D G Warnock

Summary

Affiliation: University of Alabama at Birmingham
Country: USA

Publications

  1. ncbi request reprint The epithelial sodium channel in hypertension
    D G Warnock
    Division of Nephrology, University of Alabama at Birmingham, Birmingham, AL 35294 0007, USA
    Curr Hypertens Rep 1:158-63. 1999
  2. ncbi request reprint Aldosterone-related genetic effects in hypertension
    D G Warnock
    Division of Nephrology, University of Alabama at Birmingham, 1530 3rd Avenue South, THT 647, Birmingham, AL 35294 0007, USA
    Curr Hypertens Rep 2:295-301. 2000
  3. ncbi request reprint Low renin hypertension in the next millennium
    D G Warnock
    Department of Medicine, University of Alabama at Birmingham, 35294 0007, USA
    Semin Nephrol 20:40-6. 2000
  4. ncbi request reprint Hypertension
    D G Warnock
    Department of Medicine, University of Alabama at Birmingham, 35294 0007, USA
    Semin Nephrol 19:374-80. 1999
  5. ncbi request reprint Genetic forms of human hypertension
    D G Warnock
    Departments of Medicine and Physiology and Biophysics, and Nephrology Research and Training Center, University of Alabama at Birmingham, 35294 0007, USA
    Curr Opin Nephrol Hypertens 10:493-9. 2001
  6. ncbi request reprint Liddle syndrome: genetics and mechanisms of Na+ channel defects
    D G Warnock
    Department of Medicine, University of Alabama at Birmingham, UAB Station, 35294 0007, USA
    Am J Med Sci 322:302-7. 2001
  7. ncbi request reprint The human angiotensinase C gene (HUMPCP) maps to 11q14 within 700 kb of D11S901: a candidate gene for essential hypertension
    B Watson
    Department of Medicine, University of Albama School of Medicine, Birmingham 35294 0007, USA
    Genomics 44:365-7. 1997
  8. ncbi request reprint Genetic association of 11 beta-hydroxysteroid dehydrogenase type 2 (HSD11B2) flanking microsatellites with essential hypertension in blacks
    B Watson
    Division of Nephrology, University of Alabama School of Medicine, Birmingham
    Hypertension 28:478-82. 1996
  9. ncbi request reprint Disorders of the epithelial Na(+) channel in Liddle's syndrome and autosomal recessive pseudohypoaldosteronism type 1
    Y S Oh
    Department of Medicine, Nephrology Research and Training Center, University of Alabama at Birmingham, 35294, USA
    Exp Nephrol 8:320-5. 2000
  10. ncbi request reprint Prevention, protection, and the intrarenal renin-angiotensin systems
    D G Warnock
    Division of Nephrology, Department of Medicine, Nephrology Research and Training Center, University of Alabama at Birmingham, UAB Station, Birmingham, AL 35294 0006, USA
    Semin Nephrol 21:593-602. 2001

