Research Topics
Species | J J ShackaSummaryAffiliation: University of Alabama at Birmingham Country: USA Publications
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Detail Information
Publications
Mouse models of neuronal ceroid lipofuscinoses: useful pre-clinical tools to delineate disease pathophysiology and validate therapeuticsJohn J Shacka
Neuropathology Division, Department of Pathology, University of Alabama at Birmingham, Birmingham, AL 35294, USA
Brain Res Bull 88:43-57. 2012..In this review we will assess the current state of mouse models of NCL with regards to their unique pathophysiology and how these mice have helped investigators achieve a better understanding of human NCL disease and therapy...
Lysosomal enzyme cathepsin D protects against alpha-synuclein aggregation and toxicityLiyan Qiao
Department of Pathology, University of Alabama at Birmingham, USA
Mol Brain 1:17. 2008..Our data identify a conserved CD function in α-syn degradation and identify CD as a novel target for LB disease therapeutics...
Cathepsin D deficiency and NCL/Batten disease: there's more to death than apoptosisJohn J Shacka
Department of Pathology, Neuropathology Division, University of Alabama at Birmingham, Birmingham, Alabama 35294 0017, USA
Autophagy 3:474-6. 2007..Herein, we review CD deficiency in the broader context of NCL and offer potential mechanisms for neuron death and neurodegeneration induced by CD deficiency...
Autophagy, bafilomycin and cell death: the "a-B-cs" of plecomacrolide-induced neuroprotectionJohn J Shacka
Department of Pathology, Neuropathology Division, University of Alabama at Birmingham, Birmingham, Alabama 35294 0017, USA
Autophagy 2:228-30. 2006....
Bcl-2 family and the central nervous system: from rheostat to real complexJ J Shacka
Division of Neuropathology, Department of Pathology, Birmingham, AL 35233-0017, USA
Cell Death Differ 13:1299-304. 2006
The autophagy-lysosomal degradation pathway: role in neurodegenerative disease and therapyJohn J Shacka
Division of Neuropathology, Department of Pathology, University of Alabama at Birmingham, Birmingham, AL 35294, USA
Front Biosci 13:718-36. 2008..Potential therapies for AD and PD are also discussed that may promote survival by regulating the autophagy and lysosomal degradation pathway...
p53 transcription-dependent and -independent regulation of cerebellar neural precursor cell apoptosisYing Geng
Division of Neuropathology, Department of Pathology, University of Alabama at Birmingham, Birmingham, Alabama 35294, USA
J Neuropathol Exp Neurol 66:66-74. 2007..Thus, p53 has multiple death promoting mechanisms in cerebellar NPCs...
Cathepsin D deficiency induces persistent neurodegeneration in the absence of Bax-dependent apoptosisJohn J Shacka
Department of Pathology, Neuropathology Division, University of Alabama at Birmingham, Birmingham, Alabama 35294, USA
J Neurosci 27:2081-90. 2007....
Lysosome dysfunction triggers Atg7-dependent neural apoptosisKen C Walls
Department of Pathology, University of Alabama at Birmingham, Birmingham, Alabama 35294 0017, USA
J Biol Chem 285:10497-507. 2010..These findings indicate that Atg7- and Beclin1-induced autophagy plays a cytoprotective role during starvation but that Atg7 has a unique pro-apoptotic function in response to lysosome dysfunction...
Bafilomycin A1 inhibits chloroquine-induced death of cerebellar granule neuronsJohn J Shacka
Department of Pathology, Division of Neuropathology, University of Alabama at Birmingham, SC961, 1530 3rd Ave South, Birmingham, AL 35294-0017, USA
Mol Pharmacol 69:1125-36. 2006....
Regulation of neuronal cell death and neurodegeneration by members of the Bcl-2 family: therapeutic implicationsJohn J Shacka
Division of Neuropathology, Dept of Pathology, University of Alabama at Birmingham, Birmingham, AL 35294, USA
Curr Drug Targets CNS Neurol Disord 4:25-39. 2005....
Low-dose bafilomycin attenuates neuronal cell death associated with autophagy-lysosome pathway dysfunctionVioletta N Pivtoraiko
Department of Pathology, Neuropathology Division, University of Alabama, Birmingham, AL, USA
J Neurochem 114:1193-204. 2010....
Oxidative stress and autophagy in the regulation of lysosome-dependent neuron deathVioletta N Pivtoraiko
Department of Pathology, Neuropathology Division, University of Alabama at Birmingham, Birmingham, Alabama, USA
Antioxid Redox Signal 11:481-96. 2009....
Altered regulation of phosphatidylinositol 3-kinase signaling in cathepsin D-deficient brainKen C Walls
Department of Cell Biology, Neuropathology Division, University of Alabama at Birmingham, Birmingham Alabama 35294 0017, USA
Autophagy 3:222-9. 2007..Together these observations indicate a pronounced decrease in pro-survival PI3-K signaling in CD-/- brain that may contribute to autophagic stress-induced and apoptotic neuropathology...
Kainic acid induces early and transient autophagic stress in mouse hippocampusJohn J Shacka
Division of Neuropathology, Department of Pathology, University of Alabama at Birmingham, SC 961, 1530 3rd Ave S, Birmingham, AL 35294 0017, USA
Neurosci Lett 414:57-60. 2007..Together these data indicate the transient induction of autophagic stress by KA which may serve to regulate excitotoxic death in mouse hippocampus...
Localization of electrogenic Na/bicarbonate cotransporter NBCe1 variants in rat brainD Majumdar
Department of Physiology and Biophysics, University of Alabama at Birmingham, 1918 University Boulevard, 812 MCLM, Birmingham, AL 35294, USA
Neuroscience 155:818-32. 2008..In summary, the B and C variants are the predominant NBCe1 variants in rat brain and exhibit different localization profiles...
Two distinct signaling pathways regulate peroxynitrite-induced apoptosis in PC12 cellsJ J Shacka
Department of Physiology and Biophysics, University of Alabama at Birmingham, Birmingham, AL, USA
Cell Death Differ 13:1506-14. 2006..Together, these results reveal that peroxynitrite-induced activation of the intrinsic apoptotic pathway involves interactions with the MLK/MAPK and Akt signaling pathways...
Cathepsin deficiency as a model for neuronal ceroid lipofuscinosesJohn J Shacka
Department of Pathology, Neuropathology Division, University of Alabama at Birmingham, 35294-0017, USA
Am J Pathol 167:1473-6. 2005
Erythropoietin receptor signalling is required for normal brain developmentXiaobing Yu
Laboratory of Chemical Biology, NIDDK, National Institutes of Health, Bethesda, MD 20892, USA
Development 129:505-16. 2002..5 in the developing brain, and that induction of erythropoietin and its receptor by hypoxia may contribute to selective cell survival in the brain...
