Inna E Tchivileva

Summary

Affiliation: University of North Carolina
Country: USA

Publications

  1. pmc Characterization of NF-kB-mediated inhibition of catechol-O-methyltransferase
    Inna E Tchivileva
    Center for Neurosensory Disorders, School of Dentistry, University of North Carolina, Chapel Hill, NC 27599 7455, USA
    Mol Pain 5:13. 2009
  2. pmc Signaling pathways mediating beta3-adrenergic receptor-induced production of interleukin-6 in adipocytes
    Inna E Tchivileva
    The Center for Neurosensory Disorders, School of Dentistry, University of North Carolina, Chapel Hill, NC 27599, USA
    Mol Immunol 46:2256-66. 2009
  3. pmc Effect of catechol-O-methyltransferase polymorphism on response to propranolol therapy in chronic musculoskeletal pain: a randomized, double-blind, placebo-controlled, crossover pilot study
    Inna E Tchivileva
    Center for Neurosensory Disorders, School of Dentistry, University of North Carolina, Chapel Hill, NC 27599 7450, USA
    Pharmacogenet Genomics 20:239-48. 2010
  4. ncbi request reprint Genetic architecture of human pain perception
    Luda Diatchenko
    Center for Neurosensory Disorders, University of North Carolina, 2190 Old Dental Building, Chapel Hill, NC 27599, USA
    Trends Genet 23:605-13. 2007
  5. pmc Facial pain with localized and widespread manifestations: separate pathways of vulnerability
    Gary D Slade
    Regional Center for Neurosensory Disorders, University of North Carolina at Chapel Hill, Chapel Hill, NC, USA Department of Dental Ecology, University of North Carolina at Chapel Hill, Chapel Hill, NC, USA Department of Epidemiology, University of North Carolina at Chapel Hill, Chapel Hill, NC, USA Electronic address
    Pain 154:2335-43. 2013

Detail Information

Publications5

  1. pmc Characterization of NF-kB-mediated inhibition of catechol-O-methyltransferase
    Inna E Tchivileva
    Center for Neurosensory Disorders, School of Dentistry, University of North Carolina, Chapel Hill, NC 27599 7455, USA
    Mol Pain 5:13. 2009
    ..Specifically, low COMT activity is associated with heightened pain perception and development of musculoskeletal pain in humans as well as increased experimental pain sensitivity in rodents...
  2. pmc Signaling pathways mediating beta3-adrenergic receptor-induced production of interleukin-6 in adipocytes
    Inna E Tchivileva
    The Center for Neurosensory Disorders, School of Dentistry, University of North Carolina, Chapel Hill, NC 27599, USA
    Mol Immunol 46:2256-66. 2009
    ....
  3. pmc Effect of catechol-O-methyltransferase polymorphism on response to propranolol therapy in chronic musculoskeletal pain: a randomized, double-blind, placebo-controlled, crossover pilot study
    Inna E Tchivileva
    Center for Neurosensory Disorders, School of Dentistry, University of North Carolina, Chapel Hill, NC 27599 7450, USA
    Pharmacogenet Genomics 20:239-48. 2010
    ..We hypothesized that the nonselective beta-adrenergic antagonist propranolol will reduce clinical and experimental pain in TMD patients in a manner dependent on the individuals' COMT diplotype...
  4. ncbi request reprint Genetic architecture of human pain perception
    Luda Diatchenko
    Center for Neurosensory Disorders, University of North Carolina, 2190 Old Dental Building, Chapel Hill, NC 27599, USA
    Trends Genet 23:605-13. 2007
    ..We propose how both rare deleterious genetic variants and common genetic polymorphisms are mediators of human pain perception and clinical pain phenotypes...
  5. pmc Facial pain with localized and widespread manifestations: separate pathways of vulnerability
    Gary D Slade
    Regional Center for Neurosensory Disorders, University of North Carolina at Chapel Hill, Chapel Hill, NC, USA Department of Dental Ecology, University of North Carolina at Chapel Hill, Chapel Hill, NC, USA Department of Epidemiology, University of North Carolina at Chapel Hill, Chapel Hill, NC, USA Electronic address
    Pain 154:2335-43. 2013
    ..9 E-08), although the result was not significant in the replication cohort. These findings illustrate potential for clinical classification of chronic pain based on distinct molecular profiles and genetic background. ..