Research Topics
| Heath D SchmidtSummaryAffiliation: University of Pennsylvania Country: USA Publications
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Publications
The limbic circuitry underlying cocaine seeking encompasses the PPTg/LDTHeath D Schmidt
Department of Psychiatry, University of Pennsylvania School of Medicine, Philadelphia, PA 19104, USA
Eur J Neurosci 30:1358-69. 2009..Taken together, these results suggest that cocaine priming-induced reinstatement of drug seeking is mediated in part by a serial polysynaptic limbic subcircuit encompassing the mPFC, PPTg/LDT and VTA...
Epigenetics and psychostimulant addictionHeath D Schmidt
Department of Psychiatry, University of Pennsylvania School of Medicine, Philadelphia, PA 19104, USA
Cold Spring Harb Perspect Med 3:a012047. 2013..Identifying epigenetic signatures that define psychostimulant addiction may lead to novel, efficacious treatments for drug craving and relapse...
Functional biomarkers of depression: diagnosis, treatment, and pathophysiologyHeath D Schmidt
Department of Psychiatry, Center for Neurobiology and Behavior, University of Pennsylvania School of Medicine, Philadelphia, PA 19104, USA
Neuropsychopharmacology 36:2375-94. 2011..Together, these studies highlight the need to develop a biomarker panel for depression that aims to profile diverse peripheral factors that together provide a biological signature of MDD subtypes as well as treatment response...
Epigenetic inheritance of a cocaine-resistance phenotypeFair M Vassoler
Center for Neurobiology and Behavior, Department of Psychiatry, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, Pennsylvania, USA
Nat Neurosci 16:42-7. 2013....
Increased brain-derived neurotrophic factor (BDNF) expression in the ventral tegmental area during cocaine abstinence is associated with increased histone acetylation at BDNF exon I-containing promotersHeath D Schmidt
Center for Neurobiology and Behavior, Department of Psychiatry, Perelman School of Medicine, University of Pennsylvania School, Philadelphia, Pennsylvania, USA
J Neurochem 120:202-9. 2012....
Acute cocaine increases phosphorylation of CaMKII and GluA1 in the dorsolateral striatum of drug naïve rats, but not cocaine-experienced ratsSamantha L White
Center for Neurobiology and Behavior, Department of Psychiatry, Perelman School of Medicine, University of Pennsylvania, 125 South 31st Street, Room 1102a, Philadelphia, PA 19104, USA
Neurosci Lett 537:71-6. 2013..It is possible; therefore, that increased phosphorylation of CaMKII and GluA1 following acute cocaine is a compensatory mechanism in the DL striatum...
A negative regulator of MAP kinase causes depressive behaviorVanja Duric
Department of Psychiatry, Yale University, New Haven, Connecticut, USA
Nat Med 16:1328-32. 2010..These postmortem and preclinical studies identify MKP-1 as a key factor in MDD pathophysiology and as a new target for therapeutic interventions...
Peripheral BDNF produces antidepressant-like effects in cellular and behavioral modelsHeath D Schmidt
Department of Psychiatry, Yale University School of Medicine, Connecticut Mental Health Center, New Haven, CT 06508, USA
Neuropsychopharmacology 35:2378-91. 2010..Taken together, these results indicate that peripheral/serum BDNF may not only represent a biomarker of MDD, but also have functional consequences on molecular signaling substrates, neurogenesis, and behavior...
Cocaine-induced neuroadaptations in glutamate transmission: potential therapeutic targets for craving and addictionHeath D Schmidt
Department of Psychiatry, Center for Neurobiology and Behavior, University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania, USA
Ann N Y Acad Sci 1187:35-75. 2010..Therefore, we also outline the progress and prospects of glutamate modulators for the treatment of cocaine addiction...
