Viktor Yarotskyy

Summary

Affiliation: University of Rochester
Country: USA

Publications

  1. doi request reprint Ca(2+) permeation and/or binding to CaV1.1 fine-tunes skeletal muscle Ca(2+) signaling to sustain muscle function
    Chang Seok Lee
    Department of Molecular Physiology and Biophysics, Baylor College of Medicine, One Baylor Plaza, Houston, TX 77030 USA
    Skelet Muscle 5:4. 2015
  2. pmc Monovalent cationic channel activity in the inner membrane of nuclei from skeletal muscle fibers
    Viktor Yarotskyy
    Department of Pharmacology and Physiology, University of Rochester Medical Center, Rochester, New York Electronic address
    Biophys J 107:2027-36. 2014
  3. pmc Roscovitine inhibits CaV3.1 (T-type) channels by preferentially affecting closed-state inactivation
    Viktor Yarotskyy
    Department of Pharmacology and Physiology, University of Rochester Medical Center, 601 Elmwood Avenue, Rochester, NY 14642, USA
    J Pharmacol Exp Ther 340:463-72. 2012
  4. pmc Accelerated activation of SOCE current in myotubes from two mouse models of anesthetic- and heat-induced sudden death
    Viktor Yarotskyy
    Department of Physiology and Pharmacology, University of Rochester Medical Center, Rochester, New York, United States of America
    PLoS ONE 8:e77633. 2013
  5. pmc Temperature and RyR1 regulate the activation rate of store-operated Ca²+ entry current in myotubes
    Viktor Yarotskyy
    Department of Pharmacology and Physiology, University of Rochester Medical Center, Rochester, New York, USA
    Biophys J 103:202-11. 2012
  6. pmc Muscle weakness in myotonic dystrophy associated with misregulated splicing and altered gating of Ca(V)1.1 calcium channel
    Zhen Zhi Tang
    Department of Neurology, University of Rochester Medical Center, Rochester, NY 14642, USA
    Hum Mol Genet 21:1312-24. 2012

Collaborators

  • Feliciano Protasi
  • Krzysztof Sobczak
  • Keith S Elmslie
  • Chang Seok Lee
  • Robert T Dirksen
  • Zhen Zhi Tang
  • George G Rodney
  • Alla Lyfenko
  • Keke Dong
  • Christine Beeton
  • Mary E Dickinson
  • Ross A Poche
  • Adan Dagnino-Acosta
  • Cheng Long
  • Dimitra K Georgiou
  • Susan L Hamilton
  • Pumin Zhang
  • Mark Knoblauch
  • Amy Hanna
  • Iskander I Ismailov
  • Johanna Lanner
  • Ted Tran
  • Michael W Swank
  • Richard T Moxley
  • Masayuki Nakamori
  • Charles A Thornton
  • Lan Wei
  • Katy Eichinger

Detail Information

Publications6

  1. doi request reprint Ca(2+) permeation and/or binding to CaV1.1 fine-tunes skeletal muscle Ca(2+) signaling to sustain muscle function
    Chang Seok Lee
    Department of Molecular Physiology and Biophysics, Baylor College of Medicine, One Baylor Plaza, Houston, TX 77030 USA
    Skelet Muscle 5:4. 2015
    ..1 is not required for skeletal muscle excitation-contraction coupling, but whether Ca(2+) permeation through CaV1.1 during sustained muscle activity plays a functional role in mammalian skeletal muscle has not been assessed...
  2. pmc Monovalent cationic channel activity in the inner membrane of nuclei from skeletal muscle fibers
    Viktor Yarotskyy
    Department of Pharmacology and Physiology, University of Rochester Medical Center, Rochester, New York Electronic address
    Biophys J 107:2027-36. 2014
    ..Together, these studies characterize a voltage-independent NMCC in skeletal muscle, the properties of which are ideally suited to serve as a countercurrent mechanism during calcium release from the nuclear envelope. ..
  3. pmc Roscovitine inhibits CaV3.1 (T-type) channels by preferentially affecting closed-state inactivation
    Viktor Yarotskyy
    Department of Pharmacology and Physiology, University of Rochester Medical Center, 601 Elmwood Avenue, Rochester, NY 14642, USA
    J Pharmacol Exp Ther 340:463-72. 2012
    ..The ability of roscovitine to block multiple mediators of proliferation, including CDKs and Ca(V)3.1 channels, may facilitate its anticancer properties...
  4. pmc Accelerated activation of SOCE current in myotubes from two mouse models of anesthetic- and heat-induced sudden death
    Viktor Yarotskyy
    Department of Physiology and Pharmacology, University of Rochester Medical Center, Rochester, New York, United States of America
    PLoS ONE 8:e77633. 2013
    ....
  5. pmc Temperature and RyR1 regulate the activation rate of store-operated Ca²+ entry current in myotubes
    Viktor Yarotskyy
    Department of Pharmacology and Physiology, University of Rochester Medical Center, Rochester, New York, USA
    Biophys J 103:202-11. 2012
    ....
  6. pmc Muscle weakness in myotonic dystrophy associated with misregulated splicing and altered gating of Ca(V)1.1 calcium channel
    Zhen Zhi Tang
    Department of Neurology, University of Rochester Medical Center, Rochester, NY 14642, USA
    Hum Mol Genet 21:1312-24. 2012
    ..Together, these results indicate that DM-associated splicing defects alter Ca(V)1.1 function, with potential for exacerbation of myopathy...