Sudhir V Shah

Summary

Affiliation: University of Arkansas for Medical Sciences
Country: USA

Publications

  1. ncbi request reprint Role of iron in progressive renal disease
    S Shah
    Department of Medicine, Division of Nephrology, University of Arkansas for Medical Sciences and Central Arkansas Veterans Healthcare System, Little Rock, AR, USA
    Am J Kidney Dis 37:S30-3. 2001
  2. ncbi request reprint Oxidants in chronic kidney disease
    Sudhir V Shah
    University of Arkansas for Medical Sciences, 4301 West Markham Street, Slot 501, Little Rock, AR 72205, USA
    J Am Soc Nephrol 18:16-28. 2007
  3. ncbi request reprint Oxidants and iron in progressive kidney disease
    Sudhir V Shah
    Division of Nephrology, University of Arkansas for Medical Sciences Central Arkansas Veterans Healthcare System, Little Rock, AK 72205, USA
    J Ren Nutr 16:185-9. 2006
  4. ncbi request reprint Novel mechanisms in accelerated atherosclerosis in kidney disease
    Sudhir V Shah
    Division of Nephrology, Department of Internal Medicine, University of Arkansas for Medical Sciences, Little Rock, Arkansas 72205, USA
    J Ren Nutr 18:65-9. 2008
  5. doi request reprint The third World Kidney Day: looking back and thinking forward
    Sudhir V Shah
    University of Arkansas for Medical Sciences and Central Arkansas Veterans Healthcare System, Little Rock, Arkansas, USA
    Nephrol Dial Transplant 23:471-3. 2008
  6. ncbi request reprint The third World Kidney Day: looking back and thinking forward
    Sudhir V Shah
    Division of Nephrology, Department of Internal Medicine, University of Arkansas for Medical Sciences, Little Rock, Arkansas 72205, USA
    J Nephrol 21:1-5. 2008
  7. doi request reprint The Third World Kidney Day: looking back and thinking forward
    Sudhir V Shah
    University of Arkansas for Medical Sciences, Little Rock, Arkansas 72205, USA
    Am J Kidney Dis 51:349-52. 2008
  8. doi request reprint The third World Kidney Day: looking back and thinking forward
    Sudhir V Shah
    Department of Internal Medicine, Division of Nephrology, University of Arkansas for Medical Sciences, Little Rock, AR 72205, USA
    Nephron Clin Pract 108:c241-4. 2008
  9. ncbi request reprint The role of labile iron in kidney disease and treatment with chelation
    Sudhir V Shah
    Division of Nephrology, Department of Internal Medicine, University of Arkansas for Medical Sciences, Little Rock, AR 72205, USA
    Hemoglobin 33:378-85. 2009
  10. ncbi request reprint Progress toward novel treatments for chronic kidney disease
    Sudhir V Shah
    Division of Nephrology, University of Arkansas for Medical Sciences, Little Rock, Arkansas, USA
    J Ren Nutr 20:S122-6. 2010

Detail Information

Publications70

  1. ncbi request reprint Role of iron in progressive renal disease
    S Shah
    Department of Medicine, Division of Nephrology, University of Arkansas for Medical Sciences and Central Arkansas Veterans Healthcare System, Little Rock, AR, USA
    Am J Kidney Dis 37:S30-3. 2001
    ....
  2. ncbi request reprint Oxidants in chronic kidney disease
    Sudhir V Shah
    University of Arkansas for Medical Sciences, 4301 West Markham Street, Slot 501, Little Rock, AR 72205, USA
    J Am Soc Nephrol 18:16-28. 2007
    ..It concludes by reviewing the limited human data that provide some proof of concept that the observations in experimental models may be relevant to human disease...
  3. ncbi request reprint Oxidants and iron in progressive kidney disease
    Sudhir V Shah
    Division of Nephrology, University of Arkansas for Medical Sciences Central Arkansas Veterans Healthcare System, Little Rock, AK 72205, USA
    J Ren Nutr 16:185-9. 2006
    ..Taken together, these experimental studies suggest an important role of oxidants in chronic kidney disease...
  4. ncbi request reprint Novel mechanisms in accelerated atherosclerosis in kidney disease
    Sudhir V Shah
    Division of Nephrology, Department of Internal Medicine, University of Arkansas for Medical Sciences, Little Rock, Arkansas 72205, USA
    J Ren Nutr 18:65-9. 2008
    ....
