J J Waite

Summary

Affiliation: University of California
Country: USA

Publications

  1. ncbi Deficit in selective and divided attention associated with cholinergic basal forebrain immunotoxic lesion produced by 192-saporin; motoric/sensory deficit associated with Purkinje cell immunotoxic lesion produced by OX7-saporin
    J J Waite
    Department of Neurosciences, MC 9151, University of California at San Diego, San Diego, California, 92093, USA
    Neurobiol Learn Mem 71:325-52. 1999
  2. ncbi Differential changes in rat cholinergic parameters subsequent to immunotoxic lesion of the basal forebrain nuclei
    J J Waite
    Department of Neurosciences, 9151, University of California at San Diego, La Jolla, CA 92093, USA
    Brain Res 918:113-20. 2001

Detail Information

Publications2

  1. ncbi Deficit in selective and divided attention associated with cholinergic basal forebrain immunotoxic lesion produced by 192-saporin; motoric/sensory deficit associated with Purkinje cell immunotoxic lesion produced by OX7-saporin
    J J Waite
    Department of Neurosciences, MC 9151, University of California at San Diego, San Diego, California, 92093, USA
    Neurobiol Learn Mem 71:325-52. 1999
    ..The cholinergic basal forebrain lesion may mask some of the effects of cerebellar damage up to a threshold after which effects of Purkinje cell loss predominate when 192-saporin is administered intraventricularly...
  2. ncbi Differential changes in rat cholinergic parameters subsequent to immunotoxic lesion of the basal forebrain nuclei
    J J Waite
    Department of Neurosciences, 9151, University of California at San Diego, La Jolla, CA 92093, USA
    Brain Res 918:113-20. 2001
    ..Residual cholinergic terminals in the hippocampus, but not frontal cortex, compensate for a selective cholinergic lesion by increasing the rate of synthesis and may thereby alleviate hippocampus-dependent behavioral deficits...