T E Thiele

Summary

Affiliation: University of Washington
Country: USA

Publications

  1. ncbi request reprint Ethanol-induced c-fos expression in catecholamine- and neuropeptide Y-producing neurons in rat brainstem
    T E Thiele
    Department of Psychology and the Alcohol and Drug Abuse Institute, University of Washington, Seattle 98195, USA
    Alcohol Clin Exp Res 24:802-9. 2000
  2. ncbi request reprint Central infusion of GLP-1, but not leptin, produces conditioned taste aversions in rats
    T E Thiele
    Department of Psychology, University of Washington, Seattle 98195, USA
    Am J Physiol 272:R726-30. 1997
  3. ncbi request reprint Ethanol-induced c-Fos expression in rat lines selected for low and high alcohol consumption
    T E Thiele
    Department of Psychology, University of Washington, Seattle 98195, USA
    Brain Res 756:278-82. 1997
  4. ncbi request reprint Learned tolerance to ethanol-induced c-Fos expression in rats
    T E Thiele
    Department of Psychology, University of Washington, Seattle 98195, USA
    Behav Neurosci 112:193-8. 1998
  5. ncbi request reprint Central infusion of glucagon-like peptide-1-(7-36) amide (GLP-1) receptor antagonist attenuates lithium chloride-induced c-Fos induction in rat brainstem
    T E Thiele
    Department of Psychology, University of Washington, Box 351525, Seattle, WA, USA
    Brain Res 801:164-70. 1998
  6. ncbi request reprint Neurobiological responses to ethanol in mutant mice lacking neuropeptide Y or the Y5 receptor
    T E Thiele
    Department of Psychology, University of Washington, Box 351525, Seattle, WA 98195, USA
    Pharmacol Biochem Behav 67:683-91. 2000
  7. ncbi request reprint Ethanol consumption and resistance are inversely related to neuropeptide Y levels
    T E Thiele
    Department of Psychology, University of Washington, Seattle 98195, USA
    Nature 396:366-9. 1998
  8. ncbi request reprint High ethanol consumption and low sensitivity to ethanol-induced sedation in protein kinase A-mutant mice
    T E Thiele
    Department of Psychology, University of Washington, Seattle, Washington 98195, USA
    J Neurosci 20:RC75. 2000
  9. ncbi request reprint Central infusions of leptin and GLP-1-(7-36) amide differentially stimulate c-FLI in the rat brain
    G van Dijk
    Department of Psychology, University of Washington, Seattle 98195 1525, USA
    Am J Physiol 271:R1096-100. 1996
  10. pmc Deletion of agouti-related protein blunts ethanol self-administration and binge-like drinking in mice
    M Navarro
    Department of Psychology, University of North Carolina, Chapel Hill, NC 27599 3270, USA
    Genes Brain Behav 8:450-8. 2009