  5. doi request reprint The third World Kidney Day: looking back and thinking forward
    Sudhir V Shah
    University of Arkansas for Medical Sciences and Central Arkansas Veterans Healthcare System, Little Rock, Arkansas, USA
    Nephrol Dial Transplant 23:471-3. 2008
  6. ncbi request reprint The third World Kidney Day: looking back and thinking forward
    Sudhir V Shah
    Division of Nephrology, Department of Internal Medicine, University of Arkansas for Medical Sciences, Little Rock, Arkansas 72205, USA
    J Nephrol 21:1-5. 2008
  7. doi request reprint The Third World Kidney Day: looking back and thinking forward
    Sudhir V Shah
    University of Arkansas for Medical Sciences, Little Rock, Arkansas 72205, USA
    Am J Kidney Dis 51:349-52. 2008
  8. doi request reprint The third World Kidney Day: looking back and thinking forward
    Sudhir V Shah
    Department of Internal Medicine, Division of Nephrology, University of Arkansas for Medical Sciences, Little Rock, AR 72205, USA
    Nephron Clin Pract 108:c241-4. 2008
  9. ncbi request reprint The role of labile iron in kidney disease and treatment with chelation
    Sudhir V Shah
    Division of Nephrology, Department of Internal Medicine, University of Arkansas for Medical Sciences, Little Rock, AR 72205, USA
    Hemoglobin 33:378-85. 2009
    ..Additionally, iron chelators provide protection, indicating an important role of labile iron in these diseases. These observations suggest that iron chelators may provide a new modality of prevention and treatment of kidney disease...
  10. ncbi request reprint Progress toward novel treatments for chronic kidney disease
    Sudhir V Shah
    Division of Nephrology, University of Arkansas for Medical Sciences, Little Rock, Arkansas, USA
    J Ren Nutr 20:S122-6. 2010
    ..In this review, we have outlined several promising new areas while emphasizing that large, randomized, well-controlled clinical trials are essential to reach a meaningful conclusion about the efficacy and safety of novel treatment...
  11. ncbi request reprint Role of iron in atherosclerosis
    Sudhir V Shah
    Department of Internal Medicine, Division of Nephrology, University of Arkansas for Medical Sciences and Central Arkansas Veterans Healthcare System, Little Rock, AR 72205, USA
    Am J Kidney Dis 41:S80-3. 2003
    ..Studies that address these issues, as well as those designed to establish cause and effect, are needed before one can reach meaningful conclusions about the role of iron in atherosclerosis and the therapeutic implications for patients...
  12. ncbi request reprint Induction of renal endonuclease G by cisplatin is reduced in DNase I-deficient mice
    Xiaoyan Yin
    Division of Nephrology, Department of Internal Medicine, University of Arkansas for Medical Sciences, Little Rock, Arkansas 72205, USA
    J Am Soc Nephrol 18:2544-53. 2007
    ..Furthermore, gene silencing of EndoG in vitro provided significant protection against cell death. Taken together, our data suggest that both DNase I and EndoG mediate cisplatin injury to tubular epithelial cells...
  13. doi request reprint Novel approaches targeted toward oxidative stress for the treatment of chronic kidney disease
    Sundararaman Swaminathan
    Division of Nephrology, Department of Internal Medicine, University of Arkansas for Medical Sciences, Little Rock, AR 72205, USA
    Curr Opin Nephrol Hypertens 17:143-8. 2008
    ..We review new approaches that are currently being explored to halt progression of kidney disease...
  14. ncbi request reprint p53-dependent caspase-2 activation in mitochondrial release of apoptosis-inducing factor and its role in renal tubular epithelial cell injury
    Rohit Seth
    Department of Medicine, University of Arkansas for Medical Sciences and Central Arkansas Veterans Healthcare System, Little Rock, Arkansas 72205, USA
    J Biol Chem 280:31230-9. 2005
    ..These results suggest that caspase-2 activation mediated by p53 is an important pathway involved in the mitochondrial release of AIF in response to cisplatin injury...
  15. pmc Chronic uremia stimulates LDL carbamylation and atherosclerosis
    Eugene O Apostolov
    Department of Pharmacology and Toxicology, University of Arkansas for Medical Sciences, Little Rock, AR 72205, USA
    J Am Soc Nephrol 21:1852-7. 2010
    ..In summary, these data demonstrate that elevated plasma cLDL may represent an independent risk factor for uremia-induced atherosclerosis...
