
    Fertil Steril 89:1039-48. 2008
    ..Further defining the molecular mechanisms regulating androgen biosynthesis and insulin action in PCOS patients will permit a better understanding of the syndrome and may lead to the generation of novel specific pharmacologic therapies...
  17. doi request reprint Sexual precocity and its treatment
    Deanna B Brown
    Department of Pediatrics, Vanderbilt University School of Medicine, Nashville, TN 37232, USA
    World J Pediatr 9:103-11. 2013
    ..Sexual precocity is defined as the appearance of secondary sexual characteristics before the lower limit of the normal age for pubertal onset...
  18. pmc What is metabolic syndrome, and why are children getting it?
    Ram Weiss
    Department of Pediatrics, Hadassah Hebrew University School of Medicine, Jerusalem, Israel
    Ann N Y Acad Sci 1281:123-40. 2013
    ..These culminate in an adverse biochemical phenotype, including development of altered glucose metabolism and early atherogenesis during childhood and early adulthood...
  19. ncbi request reprint Pseudohypoaldosteronism type 1 due to a novel mutation in the mineralocorticoid receptor gene
    Lindsey A Loomba-Albrecht
    Department of Pediatrics, Division of Endocrinology, University of California Davis Medical Center, Sacramento, CA 95817 2208, USA
    Horm Res Paediatr 73:482-6. 2010
    ..This report additionally highlights the importance of aldosterone-sensitive sodium transport in the neonatal period...
  20. doi request reprint An oral sulfonylurea in the treatment of transient neonatal diabetes mellitus
    Lindsey A Loomba-Albrecht
    Department of Pediatrics, Division of Endocrinology, University of California Davis Medical Center, Sacramento, California 95817 2208, USA
    Clin Ther 31:816-20. 2009
    ..TNDM can last for a median of 12 weeks and completely resolve by 18 months. Although TNDM is typically treated with subcutaneous insulin, this mode of therapy might be difficult for some caregivers...