Julie Wieseler-Frank

Summary

Affiliation: University of Colorado
Country: USA

Publications

  1. ncbi Snake venom phospholipase A2s (Asp49 and Lys49) induce mechanical allodynia upon peri-sciatic administration: involvement of spinal cord glia, proinflammatory cytokines and nitric oxide
    Marucia Chacur
    Instituto Butantan, Sao Paolo, Brazil
    Pain 108:180-91. 2004
  2. ncbi Immune-to-brain communication dynamically modulates pain: physiological and pathological consequences
    Julie Wieseler-Frank
    Department of Psychology and Center for Neuroscience, University of Colorado at Boulder, Boulder, CO 80309 0345, USA
    Brain Behav Immun 19:104-11. 2005
  3. ncbi Central proinflammatory cytokines and pain enhancement
    Julie Wieseler-Frank
    Department of Psychology and Center for Neuroscience, University of Colorado at Boulder, CO 80309 0345, USA
    Neurosignals 14:166-74. 2005
  4. ncbi Controlling neuropathic pain by adeno-associated virus driven production of the anti-inflammatory cytokine, interleukin-10
    Erin D Milligan
    Department of Psychology and the Center for Neuroscience, University of CO at Boulder, Boulder, CO 80309 USA
    Mol Pain 1:9. 2005
  5. ncbi Glial activation and pathological pain
    Julie Wieseler-Frank
    Department of Psychology, Center for Neuroscience, University of Colorado at Boulder, Boulder, CO 80309 0345, USA
    Neurochem Int 45:389-95. 2004
  6. ncbi A role for proinflammatory cytokines and fractalkine in analgesia, tolerance, and subsequent pain facilitation induced by chronic intrathecal morphine
    Ian N Johnston
    Department of Psychology, University of Colorado, Boulder, Colorado 80309, USA
    J Neurosci 24:7353-65. 2004
  7. ncbi A novel immune-to-CNS communication pathway: cells of the meninges surrounding the spinal cord CSF space produce proinflammatory cytokines in response to an inflammatory stimulus
    Julie Wieseler-Frank
    Department of Psychology, University of Colorado, Boulder, CO, USA
    Brain Behav Immun 21:711-8. 2007
  8. ncbi Controlling pathological pain by adenovirally driven spinal production of the anti-inflammatory cytokine, interleukin-10
    Erin D Milligan
    Department of Psychology and the Center for Neuroscience, University of CO at Boulder, Boulder, CO 80309, USA
    Eur J Neurosci 21:2136-48. 2005
  9. ncbi Norman Cousins Lecture. Glia as the "bad guys": implications for improving clinical pain control and the clinical utility of opioids
    Linda R Watkins
    Department of Psychology and the Center for Neuroscience, Muenzinger D 244, Campus Box 345, University of Colorado at Boulder, Boulder, CO 80309 0345, USA
    Brain Behav Immun 21:131-46. 2007
  10. ncbi Spinal gap junctions: potential involvement in pain facilitation
    Leah E Spataro
    Department of Psychology and the Center for Neuroscience, University of Colorado at Boulder, Boulder, Colorado 90309-0345, USA
    J Pain 5:392-405. 2004