  16. ncbi request reprint Mcl-1 is downregulated in cisplatin-induced apoptosis, and proteasome inhibitors restore Mcl-1 and promote survival in renal tubular epithelial cells
    Cheng Yang
    Department of Medicine, University of Arkansas for Medical Sciences, Little Rock, AR 72205, USA
    Am J Physiol Renal Physiol 292:F1710-7. 2007
    ....
  17. ncbi request reprint Regulation of caspase-3 and -9 activation in oxidant stress to RTE by forkhead transcription factors, Bcl-2 proteins, and MAP kinases
    Gur P Kaushal
    Department of Medicine, Central Arkansas Veterans Healthcare System, University of Arkansas for Medical Sciences, Slot 501, 4301 W Markham St, Little Rock, AR 72205, USA
    Am J Physiol Renal Physiol 287:F1258-68. 2004
    ..These studies suggest that enhancing renal-specific survival signals may lead to preservation of renal function during oxidant injury...
  18. doi request reprint Autophagy is associated with apoptosis in cisplatin injury to renal tubular epithelial cells
    Cheng Yang
    Department of Medicine, Central Arkansas Veterans Healthcare System and University of Arkansasfor Medical Sciences, Little Rock, Arkansas 72205, USA
    Am J Physiol Renal Physiol 294:F777-87. 2008
    ..Our results demonstrate that induction of autophagy mounts an adaptive response, suppresses cisplatin-induced apoptosis, and prolongs survival of RTEC...
  19. ncbi request reprint Carbamylated LDL
    Alexei G Basnakian
    Department of Pharmacology and Toxicology, University of Arkansas for Medical Sciences, Little Rock, Arkansas, USA
    Adv Clin Chem 51:25-52. 2010
    ..It also facilitates the proliferation of vascular smooth-muscle cell (VSMC). Studies of potential pharmacological interruption of these processes in vivo may lead to discoveries of novel therapies for atherosclerosis...
  20. pmc Endonuclease G promotes cell death of non-invasive human breast cancer cells
    Alexei G Basnakian
    Division of Nephrology, Department of Internal Medicine, University of Arkansas for Medical Sciences, Slot 501, 4301 W Markham St, Little Rock, AR 72205, USA
    Exp Cell Res 312:4139-49. 2006
    ..Our findings suggest that the presence of EndoG in non-invasive breast cancer cells determines their sensitivity to apoptosis, which may be taken into consideration for developing the chemotherapeutic strategy for cancer treatment...
  21. ncbi request reprint Carbamylated low-density lipoprotein induces death of endothelial cells: a link to atherosclerosis in patients with kidney disease
    Ercan Ok
    University of Arkansas for Medical Sciences, Little Rock, Arkansas 72205, USA
    Kidney Int 68:173-8. 2005
    ..We postulated that low-density lipoprotein (LDL) carbamylated by urea has biologic effects relevant to atherosclerosis...
  22. ncbi request reprint Sensitivity of human prostate cancer cells to chemotherapeutic drugs depends on EndoG expression regulated by promoter methylation
    Xiaoying Wang
    Division of Nephrology, Department of Internal Medicine, University of Arkansas for Medical Sciences, Little Rock, AR 72205, USA
    Cancer Lett 270:132-43. 2008
    ..These data are the first indication that EndoG may be regulated by methylation of its gene promoter, and partially by histone acetylation, and that EndoG is essential for prostate cancer cell death in the used models...
  23. ncbi request reprint Carbamylated low-density lipoprotein induces monocyte adhesion to endothelial cells through intercellular adhesion molecule-1 and vascular cell adhesion molecule-1
    Eugene O Apostolov
    Division of Nephrology, Department of Internal Medicine, University of Arkansas for Medical Sciences, 4301 W Markham St, Little Rock, AR 72205, USA
    Arterioscler Thromb Vasc Biol 27:826-32. 2007
    ..This study was aimed at testing the hypothesis that carbamylated LDL acts via inducing monocyte adhesion to endothelial cells and determining the adhesion molecules responsible for the recruitment of monocytes...
