Rit Vatsyayan

Summary

Affiliation: University of Texas Health Center
Country: USA

Publications

  1. pmc 4-Hydroxy-2-nonenal enhances tissue factor activity in human monocytic cells via p38 mitogen-activated protein kinase activation-dependent phosphatidylserine exposure
    Rit Vatsyayan
    Department of Cellular and Molecular Biology, The University of Texas Health Science Center, Tyler, TX 75708, USA
    Arterioscler Thromb Vasc Biol 33:1601-11. 2013
  2. pmc Role of tissue factor in Mycobacterium tuberculosis-induced inflammation and disease pathogenesis
    Hema Kothari
    Department of Cellular and Molecular Biology, The University of Texas Health Science Center at Tyler, Tyler, TX 75708, United States of America
    PLoS ONE 9:e114141. 2014
  3. pmc Inactivation of factor VIIa by antithrombin in vitro, ex vivo and in vivo: role of tissue factor and endothelial cell protein C receptor
    Rit Vatsyayan
    Department of Cellular and Molecular Biology, The University of Texas Health Science Center at Tyler, Tyler, Texas, United States of America
    PLoS ONE 9:e103505. 2014

Detail Information

Publications3

  1. pmc 4-Hydroxy-2-nonenal enhances tissue factor activity in human monocytic cells via p38 mitogen-activated protein kinase activation-dependent phosphatidylserine exposure
    Rit Vatsyayan
    Department of Cellular and Molecular Biology, The University of Texas Health Science Center, Tyler, TX 75708, USA
    Arterioscler Thromb Vasc Biol 33:1601-11. 2013
    ..The objective of the present study is to investigate the effect of HNE on tissue factor (TF) procoagulant activity expressed on cell surfaces...
  2. pmc Role of tissue factor in Mycobacterium tuberculosis-induced inflammation and disease pathogenesis
    Hema Kothari
    Department of Cellular and Molecular Biology, The University of Texas Health Science Center at Tyler, Tyler, TX 75708, United States of America
    PLoS ONE 9:e114141. 2014
    ..Our data indicate, in contrast to that observed in acute bacterial infections, that TF-mediated coagulation and/or signaling does not appear to contribute to the host-defense in experimental tuberculosis. ..
  3. pmc Inactivation of factor VIIa by antithrombin in vitro, ex vivo and in vivo: role of tissue factor and endothelial cell protein C receptor
    Rit Vatsyayan
    Department of Cellular and Molecular Biology, The University of Texas Health Science Center at Tyler, Tyler, Texas, United States of America
    PLoS ONE 9:e103505. 2014
    ..However, TF's role in AT inactivation of FVIIa appears to be minor and other factor(s) present in plasma, on blood cells or vascular endothelium may play a predominant role in this process...