Collaborators

Detail Information

Publications16

  1. ncbi request reprint The epithelial sodium channel in hypertension
    D G Warnock
    Division of Nephrology, University of Alabama at Birmingham, Birmingham, AL 35294 0007, USA
    Curr Hypertens Rep 1:158-63. 1999
    ..As a result, we now have clearly defined syndromes in which ENaC activity is dysregulated with subsequent development of disorders of systemic blood pressure that can be attributed to a primary renal mechanism...
  2. ncbi request reprint Aldosterone-related genetic effects in hypertension
    D G Warnock
    Division of Nephrology, University of Alabama at Birmingham, 1530 3rd Avenue South, THT 647, Birmingham, AL 35294 0007, USA
    Curr Hypertens Rep 2:295-301. 2000
    ..Applying these insights to the far more common disorder of low-renin hypertension may shed new light on the underlying pathophysiology of this common form of human hypertension...
  3. ncbi request reprint Low renin hypertension in the next millennium
    D G Warnock
    Department of Medicine, University of Alabama at Birmingham, 35294 0007, USA
    Semin Nephrol 20:40-6. 2000
    ....
  4. ncbi request reprint Hypertension
    D G Warnock
    Department of Medicine, University of Alabama at Birmingham, 35294 0007, USA
    Semin Nephrol 19:374-80. 1999
    ....
  5. ncbi request reprint Genetic forms of human hypertension
    D G Warnock
    Departments of Medicine and Physiology and Biophysics, and Nephrology Research and Training Center, University of Alabama at Birmingham, 35294 0007, USA
    Curr Opin Nephrol Hypertens 10:493-9. 2001
    ....
  6. ncbi request reprint Liddle syndrome: genetics and mechanisms of Na+ channel defects
    D G Warnock
    Department of Medicine, University of Alabama at Birmingham, UAB Station, 35294 0007, USA
    Am J Med Sci 322:302-7. 2001
    ..The focus of the current review is on Liddle syndrome ("pseudoaldosteronism")...
  7. ncbi request reprint The human angiotensinase C gene (HUMPCP) maps to 11q14 within 700 kb of D11S901: a candidate gene for essential hypertension
    B Watson
    Department of Medicine, University of Albama School of Medicine, Birmingham 35294 0007, USA
    Genomics 44:365-7. 1997
  8. ncbi request reprint Genetic association of 11 beta-hydroxysteroid dehydrogenase type 2 (HSD11B2) flanking microsatellites with essential hypertension in blacks
    B Watson
    Division of Nephrology, University of Alabama School of Medicine, Birmingham
    Hypertension 28:478-82. 1996
    ..Confirmation of our findings in another independently ascertained group of hypertensive subjects will provide a basis for proceeding with sib-pair linkage analyses...
  9. ncbi request reprint Disorders of the epithelial Na(+) channel in Liddle's syndrome and autosomal recessive pseudohypoaldosteronism type 1
    Y S Oh
    Department of Medicine, Nephrology Research and Training Center, University of Alabama at Birmingham, 35294, USA
    Exp Nephrol 8:320-5. 2000
    ..In this review, our current understanding about ENaC and the pathophysiology of Liddle's syndrome and PHA-1 caused by ENaC mutations will be discussed...
  10. ncbi request reprint Prevention, protection, and the intrarenal renin-angiotensin systems
    D G Warnock
    Division of Nephrology, Department of Medicine, Nephrology Research and Training Center, University of Alabama at Birmingham, UAB Station, Birmingham, AL 35294 0006, USA
    Semin Nephrol 21:593-602. 2001
    ..Whether or not inhibition of these pathways is involved in the renoprotective effects of ACE inhibitors and angiotensin receptor antagonists is an important issue to be addressed...
  11. ncbi request reprint Simplified citrate anticoagulation for continuous renal replacement therapy
    A J Tolwani
    Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Alabama 35294 0007, USA
    Kidney Int 60:370-4. 2001
    ..However, because of the metabolic complications of trisodium citrate, it is a complicated technique requiring specialized dialysis solutions. We have designed a simplified protocol for citrate regional anticoagulation for CRRT...
  12. pmc Membranous glomerulopathy in an adult patient with X-linked agammaglobulinemia receiving intravenous gammaglobulin
    L M Endo
    Department of Pediatrics, University of Alabama at Birmingham, Birmingham, Alabama 35294 2182, USA
    J Investig Allergol Clin Immunol 21:405-9. 2011
    ..Immune complex deposition in the subepithelial zone of glomerular capillaries can lead to membranous glomerulopathy...
  13. ncbi request reprint Interaction of syntaxins with the amiloride-sensitive epithelial sodium channel
    S Saxena
    Nephrology Research and Training Center, University of Alabama at Birmingham, Birmingham, Alabama 35294, USA
    J Biol Chem 274:20812-7. 1999
    ....
  14. ncbi request reprint Inverse relationship of urinary cyclic GMP to blood pressure reactivity in the CARDIA study: vasodilatory regulation of sympathetic vasoconstriction. Coronary Artery Risk Development in Young Adults
    J H Markovitz
    Department of Medicine, University of Alabama at Birmingham, 35205, USA
    Psychosom Med 60:319-26. 1998
    ....
  15. ncbi request reprint Cloning and functional studies of splice variants of the alpha-subunit of the amiloride-sensitive Na+ channel
    J K Tucker
    Department of Medicine, University of Alabama, Birmingham 35294, USA
    Am J Physiol 274:C1081-9. 1998
    ..These findings indicate that the extracellular domain is important to generate structural and functional diversity of h alpha ENaC and that alternative splicing may play a role in regulating hENaC activity...
  16. ncbi request reprint Liddle's syndrome: heritable human hypertension caused by mutations in the beta subunit of the epithelial sodium channel
    R A Shimkets
    Howard Hughes Medical Institute, Boyer Center for Molecular Medicine, Yale University School of Medicine, New Haven, Connecticut 06510
    Cell 79:407-14. 1994
    ..These findings demonstrate that Liddle's syndrome is caused by mutations in the beta subunit of the epithelial sodium channel and have implications for the regulation of this epithelial ion channel as well as blood pressure homeostasis...