  24. ncbi request reprint Cisplatin nephrotoxicity is mediated by deoxyribonuclease I
    Alexei G Basnakian
    University of Arkansas for Medical Sciences, Department of Internal Medicine, Division of Nephrology, 4301 W Markham Street 501, Little Rock, AR 72205, USA
    J Am Soc Nephrol 16:697-702. 2005
    ..These data provide direct evidence that DNase I is essential for kidney injury induced by cisplatin...
  25. pmc Endonuclease G mediates endothelial cell death induced by carbamylated LDL
    Eugene O Apostolov
    Dept of Pharmacology and Toxicology, Univ of Arkansas for Medical Sciences, 4301 W Markham St, Slot 638, Little Rock, AR 72205, USA
    Am J Physiol Heart Circ Physiol 300:H1997-2004. 2011
    ..In summary, our data suggest that cLDL-induced endothelial toxicity is caspase independent and results from EndoG-dependent DNA fragmentation...
  26. ncbi request reprint Modified LDLs induce proliferation-mediated death of human vascular endothelial cells through MAPK pathway
    Eugene O Apostolov
    Division of Nephrology, Department of Internal Medicine, University of Arkansas for Medical Sciences, Little Rock, Arkansas 72205, USA
    Am J Physiol Heart Circ Physiol 292:H1836-46. 2007
    ..These data are the first evidence that endothelial cell death induced by cLDL or oxLDL is mediated by cell proliferation through the mitogen-activated protein kinase pathway...
  27. pmc Gadolinium contrast agent-induced CD163+ ferroportin+ osteogenic cells in nephrogenic systemic fibrosis
    Sundararaman Swaminathan
    Division of Nephrology, Department of Internal Medicine, University of Arkansas for Medical Sciences, Little Rock, Arkansas, USA
    Am J Pathol 183:796-807. 2013
    ..Gadolinium contrast-induced CD163(+)/ferroportin(+)/FGF23(+) cells with osteogenic potential may play a role in systemic fibrosis and ectopic ossification in nephrogenic systemic fibrosis...
  28. ncbi request reprint Apoptotic pathways in ischemic acute renal failure
    Gur P Kaushal
    Department of Medicine, Division of Nephrology, University of Arkansas for Medical Sciences, Little Rock, Arkansas 72205, USA
    Kidney Int 66:500-6. 2004
    ..It is likely that the cascades that lead to apoptotic or necrotic mode of cell death are activated almost simultaneously and may share some common pathways...
  29. ncbi request reprint The role of iron in diabetes and its complications
    Sundararaman Swaminathan
    Division of Nephrology, University of Arkansas for Medical Sciences, 4301 West Markham St, Slot 501, Little Rock, AR 72205, USA
    Diabetes Care 30:1926-33. 2007
  30. pmc Meprin A metalloproteinase and its role in acute kidney injury
    Gur P Kaushal
    Central Arkansas Veterans Healthcare System, 4300 West 7th St, 111D LR, Little Rock, AR 72205, USA
    Am J Physiol Renal Physiol 304:F1150-8. 2013
    ..Meprin A, therefore, plays an important role in AKI and potentially is a unique target for therapeutic intervention during AKI...
  31. doi request reprint Design of clinical trials in acute kidney injury: a report from an NIDDK workshop--prevention trials
    Mark D Okusa
    Division of Nephrology, University of Virginia Health System, Charlottesville, VA 22908 0133, USA
    Clin J Am Soc Nephrol 7:851-5. 2012
    ..The workgroups detailed primary and secondary endpoints for studies in these groups, and explored the use of adaptive clinical trial designs for trials of novel preventive strategies to improve outcomes of patients with AKI...
  32. doi request reprint Carbamylated low-density lipoprotein induces proliferation and increases adhesion molecule expression of human coronary artery smooth muscle cells
    Gulay Asci
    Department of Internal Medicine, Division of Nephrology, Ege University School of Medicine, Izmir, Turkey
    Nephrology (Carlton) 13:480-6. 2008
    ..We investigated the effects of carbamylated low-density lipoprotein (cLDL) on human coronary artery vascular smooth muscle cells (VSMC)...
  33. doi request reprint Scavenger receptors of endothelial cells mediate the uptake and cellular proatherogenic effects of carbamylated LDL
    Eugene O Apostolov
    Department of Pharmacology and Toxicology, University of Arkansas for Medical Sciences, 4301 W Markham St, Little Rock, AR 72205, USA
    Arterioscler Thromb Vasc Biol 29:1622-30. 2009
    ..The current study was designed to determine which receptors are used by cLDL and thus cause the proatherogenic effects...