Detail Information

Publications16

  1. ncbi request reprint Ethanol-induced c-fos expression in catecholamine- and neuropeptide Y-producing neurons in rat brainstem
    T E Thiele
    Department of Psychology and the Alcohol and Drug Abuse Institute, University of Washington, Seattle 98195, USA
    Alcohol Clin Exp Res 24:802-9. 2000
    ..In the present study we used double-labeling procedures to identify the neurochemical phenotype of neurons that showed ethanol-induced cFLI in the rat brainstem...
  2. ncbi request reprint Central infusion of GLP-1, but not leptin, produces conditioned taste aversions in rats
    T E Thiele
    Department of Psychology, University of Washington, Seattle 98195, USA
    Am J Physiol 272:R726-30. 1997
    ..Hence, the results indicate that central GLP-1 produces aversive side effects, and it is argued that these nonspecific effects may explain the anorectic actions of GLP-1...
  3. ncbi request reprint Ethanol-induced c-Fos expression in rat lines selected for low and high alcohol consumption
    T E Thiele
    Department of Psychology, University of Washington, Seattle 98195, USA
    Brain Res 756:278-82. 1997
    ..0 g ethanol/kg. These data point to the LC as an area which may play a role in the differences in voluntary ethanol consumption between rat lines genetically bred for low and high ethanol preference...
  4. ncbi request reprint Learned tolerance to ethanol-induced c-Fos expression in rats
    T E Thiele
    Department of Psychology, University of Washington, Seattle 98195, USA
    Behav Neurosci 112:193-8. 1998
    ..Results suggest that tolerance to ethanol, as indexed by c-Fos expression in the PVN and the LC, is mediated in part by Pavlovian conditioned responses to cues that predict ethanol administration...
  5. ncbi request reprint Central infusion of glucagon-like peptide-1-(7-36) amide (GLP-1) receptor antagonist attenuates lithium chloride-induced c-Fos induction in rat brainstem
    T E Thiele
    Department of Psychology, University of Washington, Box 351525, Seattle, WA, USA
    Brain Res 801:164-70. 1998
    ..These results suggest that LiCl-induced c-Fos expression in the rat brainstem is mediated, at least in part, by GLP-1 receptor signaling...
  6. ncbi request reprint Neurobiological responses to ethanol in mutant mice lacking neuropeptide Y or the Y5 receptor
    T E Thiele
    Department of Psychology, University of Washington, Box 351525, Seattle, WA 98195, USA
    Pharmacol Biochem Behav 67:683-91. 2000
    ....
  7. ncbi request reprint Ethanol consumption and resistance are inversely related to neuropeptide Y levels
    T E Thiele
    Department of Psychology, University of Washington, Seattle 98195, USA
    Nature 396:366-9. 1998
    ..These data are direct evidence that alcohol consumption and resistance are inversely related to NPY levels in the brain...
  8. ncbi request reprint High ethanol consumption and low sensitivity to ethanol-induced sedation in protein kinase A-mutant mice
    T E Thiele
    Department of Psychology, University of Washington, Seattle, Washington 98195, USA
    J Neurosci 20:RC75. 2000
    ..These data demonstrate a role for the RIIbeta subunit of PKA in regulating voluntary consumption of alcohol and sensitivity to the intoxication effects that are produced by this drug...
  9. ncbi request reprint Central infusions of leptin and GLP-1-(7-36) amide differentially stimulate c-FLI in the rat brain
    G van Dijk
    Department of Psychology, University of Washington, Seattle 98195 1525, USA
    Am J Physiol 271:R1096-100. 1996
    ....
  10. pmc Deletion of agouti-related protein blunts ethanol self-administration and binge-like drinking in mice
    M Navarro
    Department of Psychology, University of North Carolina, Chapel Hill, NC 27599 3270, USA
    Genes Brain Behav 8:450-8. 2009
    ..These observations with genetically altered mice are consistent with previous pharmacological data and suggest that endogenous AgRP signaling modulates the reinforcing properties of ethanol and binge-like ethanol drinking...
  11. pmc Corticotropin releasing factor-1 receptor antagonist, CP-154,526, blocks the expression of ethanol-induced behavioral sensitization in DBA/2J mice
    J R Fee
    Department of Psychology, University of North Carolina, Davie Hall, CB 3270, Chapel Hill, NC 27599 3270, USA
    Neuroscience 150:14-21. 2007
    ..p. injection of CP-154,526, a selective corticotropin releasing factor (CRF) type-1 receptor antagonist, would block the acquisition and/or expression of ethanol-induced locomotor sensitization in male DBA/2J mice...
  12. pmc Involvement of protein kinase A in ethanol-induced locomotor activity and sensitization
    J R Fee
    Department of Psychology, University of North Carolina, Davie Hall, Chapel Hill, NC 27599 3270, USA
    Neuroscience 140:21-31. 2006
    ..To address this question, we examined the effects of repeated ethanol administration on locomotor activity RIIbeta(-/-) and littermate wild-type (RIIbeta(+/+)) mice on multiple genetic backgrounds...
  13. ncbi request reprint Dopamine mediation of the feeding response to violations of spatial and temporal expectancies
    M F Roitman
    Program in Neurobiology and Behavior, University of Washington, Box 351525, Guthrie Hall, Seattle, WA 98195, USA
    Behav Brain Res 122:193-9. 2001
    ..These findings support the idea that DA signals errors in expectancy and that DA signaling is necessary for certain behavioral responses to unexpected events...
  14. ncbi request reprint Ethanol consumption in rats selectively bred for differential saccharin intake
    N K Dess
    Department of Psychology, Occidental College, Los Angeles, CA 90041, USA
    Alcohol 16:275-8. 1998
    ..low saccharin consumption (Occidental HiS and LoS lines). HiS rats consumed more ethanol than did LoS rats. These results support the idea that individual differences in ethanol and saccharin consumption share some common mechanism(s)...
  15. pmc The protective effects of the melanocortin receptor (MCR) agonist, melanotan-II (MTII), against binge-like ethanol drinking are facilitated by deletion of the MC3 receptor in mice
    J J Olney
    Department of Psychology, University of North Carolina, Chapel Hill, NC, USA
    Neuropeptides 48:47-51. 2014
    ..These data suggest that MC3Rs oppose the protective effects of MTII against binge-like ethanol drinking, and thus selective MC3R antagonists may have potential therapeutic roles in treating excessive ethanol drinking...
  16. ncbi request reprint Approaches to understanding the neurobiological regulation of ethanol self-administration: a young investigators forum
    C L Czachowski
    Center for the Neurobehavioral Study of Alcohol, Department of Physiology and Pharmacology, Wake Forest University School of Medicine, Winston-Salem, North Carolina, USA
    Alcohol Clin Exp Res 25:293-8. 2001
    ..Thiele; and (5) Neurochemical characteristics that may predict ethanol preference in selected P and NP rats: A quantitative microdialysis study, by Simon N. Katner...