Collaborators

Detail Information

Publications12

  1. ncbi Snake venom phospholipase A2s (Asp49 and Lys49) induce mechanical allodynia upon peri-sciatic administration: involvement of spinal cord glia, proinflammatory cytokines and nitric oxide
    Marucia Chacur
    Instituto Butantan, Sao Paolo, Brazil
    Pain 108:180-91. 2004
    ..As a variety of immune cells also produce and release sPLA2s during inflammatory states, the data may have general implications for the understanding of inflammatory pain...
  2. ncbi Immune-to-brain communication dynamically modulates pain: physiological and pathological consequences
    Julie Wieseler-Frank
    Department of Psychology and Center for Neuroscience, University of Colorado at Boulder, Boulder, CO 80309 0345, USA
    Brain Behav Immun 19:104-11. 2005
    ....
  3. ncbi Central proinflammatory cytokines and pain enhancement
    Julie Wieseler-Frank
    Department of Psychology and Center for Neuroscience, University of Colorado at Boulder, CO 80309 0345, USA
    Neurosignals 14:166-74. 2005
    ..Gene therapy to augment the endogenous anti-inflammatory cytokine, interleukin-10, is one of the more promising therapies currently under study...
  4. ncbi Controlling neuropathic pain by adeno-associated virus driven production of the anti-inflammatory cytokine, interleukin-10
    Erin D Milligan
    Department of Psychology and the Center for Neuroscience, University of CO at Boulder, Boulder, CO 80309 USA
    Mol Pain 1:9. 2005
    ..Taken together, these data provide initial support that intrathecal gene therapy to drive the production of IL-10 may prove to be an efficacious treatment for neuropathic pain...
  5. ncbi Glial activation and pathological pain
    Julie Wieseler-Frank
    Department of Psychology, Center for Neuroscience, University of Colorado at Boulder, Boulder, CO 80309 0345, USA
    Neurochem Int 45:389-95. 2004
    ..Moreover, the glial-neuronal interactions discussed here are likely not exclusive to pain, but rather are likely to play significant roles in other behavioral phenomena...
  6. ncbi A role for proinflammatory cytokines and fractalkine in analgesia, tolerance, and subsequent pain facilitation induced by chronic intrathecal morphine
    Ian N Johnston
    Department of Psychology, University of Colorado, Boulder, Colorado 80309, USA
    J Neurosci 24:7353-65. 2004
    ..Taken together, these results suggest that IL-1 and fractalkine are endogenous regulators of morphine analgesia and are involved in the increases in pain sensitivity that occur after chronic opiates...
  7. ncbi A novel immune-to-CNS communication pathway: cells of the meninges surrounding the spinal cord CSF space produce proinflammatory cytokines in response to an inflammatory stimulus
    Julie Wieseler-Frank
    Department of Psychology, University of Colorado, Boulder, CO, USA
    Brain Behav Immun 21:711-8. 2007
    ..The ability of the meninges to release to proinflammatory signals suggests a potential role in the modulation of pain...
  8. ncbi Controlling pathological pain by adenovirally driven spinal production of the anti-inflammatory cytokine, interleukin-10
    Erin D Milligan
    Department of Psychology and the Center for Neuroscience, University of CO at Boulder, Boulder, CO 80309, USA
    Eur J Neurosci 21:2136-48. 2005
    ..These studies support that this gene therapy approach provides an alternative to neuronally focused drug and gene therapies for clinical pain control...
  9. ncbi Norman Cousins Lecture. Glia as the "bad guys": implications for improving clinical pain control and the clinical utility of opioids
    Linda R Watkins
    Department of Psychology and the Center for Neuroscience, Muenzinger D 244, Campus Box 345, University of Colorado at Boulder, Boulder, CO 80309 0345, USA
    Brain Behav Immun 21:131-46. 2007
    ....
  10. ncbi Spinal gap junctions: potential involvement in pain facilitation
    Leah E Spataro
    Department of Psychology and the Center for Neuroscience, University of Colorado at Boulder, Boulder, Colorado 90309-0345, USA
    J Pain 5:392-405. 2004
    ..In addition, it decreased gp120-induced proinflammatory cytokines. This suggests gap junction activation might lead to proinflammatory cytokine release by distantly activated glia...
  11. ncbi HIV-1 gp120 stimulates proinflammatory cytokine-mediated pain facilitation via activation of nitric oxide synthase-I (nNOS)
    Adelina Holguin
    Department of Psychology and the Center for Neuroscience, University of Colorado at Boulder, Boulder, CO 80309, USA
    Pain 110:517-30. 2004
    ..Furthermore, gp120 increases mRNA for IL1 converting enzyme and matrix metalloproteinase-9, enzymes responsible for activation and release of proinflammatory cytokines...
  12. ncbi Snake venom components enhance pain upon subcutaneous injection: an initial examination of spinal cord mediators
    Marucia Chacur
    , Instituto Butantan, Av. Vital Brazil, 1500, , SP, Brazil
    Pain 111:65-76. 2004
    ..Lastly, celecoxib, an inhibitor of cyclooxygenase-2, attenuated sPLA2 actions. These data provide the first evidence of spinal mediators involved in pain facilitation induced by subcutaneous venoms...