Jonathan P Sokol

Summary

Affiliation: University of Wisconsin-Madison
Country: USA

Publications

  1. pmc The use of cystatin C to inhibit epithelial-mesenchymal transition and morphological transformation stimulated by transforming growth factor-beta
    Jonathan P Sokol
    Program in Cell Biology, Department of Pediatrics, National Jewish Medical and Research Center, Denver, CO, USA
    Breast Cancer Res 7:R844-53. 2005
  2. ncbi Cystatin C antagonizes transforming growth factor beta signaling in normal and cancer cells
    Jonathan P Sokol
    Program in Cell Biology, Department of Pediatrics, National Jewish Medical and Research Center, Denver, CO 80206, USA
    Mol Cancer Res 2:183-95. 2004

Collaborators

Detail Information

Publications2

  1. pmc The use of cystatin C to inhibit epithelial-mesenchymal transition and morphological transformation stimulated by transforming growth factor-beta
    Jonathan P Sokol
    Program in Cell Biology, Department of Pediatrics, National Jewish Medical and Research Center, Denver, CO, USA
    Breast Cancer Res 7:R844-53. 2005
    ..We hypothesized that the oncogenic activities of TGF-beta, particularly its stimulation of mammary epithelial-mesenchymal transition (EMT), can be prevented by CystC...
  2. ncbi Cystatin C antagonizes transforming growth factor beta signaling in normal and cancer cells
    Jonathan P Sokol
    Program in Cell Biology, Department of Pediatrics, National Jewish Medical and Research Center, Denver, CO 80206, USA
    Mol Cancer Res 2:183-95. 2004
    ..Collectively, our findings have identified CystC as a novel TGF-beta receptor antagonist, as well as a novel CystC-mediated feedback loop that inhibits TGF-beta signaling...