  34. ncbi request reprint Kidney disease as an independent risk factor for cardiovascular events
    Jayant Kumar
    Department of Internal Medicine, Division of Nephrology, University of Arkansas for Medical Sciences and Central Arkansas Veterans Healthcare System, Little Rock 72205, USA
    J Ren Nutr 15:99-104. 2005
    ..Well-designed clinical trials should lead to the clarification of the relative importance of these factors in the pathogenesis of atherosclerotic disease...
  35. ncbi request reprint Apoptotic pathways of oxidative damage to renal tubular epithelial cells
    Alexei G Basnakian
    University of Arkansas for Medical Sciences, Little Rock, AR 72205, USA
    Antioxid Redox Signal 4:915-24. 2002
    ..Thus, the inhibition of ROM production combined with the pharmacological control of caspase and endonuclease pathways may provide future modalities in the prevention or treatment of acute renal failure in humans...
  36. ncbi request reprint Quantification of carbamylated LDL in human sera by a new sandwich ELISA
    Eugene O Apostolov
    Division of Nephrology, Department of Internal Medicine, University of Arkansas for Medical Sciences Little Rock, AR 72205, USA
    Clin Chem 51:719-28. 2005
    ..An assay for precise measurement of cLDL in serum was not previously available...
  37. doi request reprint Carbamylated low-density lipoprotein: nontraditional risk factor for cardiovascular events in patients with chronic kidney disease
    Eugene O Apostolov
    Department of Pharmacology and Toxicology, University of Arkansas for Medical Sciences, Little Rock, AR 72205, USA
    J Ren Nutr 22:134-8. 2012
    ..Future prospective studies to examine the association and/or predictive value of cLDL and studies to establish cause-effect relationship in patients with CKD are needed...
  38. ncbi request reprint Ceramide synthase is essential for endonuclease-mediated death of renal tubular epithelial cells induced by hypoxia-reoxygenation
    Alexei G Basnakian
    University of Arkansas for Medical Sciences, Little Rock, Arkansas, USA
    Am J Physiol Renal Physiol 288:F308-14. 2005
    ....
  39. pmc Metal deposition in calcific uremic arteriolopathy
    Lavanya Amuluru
    Division of Nephrology, University of Arkansas for Medical Sciences, 4301 W Markham St 501, Little Rock, AR 72205, USA
    J Am Acad Dermatol 61:73-9. 2009
    ..Although animal studies support a role for metals in the pathogenesis of CUA, metal accumulation in human tissue has not been previously evaluated...
  40. ncbi request reprint PPAR-alpha ligand ameliorates acute renal failure by reducing cisplatin-induced increased expression of renal endonuclease G
    Shenyang Li
    Dept of Medicine, University of Arkansas for Medical Sciences, Slot 501, 4301 W Markham Street, Little Rock, AR 72205, USA
    Am J Physiol Renal Physiol 287:F990-8. 2004
    ....
  41. doi request reprint Combination inhibition of the renin-angiotensin system: is more better?
    Michelle W Krause
    Division of Nephrology, Department of Internal Medicine, University of Arkansas for Medical Sciences, Central Arkansas Veterans Healthcare System, Little Rock, Arkansas, USA
    Kidney Int 80:245-55. 2011
    ..This review serves as an appraisal on the clinical evidence of combination angiotensin-converting enzyme inhibition and angiotensin II receptor blockade in both cardiovascular disease and chronic kidney disease...
  42. doi request reprint Rapid improvement of nephrogenic systemic fibrosis with rapamycin therapy: possible role of phospho-70-ribosomal-S6 kinase
    Sundararaman Swaminathan
    Division of Nephrology, Atlanta Veterans Administration Health Center, Emory University School of Medicine, Atlanta, Georgia, USA
    J Am Acad Dermatol 62:343-5. 2010
    ..While our patient did attribute disease onset to GdCA exposure, used on a single occasion for abdominal imaging, he was also exposed to iron, calcium, and darbepoetin alpha at the time of imaging...
  43. ncbi request reprint Cloning and expression of rat caspase-6 and its localization in renal ischemia/reperfusion injury
    Amar B Singh
    Department of Medicine, University of Arkansas for Medical Sciences, 4301 W Markham Street, Little Rock, AR 72205, USA
    Kidney Int 62:106-15. 2002
    ..However, the complete characterization of mRNA and protein of caspase-6 in rat and its expression in normal kidney and in disease state has not been previously elucidated...
  44. ncbi request reprint DNase I-like endonuclease in rat kidney cortex that is activated during ischemia/reperfusion injury
    Alexei G Basnakian
    Department of Internal Medicine, Division of Nephrology, University of Arkansas for Medical Sciences, 4301 W Markham Street, Slot 501, Little Rock, AR 72205, USA
    J Am Soc Nephrol 13:1000-7. 2002
    ..The data indicate that the DNase I-like endonuclease may contribute to DNA fragmentation in reperfused rat kidneys...
  45. ncbi request reprint Novel inflammatory mechanisms of accelerated atherosclerosis in kidney disease
    Sundararaman Swaminathan
    Division of Nephrology, University of Arkansas for Medical Sciences and Central Arkansas Veteran Affairs Health System, Little Rock, Arkansas 72205, USA
    Kidney Int 80:453-63. 2011
    ..In this review, we will discuss several of these novel risk factors and present the potential for the role of the immune-inflammatory system in accelerated atherosclerosis of kidney disease...
  46. pmc Meprin A and meprin alpha generate biologically functional IL-1beta from pro-IL-1beta
    Christian Herzog
    Central Arkansas Veterans Healthcare System, 4300 W 7th Street, 111D, Little Rock, AR 72205, USA
    Biochem Biophys Res Commun 379:904-8. 2009
    ..Meprin A and meprin alpha may therefore play a critical role in the production of active IL-1beta during inflammation and tissue injury...
  47. ncbi request reprint New insights into nephrogenic systemic fibrosis
    Sundararaman Swaminathan
    Division of Nephrology, Department of Internal Medicine, University of Arkansas for Medical Sciences, 4301 W Markham Street 501, Little Rock, AR 72205, USA
    J Am Soc Nephrol 18:2636-43. 2007
    ..Herein is reviewed what is currently known about this subject...
  48. ncbi request reprint Meprin, a brush-border enzyme, plays an important role in hypoxic/ischemic acute renal tubular injury in rats
    Simone Carmago
    Department of Pathology, Central Arkansas Veterans Healthcare System and the University of Arkansas for Medical Sciences, 4301 West Markham Street, Little Rock, AR 72205, USA
    Kidney Int 61:959-66. 2002
    ..The second objective was to determine if inhibition of meprin is protective against hypoxia-reoxygenation injury in vitro and ischemia-reperfusion injury in vivo...
  49. pmc Acute Kidney Injury Network: report of an initiative to improve outcomes in acute kidney injury
    Ravindra L Mehta
    Department of Medicine, University of California San Diego Medical Center 8342, 200 W, Arbor Drive, San Diego, CA 92103, USA
    Crit Care 11:R31. 2007
    ..We describe an initiative to develop uniform standards for defining and classifying AKI and to establish a forum for multidisciplinary interaction to improve care for patients with or at risk for AKI...
  50. ncbi request reprint Acute renal failure: directions for the next decade
    Norman J Siegel
    Department of Pediatrics, Division of Nephrology, Yale University School of Medicine, New Haven, Connecticut, USA
    J Am Soc Nephrol 14:2176-7. 2003
  51. pmc Challenges and advances in the treatment of AKI
    Gur P Kaushal
    Division of Nephrology, Department of Internal Medicine, University of Arkansas for Medical Sciences and Renal Section, Medicine Service, Central Arkansas Veterans Healthcare System, Little Rock, Arkansas
    J Am Soc Nephrol 25:877-83. 2014
    ....
  52. doi request reprint Design of clinical trials in AKI: a report from an NIDDK workshop. Trials of patients with sepsis and in selected hospital settings
    Bruce A Molitoris
    Department of Medicine, Indiana University, Indianapolis, IN, USA
    Clin J Am Soc Nephrol 7:856-60. 2012
    ..The groups considered trial design issues and discussed advantages and disadvantages of several short- and long-term primary endpoints in these patients...
  53. ncbi request reprint Identification and expression of deoxyribonuclease (DNase) I alternative transcripts in the rat
    Alexei G Basnakian
    Department of Internal Medicine, Division of Nephrology, University of Arkansas for Medical Sciences, 4301 West Markham Street, Slot 501, Little Rock, AR 72205, USA
    Gene 289:87-96. 2002
    ..Our data suggest that alternative splicing in 5'-UTR in the kidney may provide a prompt DNA-independent regulation of DNase I activity when DNA is damaged during ischemic injury...
  54. ncbi request reprint Hiv-associated nephropathy occurring before HIV antibody seroconversion
    M L Levin
    Divisions of Nephrology, Infectious Disease, and General Internal Medicine, Department of Medicine, and Department of Pathology, Northwestern University Medical School, Chicago, IL 60611, USA
    Am J Kidney Dis 37:E39. 2001
    ..HIVAN can occur early in the course of HIV infection, even during acute infection before seroconversion, and prolonged exposure to virus is not necessary for this renal involvement to occur in the susceptible host...
  55. ncbi request reprint World Kidney Day: an idea whose time has come
    Allan J Collins
    Natl Med J India 19:55-7. 2006
  56. ncbi request reprint Prevention of cisplatin-induced kidney epithelial cell apoptosis with a Cu superoxide dismutase-mimetic [copper2II(3,5-ditertiarybutylsalicylate)4(ethanol)4]
    Grant W Wangila
    Departments of Chemistry and Physics, University of Arkansas at Pine Bluff, 1200 North University Drive, Pine Bluff, AR 71601, USA
    Toxicol In Vitro 20:1300-12. 2006
    ..Prior treatment of patients who are to undergo treatment with Pt(II)(NH(3))(2)(Cl)(2) for their neoplastic disease with Cu(2)(II)(3,5-DTBS)(4)(Eth)(4) may be beneficial to these patients...
  57. ncbi request reprint The third World Kidney Day: looking back and thinking forward
    Sudhir V Shah
    Pediatr Nephrol 23:169-72. 2008
  58. ncbi request reprint World Kidney Day: an idea whose time has come
    Allan J Collins
    Kidney Int 69:781-2. 2006
  59. ncbi request reprint World Kidney Day: an idea whose time has come
    Allan J Collins
    Nephrology (Carlton) 11:1-2. 2006
  60. doi request reprint The Third World Kidney Day: looking back and thinking forward
    Sudhir V Shah
    Clin J Am Soc Nephrol 3:309-11. 2008
  61. ncbi request reprint Nephrogenic systemic fibrosis, gadolinium, and iron mobilization
    Sundararaman Swaminathan
    N Engl J Med 357:720-2. 2007
  62. ncbi request reprint World Kidney Day: an idea whose time has come
    Allan J Collins
    J Am Soc Nephrol 17:600-1. 2006
  63. doi request reprint The third World Kidney Day: looking back and thinking forward
    Sudhir V Shah
    Transplantation 85:495-8. 2008
  64. ncbi request reprint The third World Kidney Day: looking back and thinking forward
    Sudhir V Shah
    Nat Clin Pract Nephrol 4:1. 2008
  65. doi request reprint The Third World Kidney Day: looking back and thinking forward
    Sudhir V Shah
    Nephrology (Carlton) 13:51-2. 2008
  66. ncbi request reprint Environmental lead exposure and chronic renal disease
    Ali K Owda
    N Engl J Med 348:1810-2; author reply 1810-2. 2003
  67. ncbi request reprint Development of a clinical research agenda for acute kidney injury using an international, interdisciplinary, three-step modified Delphi process
    John A Kellum
    Department of Critical Care Medicine, University of Pittsburgh School of Medicine, Scaife Hall, Room 608, 3550 Terrace Street, Pittsburgh, PA 15261, USA
    Clin J Am Soc Nephrol 3:887-94. 2008
    ..An international, consensus-based, prioritized research agenda was sought to guide clinical and translational research in acute kidney injury...
  68. ncbi request reprint Parenteral iron therapy: beyond anaphylaxis
    Muhammad G Alam
    Kidney Int 66:457-8. 2004
  69. ncbi request reprint World Kidney Day: an idea whose time has come
    Allan J Collins
    Am J Kidney Dis 47:375-7. 2006
  70. ncbi request reprint Improving outcomes from acute kidney injury: report of an initiative
    Adeera Levin
    Am J Kidney Dis 50:1-4. 2007

Research Grants4